Targeting the MAP Kinase pathway in human acute myeloid leukemia cells using a recombinant anthrax lethal toxin. (c2013)

Includes bibliographical references (leaves 41-44).

محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: Kassab, Elias (author)
التنسيق: masterThesis
منشور في: 2013
الموضوعات:
الوصول للمادة أونلاين:http://hdl.handle.net/10725/1548
https://doi.org/10.26756/th.2013.13
الوسوم: إضافة وسم
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author Kassab, Elias
author_facet Kassab, Elias
author_role author
dc.creator.none.fl_str_mv Kassab, Elias
dc.date.none.fl_str_mv 2013-09-03T09:57:47Z
2013-09-03T09:57:47Z
2013
2013-09-03
2013-05-15
dc.identifier.none.fl_str_mv http://hdl.handle.net/10725/1548
https://doi.org/10.26756/th.2013.13
dc.language.none.fl_str_mv en
dc.publisher.none.fl_str_mv Lebanese American University
dc.rights.*.fl_str_mv info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Acute myeloid leukemia -- Pathogensis
Mitogen-activated protein kinases
Blood -- Diseases -- Molecular aspects
Cancer -- Molecular aspects
Lebanese American University -- Dissertations
Dissertations, Academic
dc.title.none.fl_str_mv Targeting the MAP Kinase pathway in human acute myeloid leukemia cells using a recombinant anthrax lethal toxin. (c2013)
dc.type.none.fl_str_mv Thesis
info:eu-repo/semantics/publishedVersion
info:eu-repo/semantics/masterThesis
description Includes bibliographical references (leaves 41-44).
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network_acronym_str LAURepo
network_name_str Lebanese American University repository
oai_identifier_str oai:laur.lau.edu.lb:10725/1548
publishDate 2013
publisher.none.fl_str_mv Lebanese American University
repository.mail.fl_str_mv
repository.name.fl_str_mv
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spelling Targeting the MAP Kinase pathway in human acute myeloid leukemia cells using a recombinant anthrax lethal toxin. (c2013)Kassab, EliasAcute myeloid leukemia -- PathogensisMitogen-activated protein kinasesBlood -- Diseases -- Molecular aspectsCancer -- Molecular aspectsLebanese American University -- DissertationsDissertations, AcademicIncludes bibliographical references (leaves 41-44).In this study, we attempt to target the mitogen-activated protein kinase (MAPK) pathway in acute myeloid leukemia (AML) cells using a recombinant anthrax lethal toxin (LeTx). Around 15,000 new case of Acute Myeloid Leukemia are diagnosed each year with a fatality rate of 65%. The poor prognosis rate is due to the high proliferative and quick progressive characteristics of AML. However, most AML patients eventually relapse due to persistence of chemotherapy-resistant blasts in the bone marrow hence the need for alternative approaches employing novel, more selective mechanisms for targeting AML blasts. One such approach consists of targeting the mitogen-activated protein (MAP) kinase (MAPK) pathway in AML cells. LeTx consists of protective antigen (PrAg) and lethal factor (LF). PrAg binds cells, is cleaved by furin, oligomerizes, binds three to four molecules of LF, and undergoes endocytosis, releasing LF into the cytosol. LF cleaves MAPK kinases, inhibiting the MAPK pathway. The MAKP pathway is a conserved pathway between eukaryotes. Through a wide range of extracellular signals, the MAPK pathway regulates growth, proliferation, differentiation and death. Its constitutive activation, particularly the Ras/Raf/MEK1/2/ERK1/2 cascade promotes proliferation and survival of most human cancer cells. We tested potency of LeTx on a panel of 11 human AML cell lines. Seven cell lines showed cytotoxic responses to LeTx. Cytotoxicity of LeTx was mimicked by the specific mitogen-activated protein/extracellular signal–regulated kinase kinase 1/2 (MEK1/2) inhibitor U0126, indicating that LeTx-induced cell death is mediated through the MEK1/2–extracellular signal–regulated kinase (ERK1/2) branch of the MAPK pathway. The four LeTx-resistant cell lines were sensitive to the phosphatidylinositol 3-kinase inhibitor LY294002. Flow cytometry analysis of MAPK pathway activation revealed presence of phospho-ERK1/2 only in LeTx-sensitive cells. In this study, we have shown that a majority of AML cell lines are sensitive to the LF-mediated inhibition of the MAPK pathway. Furthermore, we have demonstrated that LeTx-induced cytotoxicity in AML cells is non-apoptotic and dependent on phospho-ERK1/2 levels.1 bound copy: xv, 44 leaves; ill. (chiefly col.); 31 cm. Available at RNL.Lebanese American University2013-09-03T09:57:47Z2013-09-03T09:57:47Z20132013-09-032013-05-15Thesisinfo:eu-repo/semantics/publishedVersioninfo:eu-repo/semantics/masterThesishttp://hdl.handle.net/10725/1548https://doi.org/10.26756/th.2013.13eninfo:eu-repo/semantics/openAccessoai:laur.lau.edu.lb:10725/15482020-05-18T14:53:55Z
spellingShingle Targeting the MAP Kinase pathway in human acute myeloid leukemia cells using a recombinant anthrax lethal toxin. (c2013)
Kassab, Elias
Acute myeloid leukemia -- Pathogensis
Mitogen-activated protein kinases
Blood -- Diseases -- Molecular aspects
Cancer -- Molecular aspects
Lebanese American University -- Dissertations
Dissertations, Academic
status_str publishedVersion
title Targeting the MAP Kinase pathway in human acute myeloid leukemia cells using a recombinant anthrax lethal toxin. (c2013)
title_full Targeting the MAP Kinase pathway in human acute myeloid leukemia cells using a recombinant anthrax lethal toxin. (c2013)
title_fullStr Targeting the MAP Kinase pathway in human acute myeloid leukemia cells using a recombinant anthrax lethal toxin. (c2013)
title_full_unstemmed Targeting the MAP Kinase pathway in human acute myeloid leukemia cells using a recombinant anthrax lethal toxin. (c2013)
title_short Targeting the MAP Kinase pathway in human acute myeloid leukemia cells using a recombinant anthrax lethal toxin. (c2013)
title_sort Targeting the MAP Kinase pathway in human acute myeloid leukemia cells using a recombinant anthrax lethal toxin. (c2013)
topic Acute myeloid leukemia -- Pathogensis
Mitogen-activated protein kinases
Blood -- Diseases -- Molecular aspects
Cancer -- Molecular aspects
Lebanese American University -- Dissertations
Dissertations, Academic
url http://hdl.handle.net/10725/1548
https://doi.org/10.26756/th.2013.13