Dysregulated FOXM1 signaling in the regulation of cancer stem cells

<p>Since the introduction of the cancer stem cell (CSC) paradigm, significant advances have been made in understanding the functional and biological plasticity of these elusive components in malignancies. Endowed with self-renewing abilities and multilineage differentiation potential, CSCs hav...

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Main Author: Gulab Sher (12984698) (author)
Other Authors: Tariq Masoodi (8117702) (author), Kalyani Patil (2679424) (author), Sabah Akhtar (5809925) (author), Shilpa Kuttikrishnan (3520079) (author), Aamir Ahmad (233201) (author), Shahab Uddin (154400) (author)
Published: 2022
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author Gulab Sher (12984698)
author2 Tariq Masoodi (8117702)
Kalyani Patil (2679424)
Sabah Akhtar (5809925)
Shilpa Kuttikrishnan (3520079)
Aamir Ahmad (233201)
Shahab Uddin (154400)
author2_role author
author
author
author
author
author
author_facet Gulab Sher (12984698)
Tariq Masoodi (8117702)
Kalyani Patil (2679424)
Sabah Akhtar (5809925)
Shilpa Kuttikrishnan (3520079)
Aamir Ahmad (233201)
Shahab Uddin (154400)
author_role author
dc.creator.none.fl_str_mv Gulab Sher (12984698)
Tariq Masoodi (8117702)
Kalyani Patil (2679424)
Sabah Akhtar (5809925)
Shilpa Kuttikrishnan (3520079)
Aamir Ahmad (233201)
Shahab Uddin (154400)
dc.date.none.fl_str_mv 2022-11-01T00:00:00Z
dc.identifier.none.fl_str_mv 10.1016/j.semcancer.2022.07.009
dc.relation.none.fl_str_mv https://figshare.com/articles/journal_contribution/Dysregulated_FOXM1_signaling_in_the_regulation_of_cancer_stem_cells/24717759
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Biomedical and clinical sciences
Medical biotechnology
Oncology and carcinogenesis
Malignancies
Signaling pathways
FOXM1
Cancer stem cells
Self-renewal
Carcinogenesis
dc.title.none.fl_str_mv Dysregulated FOXM1 signaling in the regulation of cancer stem cells
dc.type.none.fl_str_mv Text
Journal contribution
info:eu-repo/semantics/publishedVersion
text
contribution to journal
description <p>Since the introduction of the cancer stem cell (CSC) paradigm, significant advances have been made in understanding the functional and biological plasticity of these elusive components in malignancies. Endowed with self-renewing abilities and multilineage differentiation potential, CSCs have emerged as cellular drivers of virtually all facets of tumor biology, including metastasis, tumor recurrence/relapse, and drug resistance. The functional and biological characteristics of CSCs, such as self-renewal, cell fate decisions, survival, proliferation, and differentiation are regulated by an array of extracellular factors, signaling pathways, and pluripotent transcriptional factors. Besides the well-characterized regulatory role of transcription factors OCT4, SOX2, NANOG, KLF4, and MYC in CSCs, evidence for the central role of Forkhead box transcription factor FOXM1 in the establishment, maintenance, and functions of CSCs is accumulating. Conventionally identified as a master regulator of the cell cycle, a comprehensive understanding of this molecule has revealed its multifarious oncogenic potential and uncovered its role in angiogenesis, invasion, migration, self-renewal, and drug resistance. This review compiles the large body of literature that has accumulated in recent years that provides evidence for the mechanisms by which FOXM1 expression promotes stemness in glioblastoma, breast, colon, ovarian, lung, hepatic, and pancreatic carcinomas. We have also compiled the data showing the association of stem cell mediators with FOXM1 using TCGA mRNA expression data. Further, the prognostic importance of FOXM1 and other stem cell markers is presented. The delineation of FOXM1-mediated regulation of CSCs can aid in the development of molecularly targeted pharmacological approaches directed at the selective eradication of CSCs in several human malignancies.</p><h2>Other Information</h2> <p> Published in: Seminars in Cancer Biology<br> License: <a href="http://creativecommons.org/licenses/by/4.0/" target="_blank">http://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.1016/j.semcancer.2022.07.009" target="_blank">https://dx.doi.org/10.1016/j.semcancer.2022.07.009</a></p>
eu_rights_str_mv openAccess
id Manara2_1ccb5a426967a8181c8c33f71c040341
identifier_str_mv 10.1016/j.semcancer.2022.07.009
network_acronym_str Manara2
network_name_str Manara2
oai_identifier_str oai:figshare.com:article/24717759
publishDate 2022
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rights_invalid_str_mv CC BY 4.0
spelling Dysregulated FOXM1 signaling in the regulation of cancer stem cellsGulab Sher (12984698)Tariq Masoodi (8117702)Kalyani Patil (2679424)Sabah Akhtar (5809925)Shilpa Kuttikrishnan (3520079)Aamir Ahmad (233201)Shahab Uddin (154400)Biomedical and clinical sciencesMedical biotechnologyOncology and carcinogenesisMalignanciesSignaling pathwaysFOXM1Cancer stem cellsSelf-renewalCarcinogenesis<p>Since the introduction of the cancer stem cell (CSC) paradigm, significant advances have been made in understanding the functional and biological plasticity of these elusive components in malignancies. Endowed with self-renewing abilities and multilineage differentiation potential, CSCs have emerged as cellular drivers of virtually all facets of tumor biology, including metastasis, tumor recurrence/relapse, and drug resistance. The functional and biological characteristics of CSCs, such as self-renewal, cell fate decisions, survival, proliferation, and differentiation are regulated by an array of extracellular factors, signaling pathways, and pluripotent transcriptional factors. Besides the well-characterized regulatory role of transcription factors OCT4, SOX2, NANOG, KLF4, and MYC in CSCs, evidence for the central role of Forkhead box transcription factor FOXM1 in the establishment, maintenance, and functions of CSCs is accumulating. Conventionally identified as a master regulator of the cell cycle, a comprehensive understanding of this molecule has revealed its multifarious oncogenic potential and uncovered its role in angiogenesis, invasion, migration, self-renewal, and drug resistance. This review compiles the large body of literature that has accumulated in recent years that provides evidence for the mechanisms by which FOXM1 expression promotes stemness in glioblastoma, breast, colon, ovarian, lung, hepatic, and pancreatic carcinomas. We have also compiled the data showing the association of stem cell mediators with FOXM1 using TCGA mRNA expression data. Further, the prognostic importance of FOXM1 and other stem cell markers is presented. The delineation of FOXM1-mediated regulation of CSCs can aid in the development of molecularly targeted pharmacological approaches directed at the selective eradication of CSCs in several human malignancies.</p><h2>Other Information</h2> <p> Published in: Seminars in Cancer Biology<br> License: <a href="http://creativecommons.org/licenses/by/4.0/" target="_blank">http://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.1016/j.semcancer.2022.07.009" target="_blank">https://dx.doi.org/10.1016/j.semcancer.2022.07.009</a></p>2022-11-01T00:00:00ZTextJournal contributioninfo:eu-repo/semantics/publishedVersiontextcontribution to journal10.1016/j.semcancer.2022.07.009https://figshare.com/articles/journal_contribution/Dysregulated_FOXM1_signaling_in_the_regulation_of_cancer_stem_cells/24717759CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/247177592022-11-01T00:00:00Z
spellingShingle Dysregulated FOXM1 signaling in the regulation of cancer stem cells
Gulab Sher (12984698)
Biomedical and clinical sciences
Medical biotechnology
Oncology and carcinogenesis
Malignancies
Signaling pathways
FOXM1
Cancer stem cells
Self-renewal
Carcinogenesis
status_str publishedVersion
title Dysregulated FOXM1 signaling in the regulation of cancer stem cells
title_full Dysregulated FOXM1 signaling in the regulation of cancer stem cells
title_fullStr Dysregulated FOXM1 signaling in the regulation of cancer stem cells
title_full_unstemmed Dysregulated FOXM1 signaling in the regulation of cancer stem cells
title_short Dysregulated FOXM1 signaling in the regulation of cancer stem cells
title_sort Dysregulated FOXM1 signaling in the regulation of cancer stem cells
topic Biomedical and clinical sciences
Medical biotechnology
Oncology and carcinogenesis
Malignancies
Signaling pathways
FOXM1
Cancer stem cells
Self-renewal
Carcinogenesis