Targeted resequencing of 29 candidate genes and mouse expression studies implicate ZIC3 and FOXF1 in human VATER/VACTERL association

The VATER/VACTERL association describes the combination of congenital anomalies including vertebral defects, anorectal malformations, cardiac defects, tracheoesophageal fistula with or without esophageal atresia, renal malformations, and limb defects. As mutations in ciliary genes were observed in d...

وصف كامل

محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: El Maarri, Osman (author)
مؤلفون آخرون: Hilger, Alina C. (author), Halbritter, Jan (author), Pennimpede, Tracie (author), Sarma, Georgia (author), Braun, Daniela A. (author), Kohl, Stefan (author)
التنسيق: article
منشور في: 2015
الوصول للمادة أونلاين:http://hdl.handle.net/10725/6199
http://dx.doi.org/10.1002/humu.22859
http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php
http://onlinelibrary.wiley.com/doi/10.1002/humu.22859/full
الوسوم: إضافة وسم
لا توجد وسوم, كن أول من يضع وسما على هذه التسجيلة!
_version_ 1864513478894551040
author El Maarri, Osman
author2 Hilger, Alina C.
Halbritter, Jan
Pennimpede, Tracie
Sarma, Georgia
Braun, Daniela A.
Kohl, Stefan
author2_role author
author
author
author
author
author
author_facet El Maarri, Osman
Hilger, Alina C.
Halbritter, Jan
Pennimpede, Tracie
Sarma, Georgia
Braun, Daniela A.
Kohl, Stefan
author_role author
dc.creator.none.fl_str_mv El Maarri, Osman
Hilger, Alina C.
Halbritter, Jan
Pennimpede, Tracie
Sarma, Georgia
Braun, Daniela A.
Kohl, Stefan
dc.date.none.fl_str_mv 2015
2017-09-15T07:10:02Z
2017-09-15T07:10:02Z
2017-09-15
dc.identifier.none.fl_str_mv 1098-1004
http://hdl.handle.net/10725/6199
http://dx.doi.org/10.1002/humu.22859
Hilger, A. C., Halbritter, J., Pennimpede, T., Ven, A., Sarma, G., Braun, D. A., ... & Hermann, B. G. (2015). Targeted resequencing of 29 candidate genes and mouse expression studies implicate ZIC3 and FOXF1 in human VATER/VACTERL association. Human mutation, 36(12), 1150-1154.
http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php
http://onlinelibrary.wiley.com/doi/10.1002/humu.22859/full
dc.language.none.fl_str_mv en
dc.relation.none.fl_str_mv Human Mutation
dc.rights.*.fl_str_mv info:eu-repo/semantics/openAccess
dc.title.none.fl_str_mv Targeted resequencing of 29 candidate genes and mouse expression studies implicate ZIC3 and FOXF1 in human VATER/VACTERL association
dc.type.none.fl_str_mv Article
info:eu-repo/semantics/publishedVersion
info:eu-repo/semantics/article
description The VATER/VACTERL association describes the combination of congenital anomalies including vertebral defects, anorectal malformations, cardiac defects, tracheoesophageal fistula with or without esophageal atresia, renal malformations, and limb defects. As mutations in ciliary genes were observed in diseases related to VATER/VACTERL, we performed targeted resequencing of 25 ciliary candidate genes as well as disease-associated genes (FOXF1, HOXD13, PTEN, ZIC3) in 123 patients with VATER/VACTERL or VATER/VACTERL-like phenotype. We detected no biallelic mutation in any of the 25 ciliary candidate genes; however, identified an identical, probably disease-causing ZIC3 missense mutation (p.Gly17Cys) in four patients and a FOXF1 de novo mutation (p.Gly220Cys) in a further patient. In situ hybridization analyses in mouse embryos between E9.5 and E14.5 revealed Zic3 expression in limb and prevertebral structures, and Foxf1 expression in esophageal, tracheal, vertebral, anal, and genital tubercle tissues, hence VATER/VACTERL organ systems. These data provide strong evidence that mutations in ZIC3 or FOXF1 contribute to VATER/VACTERL.
eu_rights_str_mv openAccess
format article
id LAURepo_0aab2beaa7893fe4207280fc7f495df3
identifier_str_mv 1098-1004
Hilger, A. C., Halbritter, J., Pennimpede, T., Ven, A., Sarma, G., Braun, D. A., ... & Hermann, B. G. (2015). Targeted resequencing of 29 candidate genes and mouse expression studies implicate ZIC3 and FOXF1 in human VATER/VACTERL association. Human mutation, 36(12), 1150-1154.
language_invalid_str_mv en
network_acronym_str LAURepo
network_name_str Lebanese American University repository
oai_identifier_str oai:laur.lau.edu.lb:10725/6199
publishDate 2015
repository.mail.fl_str_mv
repository.name.fl_str_mv
repository_id_str
spelling Targeted resequencing of 29 candidate genes and mouse expression studies implicate ZIC3 and FOXF1 in human VATER/VACTERL associationEl Maarri, OsmanHilger, Alina C.Halbritter, JanPennimpede, TracieSarma, GeorgiaBraun, Daniela A.Kohl, StefanThe VATER/VACTERL association describes the combination of congenital anomalies including vertebral defects, anorectal malformations, cardiac defects, tracheoesophageal fistula with or without esophageal atresia, renal malformations, and limb defects. As mutations in ciliary genes were observed in diseases related to VATER/VACTERL, we performed targeted resequencing of 25 ciliary candidate genes as well as disease-associated genes (FOXF1, HOXD13, PTEN, ZIC3) in 123 patients with VATER/VACTERL or VATER/VACTERL-like phenotype. We detected no biallelic mutation in any of the 25 ciliary candidate genes; however, identified an identical, probably disease-causing ZIC3 missense mutation (p.Gly17Cys) in four patients and a FOXF1 de novo mutation (p.Gly220Cys) in a further patient. In situ hybridization analyses in mouse embryos between E9.5 and E14.5 revealed Zic3 expression in limb and prevertebral structures, and Foxf1 expression in esophageal, tracheal, vertebral, anal, and genital tubercle tissues, hence VATER/VACTERL organ systems. These data provide strong evidence that mutations in ZIC3 or FOXF1 contribute to VATER/VACTERL.PublishedN/A2017-09-15T07:10:02Z2017-09-15T07:10:02Z20152017-09-15Articleinfo:eu-repo/semantics/publishedVersioninfo:eu-repo/semantics/article1098-1004http://hdl.handle.net/10725/6199http://dx.doi.org/10.1002/humu.22859Hilger, A. C., Halbritter, J., Pennimpede, T., Ven, A., Sarma, G., Braun, D. A., ... & Hermann, B. G. (2015). Targeted resequencing of 29 candidate genes and mouse expression studies implicate ZIC3 and FOXF1 in human VATER/VACTERL association. Human mutation, 36(12), 1150-1154.http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.phphttp://onlinelibrary.wiley.com/doi/10.1002/humu.22859/fullenHuman Mutationinfo:eu-repo/semantics/openAccessoai:laur.lau.edu.lb:10725/61992021-03-19T10:43:12Z
spellingShingle Targeted resequencing of 29 candidate genes and mouse expression studies implicate ZIC3 and FOXF1 in human VATER/VACTERL association
El Maarri, Osman
status_str publishedVersion
title Targeted resequencing of 29 candidate genes and mouse expression studies implicate ZIC3 and FOXF1 in human VATER/VACTERL association
title_full Targeted resequencing of 29 candidate genes and mouse expression studies implicate ZIC3 and FOXF1 in human VATER/VACTERL association
title_fullStr Targeted resequencing of 29 candidate genes and mouse expression studies implicate ZIC3 and FOXF1 in human VATER/VACTERL association
title_full_unstemmed Targeted resequencing of 29 candidate genes and mouse expression studies implicate ZIC3 and FOXF1 in human VATER/VACTERL association
title_short Targeted resequencing of 29 candidate genes and mouse expression studies implicate ZIC3 and FOXF1 in human VATER/VACTERL association
title_sort Targeted resequencing of 29 candidate genes and mouse expression studies implicate ZIC3 and FOXF1 in human VATER/VACTERL association
url http://hdl.handle.net/10725/6199
http://dx.doi.org/10.1002/humu.22859
http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php
http://onlinelibrary.wiley.com/doi/10.1002/humu.22859/full