Tau deletion promotes brain insulin resistance

<p dir="ltr">The molecular pathways underlying tau pathology–induced synaptic/cognitive deficits and neurodegeneration are poorly understood. One prevalent hypothesis is that hyperphosphorylation, misfolding, and fibrillization of tau impair synaptic plasticity and cause degeneration...

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Main Author: Elodie Marciniak (18892114) (author)
Other Authors: Antoine Leboucher (7454165) (author), Emilie Caron (11899199) (author), Tariq Ahmed (5768081) (author), Anne Tailleux (330488) (author), Julie Dumont (224385) (author), Tarik Issad (149334) (author), Ellen Gerhardt (185546) (author), Patrick Pagesy (149321) (author), Margaux Vileno (18892117) (author), Clément Bournonville (18892120) (author), Malika Hamdane (373776) (author), Kadiombo Bantubungi (330472) (author), Steve Lancel (146004) (author), Dominique Demeyer (75873) (author), Sabiha Eddarkaoui (1618621) (author), Emmanuelle Vallez (4006400) (author), Didier Vieau (122216) (author), Sandrine Humez (18892123) (author), Emilie Faivre (5507582) (author), Benjamin Grenier-Boley (577027) (author), Tiago F. Outeiro (191379) (author), Bart Staels (15303) (author), Philippe Amouyel (161598) (author), Detlef Balschun (8096618) (author), Luc Buee (16270421) (author), David Blum (344593) (author)
Published: 2017
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author Elodie Marciniak (18892114)
author2 Antoine Leboucher (7454165)
Emilie Caron (11899199)
Tariq Ahmed (5768081)
Anne Tailleux (330488)
Julie Dumont (224385)
Tarik Issad (149334)
Ellen Gerhardt (185546)
Patrick Pagesy (149321)
Margaux Vileno (18892117)
Clément Bournonville (18892120)
Malika Hamdane (373776)
Kadiombo Bantubungi (330472)
Steve Lancel (146004)
Dominique Demeyer (75873)
Sabiha Eddarkaoui (1618621)
Emmanuelle Vallez (4006400)
Didier Vieau (122216)
Sandrine Humez (18892123)
Emilie Faivre (5507582)
Benjamin Grenier-Boley (577027)
Tiago F. Outeiro (191379)
Bart Staels (15303)
Philippe Amouyel (161598)
Detlef Balschun (8096618)
Luc Buee (16270421)
David Blum (344593)
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
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author_facet Elodie Marciniak (18892114)
Antoine Leboucher (7454165)
Emilie Caron (11899199)
Tariq Ahmed (5768081)
Anne Tailleux (330488)
Julie Dumont (224385)
Tarik Issad (149334)
Ellen Gerhardt (185546)
Patrick Pagesy (149321)
Margaux Vileno (18892117)
Clément Bournonville (18892120)
Malika Hamdane (373776)
Kadiombo Bantubungi (330472)
Steve Lancel (146004)
Dominique Demeyer (75873)
Sabiha Eddarkaoui (1618621)
Emmanuelle Vallez (4006400)
Didier Vieau (122216)
Sandrine Humez (18892123)
Emilie Faivre (5507582)
Benjamin Grenier-Boley (577027)
Tiago F. Outeiro (191379)
Bart Staels (15303)
Philippe Amouyel (161598)
Detlef Balschun (8096618)
Luc Buee (16270421)
David Blum (344593)
author_role author
dc.creator.none.fl_str_mv Elodie Marciniak (18892114)
Antoine Leboucher (7454165)
Emilie Caron (11899199)
Tariq Ahmed (5768081)
Anne Tailleux (330488)
Julie Dumont (224385)
Tarik Issad (149334)
Ellen Gerhardt (185546)
Patrick Pagesy (149321)
Margaux Vileno (18892117)
Clément Bournonville (18892120)
Malika Hamdane (373776)
Kadiombo Bantubungi (330472)
Steve Lancel (146004)
Dominique Demeyer (75873)
Sabiha Eddarkaoui (1618621)
Emmanuelle Vallez (4006400)
Didier Vieau (122216)
Sandrine Humez (18892123)
Emilie Faivre (5507582)
Benjamin Grenier-Boley (577027)
Tiago F. Outeiro (191379)
Bart Staels (15303)
Philippe Amouyel (161598)
Detlef Balschun (8096618)
Luc Buee (16270421)
David Blum (344593)
dc.date.none.fl_str_mv 2017-06-26T03:00:00Z
dc.identifier.none.fl_str_mv 10.1084/jem.20161731
dc.relation.none.fl_str_mv https://figshare.com/articles/journal_contribution/Tau_deletion_promotes_brain_insulin_resistance/27088033
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Biomedical and clinical sciences
Neurosciences
Tau pathology
Synaptic plasticity
Hyperphosphorylation
Insulin signaling
Cognitive deficits
Neurodegeneration
Alzheimer's disease
dc.title.none.fl_str_mv Tau deletion promotes brain insulin resistance
dc.type.none.fl_str_mv Text
Journal contribution
info:eu-repo/semantics/publishedVersion
text
contribution to journal
description <p dir="ltr">The molecular pathways underlying tau pathology–induced synaptic/cognitive deficits and neurodegeneration are poorly understood. One prevalent hypothesis is that hyperphosphorylation, misfolding, and fibrillization of tau impair synaptic plasticity and cause degeneration. However, tau pathology may also result in the loss of specific physiological tau functions, which are largely unknown but could contribute to neuronal dysfunction. In the present study, we uncovered a novel function of tau in its ability to regulate brain insulin signaling. We found that tau deletion leads to an impaired hippocampal response to insulin, caused by altered IRS-1 and PTEN (phosphatase and tensin homologue on chromosome 10) activities. Our data also demonstrate that tau knockout mice exhibit an impaired hypothalamic anorexigenic effect of insulin that is associated with energy metabolism alterations. Consistently, we found that tau haplotypes are associated with glycemic traits in humans. The present data have far-reaching clinical implications and raise the hypothesis that pathophysiological tau loss-of-function favors brain insulin resistance, which is instrumental for cognitive and metabolic impairments in Alzheimer’s disease patients.</p><h2>Other Information</h2><p dir="ltr">Published in: Journal of Experimental Medicine<br>License: <a href="https://creativecommons.org/licenses/by/4.0/" target="_blank">https://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.1084/jem.20161731" target="_blank">https://dx.doi.org/10.1084/jem.20161731</a></p>
eu_rights_str_mv openAccess
id Manara2_009b8c3615c420b32bf0221ca84e1963
identifier_str_mv 10.1084/jem.20161731
network_acronym_str Manara2
network_name_str Manara2
oai_identifier_str oai:figshare.com:article/27088033
publishDate 2017
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rights_invalid_str_mv CC BY 4.0
spelling Tau deletion promotes brain insulin resistanceElodie Marciniak (18892114)Antoine Leboucher (7454165)Emilie Caron (11899199)Tariq Ahmed (5768081)Anne Tailleux (330488)Julie Dumont (224385)Tarik Issad (149334)Ellen Gerhardt (185546)Patrick Pagesy (149321)Margaux Vileno (18892117)Clément Bournonville (18892120)Malika Hamdane (373776)Kadiombo Bantubungi (330472)Steve Lancel (146004)Dominique Demeyer (75873)Sabiha Eddarkaoui (1618621)Emmanuelle Vallez (4006400)Didier Vieau (122216)Sandrine Humez (18892123)Emilie Faivre (5507582)Benjamin Grenier-Boley (577027)Tiago F. Outeiro (191379)Bart Staels (15303)Philippe Amouyel (161598)Detlef Balschun (8096618)Luc Buee (16270421)David Blum (344593)Biomedical and clinical sciencesNeurosciencesTau pathologySynaptic plasticityHyperphosphorylationInsulin signalingCognitive deficitsNeurodegenerationAlzheimer's disease<p dir="ltr">The molecular pathways underlying tau pathology–induced synaptic/cognitive deficits and neurodegeneration are poorly understood. One prevalent hypothesis is that hyperphosphorylation, misfolding, and fibrillization of tau impair synaptic plasticity and cause degeneration. However, tau pathology may also result in the loss of specific physiological tau functions, which are largely unknown but could contribute to neuronal dysfunction. In the present study, we uncovered a novel function of tau in its ability to regulate brain insulin signaling. We found that tau deletion leads to an impaired hippocampal response to insulin, caused by altered IRS-1 and PTEN (phosphatase and tensin homologue on chromosome 10) activities. Our data also demonstrate that tau knockout mice exhibit an impaired hypothalamic anorexigenic effect of insulin that is associated with energy metabolism alterations. Consistently, we found that tau haplotypes are associated with glycemic traits in humans. The present data have far-reaching clinical implications and raise the hypothesis that pathophysiological tau loss-of-function favors brain insulin resistance, which is instrumental for cognitive and metabolic impairments in Alzheimer’s disease patients.</p><h2>Other Information</h2><p dir="ltr">Published in: Journal of Experimental Medicine<br>License: <a href="https://creativecommons.org/licenses/by/4.0/" target="_blank">https://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.1084/jem.20161731" target="_blank">https://dx.doi.org/10.1084/jem.20161731</a></p>2017-06-26T03:00:00ZTextJournal contributioninfo:eu-repo/semantics/publishedVersiontextcontribution to journal10.1084/jem.20161731https://figshare.com/articles/journal_contribution/Tau_deletion_promotes_brain_insulin_resistance/27088033CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/270880332017-06-26T03:00:00Z
spellingShingle Tau deletion promotes brain insulin resistance
Elodie Marciniak (18892114)
Biomedical and clinical sciences
Neurosciences
Tau pathology
Synaptic plasticity
Hyperphosphorylation
Insulin signaling
Cognitive deficits
Neurodegeneration
Alzheimer's disease
status_str publishedVersion
title Tau deletion promotes brain insulin resistance
title_full Tau deletion promotes brain insulin resistance
title_fullStr Tau deletion promotes brain insulin resistance
title_full_unstemmed Tau deletion promotes brain insulin resistance
title_short Tau deletion promotes brain insulin resistance
title_sort Tau deletion promotes brain insulin resistance
topic Biomedical and clinical sciences
Neurosciences
Tau pathology
Synaptic plasticity
Hyperphosphorylation
Insulin signaling
Cognitive deficits
Neurodegeneration
Alzheimer's disease