Genetics of glutamate and its receptors in autism spectrum disorder

<p>Autism spectrum disorder (ASD) is a neurodevelopmental impairment characterized by deficits in social interaction skills, impaired communication, and repetitive and restricted behaviors that are thought to be due to altered neurotransmission processes. The amino acid glutamate is an essenti...

Full description

Saved in:
Bibliographic Details
Main Author: Sabah Nisar (12561961) (author)
Other Authors: Ajaz A. Bhat (12984701) (author), Tariq Masoodi (8117702) (author), Sheema Hashem (14150961) (author), Sabah Akhtar (5809925) (author), Tayyiba Akbar Ali (14152572) (author), Sara Amjad (14152575) (author), Sanjeev Chawla (771773) (author), Puneet Bagga (4055866) (author), Michael P. Frenneaux (9538623) (author), Ravinder Reddy (564813) (author), Khalid Fakhro (3441305) (author), Mohammad Haris (564809) (author)
Published: 2022
Subjects:
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1864513566749491200
author Sabah Nisar (12561961)
author2 Ajaz A. Bhat (12984701)
Tariq Masoodi (8117702)
Sheema Hashem (14150961)
Sabah Akhtar (5809925)
Tayyiba Akbar Ali (14152572)
Sara Amjad (14152575)
Sanjeev Chawla (771773)
Puneet Bagga (4055866)
Michael P. Frenneaux (9538623)
Ravinder Reddy (564813)
Khalid Fakhro (3441305)
Mohammad Haris (564809)
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author_facet Sabah Nisar (12561961)
Ajaz A. Bhat (12984701)
Tariq Masoodi (8117702)
Sheema Hashem (14150961)
Sabah Akhtar (5809925)
Tayyiba Akbar Ali (14152572)
Sara Amjad (14152575)
Sanjeev Chawla (771773)
Puneet Bagga (4055866)
Michael P. Frenneaux (9538623)
Ravinder Reddy (564813)
Khalid Fakhro (3441305)
Mohammad Haris (564809)
author_role author
dc.creator.none.fl_str_mv Sabah Nisar (12561961)
Ajaz A. Bhat (12984701)
Tariq Masoodi (8117702)
Sheema Hashem (14150961)
Sabah Akhtar (5809925)
Tayyiba Akbar Ali (14152572)
Sara Amjad (14152575)
Sanjeev Chawla (771773)
Puneet Bagga (4055866)
Michael P. Frenneaux (9538623)
Ravinder Reddy (564813)
Khalid Fakhro (3441305)
Mohammad Haris (564809)
dc.date.none.fl_str_mv 2022-11-22T21:16:36Z
dc.identifier.none.fl_str_mv 10.1038/s41380-022-01506-w
dc.relation.none.fl_str_mv https://figshare.com/articles/journal_contribution/Genetics_of_glutamate_and_its_receptors_in_autism_spectrum_disorder/21598038
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Clinical sciences
Neurosciences
Cellular and Molecular Neuroscience
Psychiatry and Mental health
Molecular Biology
dc.title.none.fl_str_mv Genetics of glutamate and its receptors in autism spectrum disorder
dc.type.none.fl_str_mv Text
Journal contribution
info:eu-repo/semantics/publishedVersion
text
contribution to journal
description <p>Autism spectrum disorder (ASD) is a neurodevelopmental impairment characterized by deficits in social interaction skills, impaired communication, and repetitive and restricted behaviors that are thought to be due to altered neurotransmission processes. The amino acid glutamate is an essential excitatory neurotransmitter in the human brain that regulates cognitive functions such as learning and memory, which are usually impaired in ASD. Over the last several years, increasing evidence from genetics, neuroimaging, protein expression, and animal model studies supporting the notion of altered glutamate metabolism has heightened the interest in evaluating glutamatergic dysfunction in ASD. Numerous pharmacological, behavioral, and imaging studies have demonstrated the imbalance in excitatory and inhibitory neurotransmitters, thus revealing the involvement of the glutamatergic system in ASD pathology. Here, we review the effects of genetic alterations on glutamate and its receptors in ASD and the role of non-invasive imaging modalities in detecting these changes. We also highlight the potential therapeutic targets associated with impaired glutamatergic pathways.</p><h2>Other Information</h2> <p> Published in: Molecular Psychiatry<br> License: <a href="https://creativecommons.org/licenses/by/4.0" target="_blank">https://creativecommons.org/licenses/by/4.0</a><br>See article on publisher's website: <a href="http://dx.doi.org/10.1038/s41380-022-01506-w" target="_blank">http://dx.doi.org/10.1038/s41380-022-01506-w</a></p>
eu_rights_str_mv openAccess
id Manara2_00ea9f4d893ffb43c8b8c780743f801d
identifier_str_mv 10.1038/s41380-022-01506-w
network_acronym_str Manara2
network_name_str Manara2
oai_identifier_str oai:figshare.com:article/21598038
publishDate 2022
repository.mail.fl_str_mv
repository.name.fl_str_mv
repository_id_str
rights_invalid_str_mv CC BY 4.0
spelling Genetics of glutamate and its receptors in autism spectrum disorderSabah Nisar (12561961)Ajaz A. Bhat (12984701)Tariq Masoodi (8117702)Sheema Hashem (14150961)Sabah Akhtar (5809925)Tayyiba Akbar Ali (14152572)Sara Amjad (14152575)Sanjeev Chawla (771773)Puneet Bagga (4055866)Michael P. Frenneaux (9538623)Ravinder Reddy (564813)Khalid Fakhro (3441305)Mohammad Haris (564809)Clinical sciencesNeurosciencesCellular and Molecular NeurosciencePsychiatry and Mental healthMolecular Biology<p>Autism spectrum disorder (ASD) is a neurodevelopmental impairment characterized by deficits in social interaction skills, impaired communication, and repetitive and restricted behaviors that are thought to be due to altered neurotransmission processes. The amino acid glutamate is an essential excitatory neurotransmitter in the human brain that regulates cognitive functions such as learning and memory, which are usually impaired in ASD. Over the last several years, increasing evidence from genetics, neuroimaging, protein expression, and animal model studies supporting the notion of altered glutamate metabolism has heightened the interest in evaluating glutamatergic dysfunction in ASD. Numerous pharmacological, behavioral, and imaging studies have demonstrated the imbalance in excitatory and inhibitory neurotransmitters, thus revealing the involvement of the glutamatergic system in ASD pathology. Here, we review the effects of genetic alterations on glutamate and its receptors in ASD and the role of non-invasive imaging modalities in detecting these changes. We also highlight the potential therapeutic targets associated with impaired glutamatergic pathways.</p><h2>Other Information</h2> <p> Published in: Molecular Psychiatry<br> License: <a href="https://creativecommons.org/licenses/by/4.0" target="_blank">https://creativecommons.org/licenses/by/4.0</a><br>See article on publisher's website: <a href="http://dx.doi.org/10.1038/s41380-022-01506-w" target="_blank">http://dx.doi.org/10.1038/s41380-022-01506-w</a></p>2022-11-22T21:16:36ZTextJournal contributioninfo:eu-repo/semantics/publishedVersiontextcontribution to journal10.1038/s41380-022-01506-whttps://figshare.com/articles/journal_contribution/Genetics_of_glutamate_and_its_receptors_in_autism_spectrum_disorder/21598038CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/215980382022-11-22T21:16:36Z
spellingShingle Genetics of glutamate and its receptors in autism spectrum disorder
Sabah Nisar (12561961)
Clinical sciences
Neurosciences
Cellular and Molecular Neuroscience
Psychiatry and Mental health
Molecular Biology
status_str publishedVersion
title Genetics of glutamate and its receptors in autism spectrum disorder
title_full Genetics of glutamate and its receptors in autism spectrum disorder
title_fullStr Genetics of glutamate and its receptors in autism spectrum disorder
title_full_unstemmed Genetics of glutamate and its receptors in autism spectrum disorder
title_short Genetics of glutamate and its receptors in autism spectrum disorder
title_sort Genetics of glutamate and its receptors in autism spectrum disorder
topic Clinical sciences
Neurosciences
Cellular and Molecular Neuroscience
Psychiatry and Mental health
Molecular Biology