Differential gene expression of tumor-infiltrating CD33<sup>+</sup> myeloid cells in advanced- versus early-stage colorectal cancer

<p>Colorectal cancer (CRC) has high mortality rates, especially in patients with advanced disease stages, who often do not respond to therapy. The cellular components of the tumor microenvironment are essentially responsible for dictating disease progression and response to therapy. Expansion...

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محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: Salman M. Toor (8854751) (author)
مؤلفون آخرون: Rowaida Z. Taha (8854754) (author), Varun Sasidharan Nair (5396393) (author), Reem Saleh (3513056) (author), Khaled Murshed (8309781) (author), Mohamed Abu Nada (8309784) (author), Eyad Elkord (5396390) (author)
منشور في: 2020
الموضوعات:
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author Salman M. Toor (8854751)
author2 Rowaida Z. Taha (8854754)
Varun Sasidharan Nair (5396393)
Reem Saleh (3513056)
Khaled Murshed (8309781)
Mohamed Abu Nada (8309784)
Eyad Elkord (5396390)
author2_role author
author
author
author
author
author
author_facet Salman M. Toor (8854751)
Rowaida Z. Taha (8854754)
Varun Sasidharan Nair (5396393)
Reem Saleh (3513056)
Khaled Murshed (8309781)
Mohamed Abu Nada (8309784)
Eyad Elkord (5396390)
author_role author
dc.creator.none.fl_str_mv Salman M. Toor (8854751)
Rowaida Z. Taha (8854754)
Varun Sasidharan Nair (5396393)
Reem Saleh (3513056)
Khaled Murshed (8309781)
Mohamed Abu Nada (8309784)
Eyad Elkord (5396390)
dc.date.none.fl_str_mv 2020-09-30T06:00:00Z
dc.identifier.none.fl_str_mv 10.1007/s00262-020-02727-0
dc.relation.none.fl_str_mv https://figshare.com/articles/journal_contribution/Differential_gene_expression_of_tumor-infiltrating_CD33_myeloid_cells_in_advanced-_versus_early-stage_colorectal_cancer/21596958
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Biomedical and clinical sciences
Immunology
Oncology and carcinogenesis
Cancer Research
Oncology
Immunology
Immunology and Allergy
dc.title.none.fl_str_mv Differential gene expression of tumor-infiltrating CD33<sup>+</sup> myeloid cells in advanced- versus early-stage colorectal cancer
dc.type.none.fl_str_mv Text
Journal contribution
info:eu-repo/semantics/publishedVersion
text
contribution to journal
description <p>Colorectal cancer (CRC) has high mortality rates, especially in patients with advanced disease stages, who often do not respond to therapy. The cellular components of the tumor microenvironment are essentially responsible for dictating disease progression and response to therapy. Expansion of different myeloid cell subsets in CRC tumors has been reported previously. However, tumor-infiltrating myeloid cells have both pro- and anti-tumor roles in disease progression. In this study, we performed transcriptomic profiling of cells of myeloid lineage (CD33+) from bulk CRC tumors at varying disease stages. We identified differentially expressed genes and pathways between CRC patients with advanced stage and early stages. We found that pro-angiogenic and hypoxia-related genes were upregulated, while genes related to immune and inflammatory responses were downregulated in CD33+ myeloid cells from patients with advanced stages, implying that immune cell recruitment and activation could be compromised in advanced disease stages. Moreover, we identified a unique “poor prognosis CD33+ gene signature” by aligning top upregulated and downregulated genes in tumor-infiltrating myeloid cells from our analyses with data from The Cancer Genome Atlas. Our results showed that this gene signature is an independent prognostic indicator for disease-specific survival in CRC patients, potentially reflecting its clinical importance.</p><h2>Other Information</h2> <p> Published in: Cancer Immunology, Immunotherapy<br> License: <a href="https://creativecommons.org/licenses/by/4.0" target="_blank">https://creativecommons.org/licenses/by/4.0</a><br>See article on publisher's website: <a href="http://dx.doi.org/10.1007/s00262-020-02727-0" target="_blank">http://dx.doi.org/10.1007/s00262-020-02727-0</a></p>
eu_rights_str_mv openAccess
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identifier_str_mv 10.1007/s00262-020-02727-0
network_acronym_str Manara2
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oai_identifier_str oai:figshare.com:article/21596958
publishDate 2020
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rights_invalid_str_mv CC BY 4.0
spelling Differential gene expression of tumor-infiltrating CD33<sup>+</sup> myeloid cells in advanced- versus early-stage colorectal cancerSalman M. Toor (8854751)Rowaida Z. Taha (8854754)Varun Sasidharan Nair (5396393)Reem Saleh (3513056)Khaled Murshed (8309781)Mohamed Abu Nada (8309784)Eyad Elkord (5396390)Biomedical and clinical sciencesImmunologyOncology and carcinogenesisCancer ResearchOncologyImmunologyImmunology and Allergy<p>Colorectal cancer (CRC) has high mortality rates, especially in patients with advanced disease stages, who often do not respond to therapy. The cellular components of the tumor microenvironment are essentially responsible for dictating disease progression and response to therapy. Expansion of different myeloid cell subsets in CRC tumors has been reported previously. However, tumor-infiltrating myeloid cells have both pro- and anti-tumor roles in disease progression. In this study, we performed transcriptomic profiling of cells of myeloid lineage (CD33+) from bulk CRC tumors at varying disease stages. We identified differentially expressed genes and pathways between CRC patients with advanced stage and early stages. We found that pro-angiogenic and hypoxia-related genes were upregulated, while genes related to immune and inflammatory responses were downregulated in CD33+ myeloid cells from patients with advanced stages, implying that immune cell recruitment and activation could be compromised in advanced disease stages. Moreover, we identified a unique “poor prognosis CD33+ gene signature” by aligning top upregulated and downregulated genes in tumor-infiltrating myeloid cells from our analyses with data from The Cancer Genome Atlas. Our results showed that this gene signature is an independent prognostic indicator for disease-specific survival in CRC patients, potentially reflecting its clinical importance.</p><h2>Other Information</h2> <p> Published in: Cancer Immunology, Immunotherapy<br> License: <a href="https://creativecommons.org/licenses/by/4.0" target="_blank">https://creativecommons.org/licenses/by/4.0</a><br>See article on publisher's website: <a href="http://dx.doi.org/10.1007/s00262-020-02727-0" target="_blank">http://dx.doi.org/10.1007/s00262-020-02727-0</a></p>2020-09-30T06:00:00ZTextJournal contributioninfo:eu-repo/semantics/publishedVersiontextcontribution to journal10.1007/s00262-020-02727-0https://figshare.com/articles/journal_contribution/Differential_gene_expression_of_tumor-infiltrating_CD33_myeloid_cells_in_advanced-_versus_early-stage_colorectal_cancer/21596958CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/215969582020-09-30T06:00:00Z
spellingShingle Differential gene expression of tumor-infiltrating CD33<sup>+</sup> myeloid cells in advanced- versus early-stage colorectal cancer
Salman M. Toor (8854751)
Biomedical and clinical sciences
Immunology
Oncology and carcinogenesis
Cancer Research
Oncology
Immunology
Immunology and Allergy
status_str publishedVersion
title Differential gene expression of tumor-infiltrating CD33<sup>+</sup> myeloid cells in advanced- versus early-stage colorectal cancer
title_full Differential gene expression of tumor-infiltrating CD33<sup>+</sup> myeloid cells in advanced- versus early-stage colorectal cancer
title_fullStr Differential gene expression of tumor-infiltrating CD33<sup>+</sup> myeloid cells in advanced- versus early-stage colorectal cancer
title_full_unstemmed Differential gene expression of tumor-infiltrating CD33<sup>+</sup> myeloid cells in advanced- versus early-stage colorectal cancer
title_short Differential gene expression of tumor-infiltrating CD33<sup>+</sup> myeloid cells in advanced- versus early-stage colorectal cancer
title_sort Differential gene expression of tumor-infiltrating CD33<sup>+</sup> myeloid cells in advanced- versus early-stage colorectal cancer
topic Biomedical and clinical sciences
Immunology
Oncology and carcinogenesis
Cancer Research
Oncology
Immunology
Immunology and Allergy