Deletion of the Candida albicans PIR32 Results in Increased Virulence, Stress Response, and Upregulation of Cell Wall Chitin Deposition

Candida albicans is a common opportunistic pathogen that causes a wide variety of diseases in a human immunocompromised host leading to death. In a pathogen, cell wall proteins are important for stability as well as for acting as antigenic determinants and virulence factors. Pir32 is a cell wall pro...

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Main Author: Bahnan, Wael (author)
Other Authors: Koussa, Joseph (author), Younes, Samer (author), Abi Rizk, Marybel (author), Khalil, Bassem (author), El Sitt, Sally (author), Hanna, Samer (author), El-Sibai, Mirvat (author), Khalaf, Roy A. (author)
Format: article
Published: 2012
Online Access:http://hdl.handle.net/10725/3360
http://dx.doi.org/10.1007/s11046-012-9533-z
http://link.springer.com/article/10.1007/s11046-012-9533-z
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author Bahnan, Wael
author2 Koussa, Joseph
Younes, Samer
Abi Rizk, Marybel
Khalil, Bassem
El Sitt, Sally
Hanna, Samer
El-Sibai, Mirvat
Khalaf, Roy A.
author2_role author
author
author
author
author
author
author
author
author_facet Bahnan, Wael
Koussa, Joseph
Younes, Samer
Abi Rizk, Marybel
Khalil, Bassem
El Sitt, Sally
Hanna, Samer
El-Sibai, Mirvat
Khalaf, Roy A.
author_role author
dc.creator.none.fl_str_mv Bahnan, Wael
Koussa, Joseph
Younes, Samer
Abi Rizk, Marybel
Khalil, Bassem
El Sitt, Sally
Hanna, Samer
El-Sibai, Mirvat
Khalaf, Roy A.
dc.date.none.fl_str_mv 2012
2016-03-18T08:14:00Z
2016-03-18T08:14:00Z
2016-03-18
dc.identifier.none.fl_str_mv 0301-486X
http://hdl.handle.net/10725/3360
http://dx.doi.org/10.1007/s11046-012-9533-z
Bahnan, W., Koussa, J., Younes, S., Rizk, M. A., Khalil, B., El Sitt, S., ... & Khalaf, R. A. (2012). Deletion of the Candida albicans PIR32 results in increased virulence, stress response, and upregulation of cell wall chitin deposition. Mycopathologia, 174(2), 107-119.
http://link.springer.com/article/10.1007/s11046-012-9533-z
dc.language.none.fl_str_mv en
dc.relation.none.fl_str_mv Mycopathologia
dc.rights.*.fl_str_mv info:eu-repo/semantics/openAccess
dc.title.none.fl_str_mv Deletion of the Candida albicans PIR32 Results in Increased Virulence, Stress Response, and Upregulation of Cell Wall Chitin Deposition
dc.type.none.fl_str_mv Article
info:eu-repo/semantics/publishedVersion
info:eu-repo/semantics/article
description Candida albicans is a common opportunistic pathogen that causes a wide variety of diseases in a human immunocompromised host leading to death. In a pathogen, cell wall proteins are important for stability as well as for acting as antigenic determinants and virulence factors. Pir32 is a cell wall protein and member of the Pir protein family previously shown to be upregulated in response to macrophage contact and whose other member, Pir1, was found to be necessary for cell wall rigidity. The purpose of this study is to characterize Pir32 by generating a homozygous null strain and comparing the phenotype of the null with that of the wild-type parental strain as far as filamentation, virulence in a mouse model of disseminated candidiasis, resistance to oxidative stress and cell wall disrupting agents, in addition to adhesion, biofilm capacities, and cell wall chitin content. Our mutant was shown to be hyperfilamentous, resistant to sodium dodecyl sulfate, hydrogen peroxide, sodium chloride, and more virulent in a mouse model when compared to the wild type. These results were unexpected, considering that most cell wall mutations weaken the wall and render it more susceptible to external stress factors and suggests the possibility of a cell surface compensatory mechanism. As such, we measured cell wall chitin deposition and found a twofold increase in the mutant, possibly explaining the above-observed phenotypes.
eu_rights_str_mv openAccess
format article
id LAURepo_2f1f0ba9f6aaefd48e05eb0b7e16b80a
identifier_str_mv 0301-486X
Bahnan, W., Koussa, J., Younes, S., Rizk, M. A., Khalil, B., El Sitt, S., ... & Khalaf, R. A. (2012). Deletion of the Candida albicans PIR32 results in increased virulence, stress response, and upregulation of cell wall chitin deposition. Mycopathologia, 174(2), 107-119.
language_invalid_str_mv en
network_acronym_str LAURepo
network_name_str Lebanese American University repository
oai_identifier_str oai:laur.lau.edu.lb:10725/3360
publishDate 2012
repository.mail.fl_str_mv
repository.name.fl_str_mv
repository_id_str
spelling Deletion of the Candida albicans PIR32 Results in Increased Virulence, Stress Response, and Upregulation of Cell Wall Chitin DepositionBahnan, WaelKoussa, JosephYounes, SamerAbi Rizk, MarybelKhalil, BassemEl Sitt, SallyHanna, SamerEl-Sibai, MirvatKhalaf, Roy A.Candida albicans is a common opportunistic pathogen that causes a wide variety of diseases in a human immunocompromised host leading to death. In a pathogen, cell wall proteins are important for stability as well as for acting as antigenic determinants and virulence factors. Pir32 is a cell wall protein and member of the Pir protein family previously shown to be upregulated in response to macrophage contact and whose other member, Pir1, was found to be necessary for cell wall rigidity. The purpose of this study is to characterize Pir32 by generating a homozygous null strain and comparing the phenotype of the null with that of the wild-type parental strain as far as filamentation, virulence in a mouse model of disseminated candidiasis, resistance to oxidative stress and cell wall disrupting agents, in addition to adhesion, biofilm capacities, and cell wall chitin content. Our mutant was shown to be hyperfilamentous, resistant to sodium dodecyl sulfate, hydrogen peroxide, sodium chloride, and more virulent in a mouse model when compared to the wild type. These results were unexpected, considering that most cell wall mutations weaken the wall and render it more susceptible to external stress factors and suggests the possibility of a cell surface compensatory mechanism. As such, we measured cell wall chitin deposition and found a twofold increase in the mutant, possibly explaining the above-observed phenotypes.PublishedN/A2016-03-18T08:14:00Z2016-03-18T08:14:00Z20122016-03-18Articleinfo:eu-repo/semantics/publishedVersioninfo:eu-repo/semantics/article0301-486Xhttp://hdl.handle.net/10725/3360http://dx.doi.org/10.1007/s11046-012-9533-zBahnan, W., Koussa, J., Younes, S., Rizk, M. A., Khalil, B., El Sitt, S., ... & Khalaf, R. A. (2012). Deletion of the Candida albicans PIR32 results in increased virulence, stress response, and upregulation of cell wall chitin deposition. Mycopathologia, 174(2), 107-119.http://link.springer.com/article/10.1007/s11046-012-9533-zenMycopathologiainfo:eu-repo/semantics/openAccessoai:laur.lau.edu.lb:10725/33602020-05-07T23:10:12Z
spellingShingle Deletion of the Candida albicans PIR32 Results in Increased Virulence, Stress Response, and Upregulation of Cell Wall Chitin Deposition
Bahnan, Wael
status_str publishedVersion
title Deletion of the Candida albicans PIR32 Results in Increased Virulence, Stress Response, and Upregulation of Cell Wall Chitin Deposition
title_full Deletion of the Candida albicans PIR32 Results in Increased Virulence, Stress Response, and Upregulation of Cell Wall Chitin Deposition
title_fullStr Deletion of the Candida albicans PIR32 Results in Increased Virulence, Stress Response, and Upregulation of Cell Wall Chitin Deposition
title_full_unstemmed Deletion of the Candida albicans PIR32 Results in Increased Virulence, Stress Response, and Upregulation of Cell Wall Chitin Deposition
title_short Deletion of the Candida albicans PIR32 Results in Increased Virulence, Stress Response, and Upregulation of Cell Wall Chitin Deposition
title_sort Deletion of the Candida albicans PIR32 Results in Increased Virulence, Stress Response, and Upregulation of Cell Wall Chitin Deposition
url http://hdl.handle.net/10725/3360
http://dx.doi.org/10.1007/s11046-012-9533-z
http://link.springer.com/article/10.1007/s11046-012-9533-z