Transglutaminase inhibition protects against oxidative stress-induced neuronal death downstream of pathological ERK activation

Molecular deletion of transglutaminase 2 (TG2) has been shown to improve function and survival in a host of neurological conditions including stroke, Huntington's disease, and Parkinson's disease. However, unifying schemes by which these cross-linking or polyaminating enzymes participate b...

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Bibliographic Details
Main Author:	Sleiman, Sama F. (author)
Other Authors:	Basso, Manuela (author), Xia, Li (author), Berlin, Jill (author), Ko, Brendan (author), Haskew-Layton, Renee (author), Kim, Eunhee (author), Antonyak, Marc A. (author), Cerione, Richard A. (author), Lismaa, Siri E. (author), Willis, Dianna (author), Cho, Sunghee (author), Ratan, Rajiv R. (author)
Format:	article
Published:	2012
Online Access:	http://hdl.handle.net/10725/6138 https://doi.org/10.1523/JNEUROSCI.3353-11.2012 http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php http://www.jneurosci.org/content/32/19/6561.short
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Transglutaminase inhibition protects against oxidative stress-induced neuronal death downstream of pathological ERK activation

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