SARS-CoV-2-mediated liver injury: pathophysiology and mechanisms of disease

Background SARS-CoV-2-induced severe inflammatory response can be associated with severe medical consequences leading to multi-organ failure, including the liver. The main mechanism behind this assault is the aggressive cytokine storm that induces cytotoxicity in various organs. Of interest, hepatic...

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التفاصيل البيبلوغرافية
المؤلف الرئيسي: Choaib, Ali (author)
مؤلفون آخرون: Issa, Elio (author), El Choueiry, Francesca (author), Nasser Eldin, Jade (author), Shbaklo, Khodor (author), Alhajj, Maryline (author), Sawaya, Ramy Touma (author), Assi, Ghaith (author), Nader, Moni (author), Chatila, Rajaa (author), Faour, Wissam H. (author)
التنسيق: article
منشور في: 2022
الوصول للمادة أونلاين:http://hdl.handle.net/10725/15576
https://doi.org/10.1007/s00011-022-01683-y
http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php
https://link.springer.com/article/10.1007/s00011-022-01683-y
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author Choaib, Ali
author2 Issa, Elio
El Choueiry, Francesca
Nasser Eldin, Jade
Shbaklo, Khodor
Alhajj, Maryline
Sawaya, Ramy Touma
Assi, Ghaith
Nader, Moni
Chatila, Rajaa
Faour, Wissam H.
author2_role author
author
author
author
author
author
author
author
author
author
author_facet Choaib, Ali
Issa, Elio
El Choueiry, Francesca
Nasser Eldin, Jade
Shbaklo, Khodor
Alhajj, Maryline
Sawaya, Ramy Touma
Assi, Ghaith
Nader, Moni
Chatila, Rajaa
Faour, Wissam H.
author_role author
dc.creator.none.fl_str_mv Choaib, Ali
Issa, Elio
El Choueiry, Francesca
Nasser Eldin, Jade
Shbaklo, Khodor
Alhajj, Maryline
Sawaya, Ramy Touma
Assi, Ghaith
Nader, Moni
Chatila, Rajaa
Faour, Wissam H.
dc.date.none.fl_str_mv 2022-12-20
2023
2024-05-17T13:01:14Z
2024-05-17T13:01:14Z
dc.identifier.none.fl_str_mv 1023-3830
http://hdl.handle.net/10725/15576
https://doi.org/10.1007/s00011-022-01683-y
Choaib, A., Issa, E., El Choueiry, F., Eldin, J. N., Shbaklo, K., Alhajj, M., ... & Faour, W. H. (2023). SARS-CoV-2-mediated liver injury: Pathophysiology and mechanisms of disease. Inflammation Research, 72(2), 301-312.
http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php
https://link.springer.com/article/10.1007/s00011-022-01683-y
dc.language.none.fl_str_mv en
dc.relation.none.fl_str_mv Inflammation Research
dc.rights.*.fl_str_mv info:eu-repo/semantics/openAccess
dc.title.none.fl_str_mv SARS-CoV-2-mediated liver injury: pathophysiology and mechanisms of disease
dc.type.none.fl_str_mv Article
info:eu-repo/semantics/publishedVersion
info:eu-repo/semantics/article
description Background SARS-CoV-2-induced severe inflammatory response can be associated with severe medical consequences leading to multi-organ failure, including the liver. The main mechanism behind this assault is the aggressive cytokine storm that induces cytotoxicity in various organs. Of interest, hepatic stellate cells (HSC) respond acutely to liver injury through several molecular mechanisms, hence furthering the perpetuation of the cytokine storm and its resultant tissue damage. In addition, hepatocytes undergo apoptosis or necrosis resulting in the release of pro-inflammatory and pro-fibrogenic mediators that lead to chronic liver inflammation. Aims The aim of this review is to summarize available data on SARS-CoV-2-induced liver inflammation in addition to evaluate the potential effect of anti-inflammatory drugs in attenuating SARS-CoV-2-induced liver inflammation. Methods Thorough PubMed search was done to gather and summarize published data on SARS-CoV-2-induced liver inflammation. Additionally, various anti-inflammatory potential treatments were also documented. Results Published data documented SARS-CoV-2 infection of liver tissues and is prominent in most liver cells. Also, histological analysis showed various features of tissues damage, e.g., hepatocellular necrosis, mitosis, cellular infiltration, and fatty degeneration in addition to microvesicular steatosis and inflammation. Finally, the efficacy of the different drugs used to treat SARS-CoV-2-induced liver injury, in particular the anti-inflammatory remedies, are likely to have some beneficial effect to treat liver injury in COVID-19. Conclusion SARS-CoV-2-induced liver inflammation is a serious condition, and drugs with potent anti-inflammatory effect can play a major role in preventing irreversible liver damage in COVID-19.
eu_rights_str_mv openAccess
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Choaib, A., Issa, E., El Choueiry, F., Eldin, J. N., Shbaklo, K., Alhajj, M., ... & Faour, W. H. (2023). SARS-CoV-2-mediated liver injury: Pathophysiology and mechanisms of disease. Inflammation Research, 72(2), 301-312.
language_invalid_str_mv en
network_acronym_str LAURepo
network_name_str Lebanese American University repository
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spelling SARS-CoV-2-mediated liver injury: pathophysiology and mechanisms of diseaseChoaib, AliIssa, ElioEl Choueiry, FrancescaNasser Eldin, JadeShbaklo, KhodorAlhajj, MarylineSawaya, Ramy ToumaAssi, GhaithNader, MoniChatila, RajaaFaour, Wissam H.Background SARS-CoV-2-induced severe inflammatory response can be associated with severe medical consequences leading to multi-organ failure, including the liver. The main mechanism behind this assault is the aggressive cytokine storm that induces cytotoxicity in various organs. Of interest, hepatic stellate cells (HSC) respond acutely to liver injury through several molecular mechanisms, hence furthering the perpetuation of the cytokine storm and its resultant tissue damage. In addition, hepatocytes undergo apoptosis or necrosis resulting in the release of pro-inflammatory and pro-fibrogenic mediators that lead to chronic liver inflammation. Aims The aim of this review is to summarize available data on SARS-CoV-2-induced liver inflammation in addition to evaluate the potential effect of anti-inflammatory drugs in attenuating SARS-CoV-2-induced liver inflammation. Methods Thorough PubMed search was done to gather and summarize published data on SARS-CoV-2-induced liver inflammation. Additionally, various anti-inflammatory potential treatments were also documented. Results Published data documented SARS-CoV-2 infection of liver tissues and is prominent in most liver cells. Also, histological analysis showed various features of tissues damage, e.g., hepatocellular necrosis, mitosis, cellular infiltration, and fatty degeneration in addition to microvesicular steatosis and inflammation. Finally, the efficacy of the different drugs used to treat SARS-CoV-2-induced liver injury, in particular the anti-inflammatory remedies, are likely to have some beneficial effect to treat liver injury in COVID-19. Conclusion SARS-CoV-2-induced liver inflammation is a serious condition, and drugs with potent anti-inflammatory effect can play a major role in preventing irreversible liver damage in COVID-19.Published2024-05-17T13:01:14Z2024-05-17T13:01:14Z20232022-12-20Articleinfo:eu-repo/semantics/publishedVersioninfo:eu-repo/semantics/article1023-3830http://hdl.handle.net/10725/15576https://doi.org/10.1007/s00011-022-01683-yChoaib, A., Issa, E., El Choueiry, F., Eldin, J. N., Shbaklo, K., Alhajj, M., ... & Faour, W. H. (2023). SARS-CoV-2-mediated liver injury: Pathophysiology and mechanisms of disease. Inflammation Research, 72(2), 301-312.http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.phphttps://link.springer.com/article/10.1007/s00011-022-01683-yenInflammation Researchinfo:eu-repo/semantics/openAccessoai:laur.lau.edu.lb:10725/155762024-07-03T06:57:39Z
spellingShingle SARS-CoV-2-mediated liver injury: pathophysiology and mechanisms of disease
Choaib, Ali
status_str publishedVersion
title SARS-CoV-2-mediated liver injury: pathophysiology and mechanisms of disease
title_full SARS-CoV-2-mediated liver injury: pathophysiology and mechanisms of disease
title_fullStr SARS-CoV-2-mediated liver injury: pathophysiology and mechanisms of disease
title_full_unstemmed SARS-CoV-2-mediated liver injury: pathophysiology and mechanisms of disease
title_short SARS-CoV-2-mediated liver injury: pathophysiology and mechanisms of disease
title_sort SARS-CoV-2-mediated liver injury: pathophysiology and mechanisms of disease
url http://hdl.handle.net/10725/15576
https://doi.org/10.1007/s00011-022-01683-y
http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php
https://link.springer.com/article/10.1007/s00011-022-01683-y