Reaction–diffusion model of atherosclerosis development

Atherosclerosis begins as an inflammation in blood vessel walls (intima). The inflammatory response of the organism leads to the recruitment of monocytes. Trapped in the intima, they differentiate into macrophages and foam cells leading to the production of inflammatory cytokines and further recruit...

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Main Author: El Khatib, N. (author)
Other Authors: Génieys, S. (author), Kazmierczak, B. (author), Volpert, V. (author)
Format: article
Published: 2012
Online Access:http://hdl.handle.net/10725/2355
http://dx.doi.org/10.1007/s00285-011-0461-1
http://link.springer.com/article/10.1007/s00285-011-0461-1
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author El Khatib, N.
author2 Génieys, S.
Kazmierczak, B.
Volpert, V.
author2_role author
author
author
author_facet El Khatib, N.
Génieys, S.
Kazmierczak, B.
Volpert, V.
author_role author
dc.creator.none.fl_str_mv El Khatib, N.
Génieys, S.
Kazmierczak, B.
Volpert, V.
dc.date.none.fl_str_mv 2012-08-21
2015-10-27T12:50:42Z
2015-10-27T12:50:42Z
2015-10-27
dc.identifier.none.fl_str_mv 0303-6812
http://hdl.handle.net/10725/2355
http://dx.doi.org/10.1007/s00285-011-0461-1
El Khatib, N., Génieys, S., Kazmierczak, B., & Volpert, V. (2012). Reaction–diffusion model of atherosclerosis development. Journal of mathematical biology, 65(2), 349-374.
http://link.springer.com/article/10.1007/s00285-011-0461-1
dc.language.none.fl_str_mv en
dc.relation.none.fl_str_mv Journal of Mathematical Biology
dc.rights.*.fl_str_mv info:eu-repo/semantics/openAccess
dc.title.none.fl_str_mv Reaction–diffusion model of atherosclerosis development
dc.type.none.fl_str_mv Article
info:eu-repo/semantics/publishedVersion
info:eu-repo/semantics/article
description Atherosclerosis begins as an inflammation in blood vessel walls (intima). The inflammatory response of the organism leads to the recruitment of monocytes. Trapped in the intima, they differentiate into macrophages and foam cells leading to the production of inflammatory cytokines and further recruitment of white blood cells. This self-accelerating process, strongly influenced by low-density lipoproteins (cholesterol), results in a dramatic increase of the width of blood vessel walls, formation of an atherosclerotic plaque and, possibly, of its rupture. We suggest a 2D mathematical model of the initiation and development of atherosclerosis which takes into account the concentration of blood cells inside the intima and of pro- and anti-inflammatory cytokines. The model represents a reaction–diffusion system in a strip with nonlinear boundary conditions which describe the recruitment of monocytes as a function of the concentration of inflammatory cytokines. We prove the existence of travelling waves described by this system and confirm our previous results which suggest that atherosclerosis develops as a reaction–diffusion wave. The theoretical results are confirmed by the results of numerical simulations.
eu_rights_str_mv openAccess
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id LAURepo_30604cb8f6fe3aa17b1104c20f024f05
identifier_str_mv 0303-6812
El Khatib, N., Génieys, S., Kazmierczak, B., & Volpert, V. (2012). Reaction–diffusion model of atherosclerosis development. Journal of mathematical biology, 65(2), 349-374.
language_invalid_str_mv en
network_acronym_str LAURepo
network_name_str Lebanese American University repository
oai_identifier_str oai:laur.lau.edu.lb:10725/2355
publishDate 2012
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spelling Reaction–diffusion model of atherosclerosis developmentEl Khatib, N.Génieys, S.Kazmierczak, B.Volpert, V.Atherosclerosis begins as an inflammation in blood vessel walls (intima). The inflammatory response of the organism leads to the recruitment of monocytes. Trapped in the intima, they differentiate into macrophages and foam cells leading to the production of inflammatory cytokines and further recruitment of white blood cells. This self-accelerating process, strongly influenced by low-density lipoproteins (cholesterol), results in a dramatic increase of the width of blood vessel walls, formation of an atherosclerotic plaque and, possibly, of its rupture. We suggest a 2D mathematical model of the initiation and development of atherosclerosis which takes into account the concentration of blood cells inside the intima and of pro- and anti-inflammatory cytokines. The model represents a reaction–diffusion system in a strip with nonlinear boundary conditions which describe the recruitment of monocytes as a function of the concentration of inflammatory cytokines. We prove the existence of travelling waves described by this system and confirm our previous results which suggest that atherosclerosis develops as a reaction–diffusion wave. The theoretical results are confirmed by the results of numerical simulations.PublishedN/A2015-10-27T12:50:42Z2015-10-27T12:50:42Z2012-08-212015-10-27Articleinfo:eu-repo/semantics/publishedVersioninfo:eu-repo/semantics/article0303-6812http://hdl.handle.net/10725/2355http://dx.doi.org/10.1007/s00285-011-0461-1El Khatib, N., Génieys, S., Kazmierczak, B., & Volpert, V. (2012). Reaction–diffusion model of atherosclerosis development. Journal of mathematical biology, 65(2), 349-374.http://link.springer.com/article/10.1007/s00285-011-0461-1enJournal of Mathematical Biologyinfo:eu-repo/semantics/openAccessoai:laur.lau.edu.lb:10725/23552016-08-02T11:05:50Z
spellingShingle Reaction–diffusion model of atherosclerosis development
El Khatib, N.
status_str publishedVersion
title Reaction–diffusion model of atherosclerosis development
title_full Reaction–diffusion model of atherosclerosis development
title_fullStr Reaction–diffusion model of atherosclerosis development
title_full_unstemmed Reaction–diffusion model of atherosclerosis development
title_short Reaction–diffusion model of atherosclerosis development
title_sort Reaction–diffusion model of atherosclerosis development
url http://hdl.handle.net/10725/2355
http://dx.doi.org/10.1007/s00285-011-0461-1
http://link.springer.com/article/10.1007/s00285-011-0461-1