Mathematical modelling of atherosclerosis as an inflammatory disease

Atherosclerosis is an inflammatory disease. The atherosclerosis process starts when low-density lipoproteins (LDLs) enter the intima of the blood vessel, where they are oxidized (ox-LDLs). The anti-inflammatory response triggers the recruitment of monocytes. Once in the intima, the monocytes are tra...

وصف كامل

محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: El Khatib, N (author)
مؤلفون آخرون: Genieys, S. (author), Kazmierczak, B. (author), Volpert, V. (author)
التنسيق: article
منشور في: 2009
الوصول للمادة أونلاين:http://hdl.handle.net/10725/5001
http://dx.doi.org/10.1098/rsta.2009.0142
http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php
http://rsta.royalsocietypublishing.org/content/367/1908/4877.short
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author El Khatib, N,
author2 Genieys, S.
Kazmierczak, B.
Volpert, V.
author2_role author
author
author
author_facet El Khatib, N,
Genieys, S.
Kazmierczak, B.
Volpert, V.
author_role author
dc.creator.none.fl_str_mv El Khatib, N,
Genieys, S.
Kazmierczak, B.
Volpert, V.
dc.date.none.fl_str_mv 2009
2017-01-10T08:29:25Z
2017-01-10T08:29:25Z
2017-01-10
dc.identifier.none.fl_str_mv 1364-503X
http://hdl.handle.net/10725/5001
http://dx.doi.org/10.1098/rsta.2009.0142
El Khatib, N., Genieys, S., Kazmierczak, B., & Volpert, V. (2009). Mathematical modelling of atherosclerosis as an inflammatory disease. Philosophical Transactions of the Royal Society of London A: Mathematical, Physical and Engineering Sciences, 367(1908), 4877-4886.
http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php
http://rsta.royalsocietypublishing.org/content/367/1908/4877.short
dc.language.none.fl_str_mv en
dc.relation.none.fl_str_mv Royal Society of London. Philosophical Transactions A. Mathematical, Physical and Engineering Sciences
dc.rights.*.fl_str_mv info:eu-repo/semantics/openAccess
dc.title.none.fl_str_mv Mathematical modelling of atherosclerosis as an inflammatory disease
dc.type.none.fl_str_mv Article
info:eu-repo/semantics/publishedVersion
info:eu-repo/semantics/article
description Atherosclerosis is an inflammatory disease. The atherosclerosis process starts when low-density lipoproteins (LDLs) enter the intima of the blood vessel, where they are oxidized (ox-LDLs). The anti-inflammatory response triggers the recruitment of monocytes. Once in the intima, the monocytes are transformed into macrophages and foam cells, leading to the production of inflammatory cytokines and further recruitment of monocytes. This auto-amplified process leads to the formation of an atherosclerotic plaque and, possibly, to its rupture. In this paper we develop two mathematical models based on reaction–diffusion equations in order to explain the inflammatory process. The first model is one-dimensional: it does not consider the intima’s thickness and shows that low ox-LDL concentrations in the intima do not lead to a chronic inflammatory reaction. Intermediate ox-LDL concentrations correspond to a bistable system, which can lead to a travelling wave that can be initiated by certain conditions, such as infection or injury. High ox-LDL concentrations correspond to a monostable system, and even a small perturbation of the non-inflammatory case leads to travelling-wave propagation, which corresponds to a chronic inflammatory response. The second model we suggest is two-dimensional: it represents a reaction–diffusion system in a strip with nonlinear boundary conditions to describe the recruitment of monocytes as a function of the cytokines’ concentration. We prove the existence of travelling waves and confirm our previous results, which show that atherosclerosis develops as a reaction–diffusion wave. The results of the two models are confirmed by numerical simulations. The latter show that the two-dimensional model converges to the one-dimensional one if the thickness of the intima tends to zero.
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El Khatib, N., Genieys, S., Kazmierczak, B., & Volpert, V. (2009). Mathematical modelling of atherosclerosis as an inflammatory disease. Philosophical Transactions of the Royal Society of London A: Mathematical, Physical and Engineering Sciences, 367(1908), 4877-4886.
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spelling Mathematical modelling of atherosclerosis as an inflammatory diseaseEl Khatib, N,Genieys, S.Kazmierczak, B.Volpert, V.Atherosclerosis is an inflammatory disease. The atherosclerosis process starts when low-density lipoproteins (LDLs) enter the intima of the blood vessel, where they are oxidized (ox-LDLs). The anti-inflammatory response triggers the recruitment of monocytes. Once in the intima, the monocytes are transformed into macrophages and foam cells, leading to the production of inflammatory cytokines and further recruitment of monocytes. This auto-amplified process leads to the formation of an atherosclerotic plaque and, possibly, to its rupture. In this paper we develop two mathematical models based on reaction–diffusion equations in order to explain the inflammatory process. The first model is one-dimensional: it does not consider the intima’s thickness and shows that low ox-LDL concentrations in the intima do not lead to a chronic inflammatory reaction. Intermediate ox-LDL concentrations correspond to a bistable system, which can lead to a travelling wave that can be initiated by certain conditions, such as infection or injury. High ox-LDL concentrations correspond to a monostable system, and even a small perturbation of the non-inflammatory case leads to travelling-wave propagation, which corresponds to a chronic inflammatory response. The second model we suggest is two-dimensional: it represents a reaction–diffusion system in a strip with nonlinear boundary conditions to describe the recruitment of monocytes as a function of the cytokines’ concentration. We prove the existence of travelling waves and confirm our previous results, which show that atherosclerosis develops as a reaction–diffusion wave. The results of the two models are confirmed by numerical simulations. The latter show that the two-dimensional model converges to the one-dimensional one if the thickness of the intima tends to zero.PublishedN/A2017-01-10T08:29:25Z2017-01-10T08:29:25Z20092017-01-10Articleinfo:eu-repo/semantics/publishedVersioninfo:eu-repo/semantics/article1364-503Xhttp://hdl.handle.net/10725/5001http://dx.doi.org/10.1098/rsta.2009.0142El Khatib, N., Genieys, S., Kazmierczak, B., & Volpert, V. (2009). Mathematical modelling of atherosclerosis as an inflammatory disease. Philosophical Transactions of the Royal Society of London A: Mathematical, Physical and Engineering Sciences, 367(1908), 4877-4886.http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.phphttp://rsta.royalsocietypublishing.org/content/367/1908/4877.shortenRoyal Society of London. Philosophical Transactions A. Mathematical, Physical and Engineering Sciencesinfo:eu-repo/semantics/openAccessoai:laur.lau.edu.lb:10725/50012021-03-19T10:00:53Z
spellingShingle Mathematical modelling of atherosclerosis as an inflammatory disease
El Khatib, N,
status_str publishedVersion
title Mathematical modelling of atherosclerosis as an inflammatory disease
title_full Mathematical modelling of atherosclerosis as an inflammatory disease
title_fullStr Mathematical modelling of atherosclerosis as an inflammatory disease
title_full_unstemmed Mathematical modelling of atherosclerosis as an inflammatory disease
title_short Mathematical modelling of atherosclerosis as an inflammatory disease
title_sort Mathematical modelling of atherosclerosis as an inflammatory disease
url http://hdl.handle.net/10725/5001
http://dx.doi.org/10.1098/rsta.2009.0142
http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php
http://rsta.royalsocietypublishing.org/content/367/1908/4877.short