Cigarette smoke extract induces p38-mediated expression and ROS/rho-mediated translocation of alpha 2C adrenoceptor in human microvascular smooth muscle cells

<p dir="ltr"><u>Raynaud's phenomenon</u> (RP) is a vascular disease characterized by exaggerated <u>vasoconstriction</u> in response to stressors, mainly cold and emotional stress. This<u> vasoconstriction</u> is mediated solely by alpha 2C-ad...

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محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: Manal M. Fardoun (22303813) (author)
مؤلفون آخرون: Ayah Matar (22303816) (author), Maha Khachab (21633485) (author), Ali H. Dakroub (22303819) (author), Ali H. Eid (5461829) (author)
منشور في: 2025
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author Manal M. Fardoun (22303813)
author2 Ayah Matar (22303816)
Maha Khachab (21633485)
Ali H. Dakroub (22303819)
Ali H. Eid (5461829)
author2_role author
author
author
author
author_facet Manal M. Fardoun (22303813)
Ayah Matar (22303816)
Maha Khachab (21633485)
Ali H. Dakroub (22303819)
Ali H. Eid (5461829)
author_role author
dc.creator.none.fl_str_mv Manal M. Fardoun (22303813)
Ayah Matar (22303816)
Maha Khachab (21633485)
Ali H. Dakroub (22303819)
Ali H. Eid (5461829)
dc.date.none.fl_str_mv 2025-05-21T09:00:00Z
dc.identifier.none.fl_str_mv 10.1016/j.pcad.2025.01.002
dc.relation.none.fl_str_mv https://figshare.com/articles/journal_contribution/Cigarette_smoke_extract_induces_p38-mediated_expression_and_ROS_rho-mediated_translocation_of_alpha_2C_adrenoceptor_in_human_microvascular_smooth_muscle_cells/30198232
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Biomedical and clinical sciences
Cardiovascular medicine and haematology
Pharmacology and pharmaceutical sciences
Health sciences
Public health
Cigarette smoke extract
Reactive oxygen species
α2C-adrenoceptor
Raynaud's phenomenon
dc.title.none.fl_str_mv Cigarette smoke extract induces p38-mediated expression and ROS/rho-mediated translocation of alpha 2C adrenoceptor in human microvascular smooth muscle cells
dc.type.none.fl_str_mv Text
Journal contribution
info:eu-repo/semantics/publishedVersion
text
contribution to journal
description <p dir="ltr"><u>Raynaud's phenomenon</u> (RP) is a vascular disease characterized by exaggerated <u>vasoconstriction</u> in response to stressors, mainly cold and emotional stress. This<u> vasoconstriction</u> is mediated solely by alpha 2C-adrenoceptors (α<sub>2</sub><sub>C</sub>-AR) expressed in <u>vascular smooth muscle cells </u>of dermal <u>arterioles</u>. Several factors, among which is cigarette smoking, are associated with aggravated symptoms of and increased risk for RP. Evidence shows that cigarette smoking induces the production of <u>reactive oxygen species</u> (ROS), which is a major driver of RP pathogenesis. However, the exact mechanism by which smoking contributes to RP or α<sub>2</sub><sub>C</sub>-AR remains unclear. Here, we show that cigarette smoke extract (CSE) upregulates the expression of α<sub>2</sub><sub>C</sub>-AR in a concentration- and time-dependent manner in VSMCs extracted from human dermal arterioles. This increase is associated with the activation of <u>p38 MAPK</u>, as pretreatment with SB-202190, a p38 specific inhibitor, attenuated CSE-induced α<sub>2</sub><sub>C</sub>-AR expression. Furthermore, our results show that CSE induces ROS production followed by increased RhoA activation. We also show that CSE induces translocation of vascular α<sub>2</sub><sub>C</sub>-AR to the plasma membrane, and that this mobilization is attenuated by inhibiting ROS via <i>N</i>-acetylcysteine or <u>apocynin</u>. Similarly, inhibition of <u>Rho kinase</u> via H- 11522 abolished CSE-induced α<sub>2</sub><sub>C</sub>-AR translocation. Collectively, these results indicate that CSE activates two different <u>signaling pathways</u> to induce the expression and the translocation of α<sub>2</sub><sub>C</sub>-AR. While CSE activates a p38-dependent mechanism to increase α<sub>2</sub><sub>C</sub>AR expression, it initiates the receptor's spatial and functional <u>rescue</u> via a ROS/RhoA signaling pathway. These results provide mechanistic insight into the effect of cigarette smoking on RP, and further reinforce that smoking avoidance/cessation is critical to manage this disease, especially in the <u>absence</u> of a definitive drug for RP.</p><h2>Other Information</h2><p dir="ltr">Published in: Progress in Cardiovascular Diseases<br>License: <a href="http://creativecommons.org/licenses/by/4.0/" target="_blank">http://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.1016/j.pcad.2025.01.002" target="_blank">https://dx.doi.org/10.1016/j.pcad.2025.01.002</a></p>
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identifier_str_mv 10.1016/j.pcad.2025.01.002
network_acronym_str Manara2
network_name_str Manara2
oai_identifier_str oai:figshare.com:article/30198232
publishDate 2025
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spelling Cigarette smoke extract induces p38-mediated expression and ROS/rho-mediated translocation of alpha 2C adrenoceptor in human microvascular smooth muscle cellsManal M. Fardoun (22303813)Ayah Matar (22303816)Maha Khachab (21633485)Ali H. Dakroub (22303819)Ali H. Eid (5461829)Biomedical and clinical sciencesCardiovascular medicine and haematologyPharmacology and pharmaceutical sciencesHealth sciencesPublic healthCigarette smoke extractReactive oxygen speciesα2C-adrenoceptorRaynaud's phenomenon<p dir="ltr"><u>Raynaud's phenomenon</u> (RP) is a vascular disease characterized by exaggerated <u>vasoconstriction</u> in response to stressors, mainly cold and emotional stress. This<u> vasoconstriction</u> is mediated solely by alpha 2C-adrenoceptors (α<sub>2</sub><sub>C</sub>-AR) expressed in <u>vascular smooth muscle cells </u>of dermal <u>arterioles</u>. Several factors, among which is cigarette smoking, are associated with aggravated symptoms of and increased risk for RP. Evidence shows that cigarette smoking induces the production of <u>reactive oxygen species</u> (ROS), which is a major driver of RP pathogenesis. However, the exact mechanism by which smoking contributes to RP or α<sub>2</sub><sub>C</sub>-AR remains unclear. Here, we show that cigarette smoke extract (CSE) upregulates the expression of α<sub>2</sub><sub>C</sub>-AR in a concentration- and time-dependent manner in VSMCs extracted from human dermal arterioles. This increase is associated with the activation of <u>p38 MAPK</u>, as pretreatment with SB-202190, a p38 specific inhibitor, attenuated CSE-induced α<sub>2</sub><sub>C</sub>-AR expression. Furthermore, our results show that CSE induces ROS production followed by increased RhoA activation. We also show that CSE induces translocation of vascular α<sub>2</sub><sub>C</sub>-AR to the plasma membrane, and that this mobilization is attenuated by inhibiting ROS via <i>N</i>-acetylcysteine or <u>apocynin</u>. Similarly, inhibition of <u>Rho kinase</u> via H- 11522 abolished CSE-induced α<sub>2</sub><sub>C</sub>-AR translocation. Collectively, these results indicate that CSE activates two different <u>signaling pathways</u> to induce the expression and the translocation of α<sub>2</sub><sub>C</sub>-AR. While CSE activates a p38-dependent mechanism to increase α<sub>2</sub><sub>C</sub>AR expression, it initiates the receptor's spatial and functional <u>rescue</u> via a ROS/RhoA signaling pathway. These results provide mechanistic insight into the effect of cigarette smoking on RP, and further reinforce that smoking avoidance/cessation is critical to manage this disease, especially in the <u>absence</u> of a definitive drug for RP.</p><h2>Other Information</h2><p dir="ltr">Published in: Progress in Cardiovascular Diseases<br>License: <a href="http://creativecommons.org/licenses/by/4.0/" target="_blank">http://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.1016/j.pcad.2025.01.002" target="_blank">https://dx.doi.org/10.1016/j.pcad.2025.01.002</a></p>2025-05-21T09:00:00ZTextJournal contributioninfo:eu-repo/semantics/publishedVersiontextcontribution to journal10.1016/j.pcad.2025.01.002https://figshare.com/articles/journal_contribution/Cigarette_smoke_extract_induces_p38-mediated_expression_and_ROS_rho-mediated_translocation_of_alpha_2C_adrenoceptor_in_human_microvascular_smooth_muscle_cells/30198232CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/301982322025-05-21T09:00:00Z
spellingShingle Cigarette smoke extract induces p38-mediated expression and ROS/rho-mediated translocation of alpha 2C adrenoceptor in human microvascular smooth muscle cells
Manal M. Fardoun (22303813)
Biomedical and clinical sciences
Cardiovascular medicine and haematology
Pharmacology and pharmaceutical sciences
Health sciences
Public health
Cigarette smoke extract
Reactive oxygen species
α2C-adrenoceptor
Raynaud's phenomenon
status_str publishedVersion
title Cigarette smoke extract induces p38-mediated expression and ROS/rho-mediated translocation of alpha 2C adrenoceptor in human microvascular smooth muscle cells
title_full Cigarette smoke extract induces p38-mediated expression and ROS/rho-mediated translocation of alpha 2C adrenoceptor in human microvascular smooth muscle cells
title_fullStr Cigarette smoke extract induces p38-mediated expression and ROS/rho-mediated translocation of alpha 2C adrenoceptor in human microvascular smooth muscle cells
title_full_unstemmed Cigarette smoke extract induces p38-mediated expression and ROS/rho-mediated translocation of alpha 2C adrenoceptor in human microvascular smooth muscle cells
title_short Cigarette smoke extract induces p38-mediated expression and ROS/rho-mediated translocation of alpha 2C adrenoceptor in human microvascular smooth muscle cells
title_sort Cigarette smoke extract induces p38-mediated expression and ROS/rho-mediated translocation of alpha 2C adrenoceptor in human microvascular smooth muscle cells
topic Biomedical and clinical sciences
Cardiovascular medicine and haematology
Pharmacology and pharmaceutical sciences
Health sciences
Public health
Cigarette smoke extract
Reactive oxygen species
α2C-adrenoceptor
Raynaud's phenomenon