DIAPH3 predicts survival of patients with <i>MGMT</i>-methylated glioblastoma

<h3>Background</h3><p dir="ltr">Glioblastoma is one of the most aggressive primary brain tumors, with a poor outcome despite multimodal treatment. Methylation of the <i>MGMT</i> promoter, which predicts the response to temozolomide, is a well-established progn...

وصف كامل

محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: Georges Chehade (18007084) (author)
مؤلفون آخرون: Nady El Hajj (686554) (author), Mohamed Aittaleb (18007087) (author), Maisa I. Alkailani (15070764) (author), Yosra Bejaoui (8552574) (author), Asma Mahdi (18007090) (author), Arwa A. H. Aldaalis (18007093) (author), Michael Verbiest (532371) (author), Julie Lelotte (9223075) (author), Nuria Ruiz-Reig (14434026) (author), Irene Durá (18007096) (author), Christian Raftopoulos (843696) (author), Nicolas Tajeddine (18007099) (author), Fadel Tissir (245153) (author)
منشور في: 2024
الموضوعات:
الوسوم: إضافة وسم
لا توجد وسوم, كن أول من يضع وسما على هذه التسجيلة!
الوصف
الملخص:<h3>Background</h3><p dir="ltr">Glioblastoma is one of the most aggressive primary brain tumors, with a poor outcome despite multimodal treatment. Methylation of the <i>MGMT</i> promoter, which predicts the response to temozolomide, is a well-established prognostic marker for glioblastoma. However, a difference in survival can still be detected within the <i>MGMT</i> methylated group, with some patients exhibiting a shorter survival than others, emphasizing the need for additional predictive factors.</p><h3>Methods</h3><p dir="ltr">We analyzed <i>DIAPH3</i> expression in glioblastoma samples from the cancer genome atlas (TCGA). We also retrospectively analyzed one hundred seventeen histological glioblastomas from patients operated on at Saint-Luc University Hospital between May 2013 and August 2019. We analyzed the<i> DIAPH3</i> expression, explored the relationship between mRNA levels and Patient’s survival after the surgical resection. Finally, we assessed the methylation pattern of the <i>DIAPH3</i> promoter using a targeted deep bisulfite sequencing approach.</p><h3>Results</h3><p dir="ltr">We found that 36% and 1% of the TCGA glioblastoma samples exhibit copy number alterations and mutations in <i>DIAPH3</i>, respectively. We scrutinized the expression of <i>DIAPH3</i> at single cell level and detected an overlap with MKI67 expression in glioblastoma proliferating cells, including neural progenitor-like, oligodendrocyte progenitor-like and astrocyte-like states. We quantitatively analyzed <i>DIAPH3</i> expression in our cohort and uncovered a positive correlation between <i>DIAPH3</i> mRNA level and patient’s survival. The effect of <i>DIAPH3</i> was prominent in <i>MGMT</i>-methylated glioblastoma. Finally, we report that the expression of <i>DIAPH3</i> is at least partially regulated by the methylation of three CpG sites in the promoter region.</p><h3>Conclusion</h3><p dir="ltr">We propose that combining the <i>DIAPH3</i> expression with <i>MGMT</i> methylation could offer a better prediction of survival and more adapted postsurgical treatment for patients with <i>MGMT</i>-methylated glioblastoma.</p><h2>Other Information</h2><p dir="ltr">Published in: Frontiers in Oncology<br>License: <a href="https://creativecommons.org/licenses/by/4.0/" target="_blank">https://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.3389/fonc.2024.1359652" target="_blank">https://dx.doi.org/10.3389/fonc.2024.1359652</a></p>