Tau deletion promotes brain insulin resistance

<p dir="ltr">The molecular pathways underlying tau pathology–induced synaptic/cognitive deficits and neurodegeneration are poorly understood. One prevalent hypothesis is that hyperphosphorylation, misfolding, and fibrillization of tau impair synaptic plasticity and cause degeneration...

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المؤلف الرئيسي:	Elodie Marciniak (18892114) (author)
مؤلفون آخرون:	Antoine Leboucher (7454165) (author), Emilie Caron (11899199) (author), Tariq Ahmed (5768081) (author), Anne Tailleux (330488) (author), Julie Dumont (224385) (author), Tarik Issad (149334) (author), Ellen Gerhardt (185546) (author), Patrick Pagesy (149321) (author), Margaux Vileno (18892117) (author), Clément Bournonville (18892120) (author), Malika Hamdane (373776) (author), Kadiombo Bantubungi (330472) (author), Steve Lancel (146004) (author), Dominique Demeyer (75873) (author), Sabiha Eddarkaoui (1618621) (author), Emmanuelle Vallez (4006400) (author), Didier Vieau (122216) (author), Sandrine Humez (18892123) (author), Emilie Faivre (5507582) (author), Benjamin Grenier-Boley (577027) (author), Tiago F. Outeiro (191379) (author), Bart Staels (15303) (author), Philippe Amouyel (161598) (author), Detlef Balschun (8096618) (author), Luc Buee (16270421) (author), David Blum (344593) (author)
منشور في:	2017
الموضوعات:	Biomedical and clinical sciences Neurosciences Tau pathology Synaptic plasticity Hyperphosphorylation Insulin signaling Cognitive deficits Neurodegeneration Alzheimer's disease
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Tau deletion promotes brain insulin resistance

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