Beta-Tocotrienol Exhibits More Cytotoxic Effects than Gamma-Tocotrienol on Breast Cancer Cells by Promoting Apoptosis via a P53-Independent PI3-Kinase Dependent Pathway

Studies on tocotrienols have progressively revealed the benefits of these vitamin E isoforms on human health. Beta-tocotrienol (beta-T3) is known to be less available in nature compared to other vitamin E members, which may explain the restricted number of studies on beta-T3. In the present study, w...

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Main Author: Idriss, Maya (author)
Other Authors: Hodroj, Mohammad Hassan (author), Fakhoury, Rajaa (author), Rizk, Sandra (author)
Format: article
Published: 2020
Online Access:http://hdl.handle.net/10725/11977
https://doi.org/10.3390/biom10040577
http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php
https://www.mdpi.com/2218-273X/10/4/577/htm
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author Idriss, Maya
author2 Hodroj, Mohammad Hassan
Fakhoury, Rajaa
Rizk, Sandra
author2_role author
author
author
author_facet Idriss, Maya
Hodroj, Mohammad Hassan
Fakhoury, Rajaa
Rizk, Sandra
author_role author
dc.creator.none.fl_str_mv Idriss, Maya
Hodroj, Mohammad Hassan
Fakhoury, Rajaa
Rizk, Sandra
dc.date.none.fl_str_mv 2020-07-15T10:01:26Z
2020-07-15T10:01:26Z
2020
2020-07-15
dc.identifier.none.fl_str_mv 2218-273X
http://hdl.handle.net/10725/11977
https://doi.org/10.3390/biom10040577
Idriss, M., Hodroj, M. H., Fakhoury, R., & Rizk, S. (2020). Beta-Tocotrienol Exhibits More Cytotoxic Effects than Gamma-Tocotrienol on Breast Cancer Cells by Promoting Apoptosis via a P53-Independent PI3-Kinase Dependent Pathway. Biomolecules, 10(4), 1-19
http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php
https://www.mdpi.com/2218-273X/10/4/577/htm
dc.language.none.fl_str_mv en
dc.relation.none.fl_str_mv Biomolecules
dc.rights.*.fl_str_mv info:eu-repo/semantics/openAccess
dc.title.none.fl_str_mv Beta-Tocotrienol Exhibits More Cytotoxic Effects than Gamma-Tocotrienol on Breast Cancer Cells by Promoting Apoptosis via a P53-Independent PI3-Kinase Dependent Pathway
dc.type.none.fl_str_mv Article
info:eu-repo/semantics/publishedVersion
info:eu-repo/semantics/article
description Studies on tocotrienols have progressively revealed the benefits of these vitamin E isoforms on human health. Beta-tocotrienol (beta-T3) is known to be less available in nature compared to other vitamin E members, which may explain the restricted number of studies on beta-T3. In the present study, we aim to investigate the anti-proliferative effects and the pro-apoptotic mechanisms of beta-T3 on two human breast adenocarcinoma cell lines MDA-MB-231 and MCF7. To assess cell viability, both cell lines were incubated for 24 and 48 h, with different concentrations of beta-T3 and gamma-T3, the latter being a widely studied vitamin E isoform with potent anti-cancerous properties. Cell cycle progression and apoptosis induction upon treatment with various concentrations of the beta-T3 isoform were assessed. The effect of beta-T3 on the expression level of several apoptosis-related proteins p53, cytochrome C, cleaved-PARP-1, Bax, Bcl-2, and caspase-3, in addition to key cell survival proteins p-PI3K and p-GSK-3 α/β was determined using western blot analysis. Beta-tocotrienol exhibited a significantly more potent anti-proliferative effect than gamma-tocotrienol on both cell lines regardless of their hormonal receptor status. Beta-T3 induced a mild G1 arrest on both cell lines, and triggered a mitochondrial stress-mediated apoptotic response in MDA-MB-231 cells. Mechanistically, beta-T3′s anti-neoplastic activity involved the downregulation of phosphorylated PI3K and GSK-3 cell survival proteins. These findings suggest that vitamin E beta-T3 should be considered as a promising anti-cancer agent, more effective than gamma-T3 for treating human breast cancer and deserves to be further studied to investigate its effects in vitro and on other cancer types.
eu_rights_str_mv openAccess
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id LAURepo_5a074c836e5f01c015b5d6b06f0ce78b
identifier_str_mv 2218-273X
Idriss, M., Hodroj, M. H., Fakhoury, R., & Rizk, S. (2020). Beta-Tocotrienol Exhibits More Cytotoxic Effects than Gamma-Tocotrienol on Breast Cancer Cells by Promoting Apoptosis via a P53-Independent PI3-Kinase Dependent Pathway. Biomolecules, 10(4), 1-19
language_invalid_str_mv en
network_acronym_str LAURepo
network_name_str Lebanese American University repository
oai_identifier_str oai:laur.lau.edu.lb:10725/11977
publishDate 2020
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spelling Beta-Tocotrienol Exhibits More Cytotoxic Effects than Gamma-Tocotrienol on Breast Cancer Cells by Promoting Apoptosis via a P53-Independent PI3-Kinase Dependent PathwayIdriss, MayaHodroj, Mohammad HassanFakhoury, RajaaRizk, SandraStudies on tocotrienols have progressively revealed the benefits of these vitamin E isoforms on human health. Beta-tocotrienol (beta-T3) is known to be less available in nature compared to other vitamin E members, which may explain the restricted number of studies on beta-T3. In the present study, we aim to investigate the anti-proliferative effects and the pro-apoptotic mechanisms of beta-T3 on two human breast adenocarcinoma cell lines MDA-MB-231 and MCF7. To assess cell viability, both cell lines were incubated for 24 and 48 h, with different concentrations of beta-T3 and gamma-T3, the latter being a widely studied vitamin E isoform with potent anti-cancerous properties. Cell cycle progression and apoptosis induction upon treatment with various concentrations of the beta-T3 isoform were assessed. The effect of beta-T3 on the expression level of several apoptosis-related proteins p53, cytochrome C, cleaved-PARP-1, Bax, Bcl-2, and caspase-3, in addition to key cell survival proteins p-PI3K and p-GSK-3 α/β was determined using western blot analysis. Beta-tocotrienol exhibited a significantly more potent anti-proliferative effect than gamma-tocotrienol on both cell lines regardless of their hormonal receptor status. Beta-T3 induced a mild G1 arrest on both cell lines, and triggered a mitochondrial stress-mediated apoptotic response in MDA-MB-231 cells. Mechanistically, beta-T3′s anti-neoplastic activity involved the downregulation of phosphorylated PI3K and GSK-3 cell survival proteins. These findings suggest that vitamin E beta-T3 should be considered as a promising anti-cancer agent, more effective than gamma-T3 for treating human breast cancer and deserves to be further studied to investigate its effects in vitro and on other cancer types.PublishedN/A2020-07-15T10:01:26Z2020-07-15T10:01:26Z20202020-07-15Articleinfo:eu-repo/semantics/publishedVersioninfo:eu-repo/semantics/article2218-273Xhttp://hdl.handle.net/10725/11977https://doi.org/10.3390/biom10040577Idriss, M., Hodroj, M. H., Fakhoury, R., & Rizk, S. (2020). Beta-Tocotrienol Exhibits More Cytotoxic Effects than Gamma-Tocotrienol on Breast Cancer Cells by Promoting Apoptosis via a P53-Independent PI3-Kinase Dependent Pathway. Biomolecules, 10(4), 1-19http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.phphttps://www.mdpi.com/2218-273X/10/4/577/htmenBiomoleculesinfo:eu-repo/semantics/openAccessoai:laur.lau.edu.lb:10725/119772021-03-19T10:47:40Z
spellingShingle Beta-Tocotrienol Exhibits More Cytotoxic Effects than Gamma-Tocotrienol on Breast Cancer Cells by Promoting Apoptosis via a P53-Independent PI3-Kinase Dependent Pathway
Idriss, Maya
status_str publishedVersion
title Beta-Tocotrienol Exhibits More Cytotoxic Effects than Gamma-Tocotrienol on Breast Cancer Cells by Promoting Apoptosis via a P53-Independent PI3-Kinase Dependent Pathway
title_full Beta-Tocotrienol Exhibits More Cytotoxic Effects than Gamma-Tocotrienol on Breast Cancer Cells by Promoting Apoptosis via a P53-Independent PI3-Kinase Dependent Pathway
title_fullStr Beta-Tocotrienol Exhibits More Cytotoxic Effects than Gamma-Tocotrienol on Breast Cancer Cells by Promoting Apoptosis via a P53-Independent PI3-Kinase Dependent Pathway
title_full_unstemmed Beta-Tocotrienol Exhibits More Cytotoxic Effects than Gamma-Tocotrienol on Breast Cancer Cells by Promoting Apoptosis via a P53-Independent PI3-Kinase Dependent Pathway
title_short Beta-Tocotrienol Exhibits More Cytotoxic Effects than Gamma-Tocotrienol on Breast Cancer Cells by Promoting Apoptosis via a P53-Independent PI3-Kinase Dependent Pathway
title_sort Beta-Tocotrienol Exhibits More Cytotoxic Effects than Gamma-Tocotrienol on Breast Cancer Cells by Promoting Apoptosis via a P53-Independent PI3-Kinase Dependent Pathway
url http://hdl.handle.net/10725/11977
https://doi.org/10.3390/biom10040577
http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php
https://www.mdpi.com/2218-273X/10/4/577/htm