Uhrf1 regulates adipogenesis through GPNMB mediated TGFBeta axis

<p><strong>Poster by Muneera Vakayil (Weill Cornell Medicine - Qatar, Hamad Bin Khalifa University), Aisha Madani (Weill Cornell Medicine - Qatar), Yasser Majeed (Weill Cornell Medicine - Qatar), and Nayef Mazloum (Weill Cornell Medicine - Qatar)</strong></p> <p>Backgro...

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محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: Muneera Vakayil (12037693) (author)
مؤلفون آخرون: Aisha Madani (15430957) (author), Yasser Majeed (8779407) (author), Nayef Mazloum (1275666) (author)
منشور في: 2023
الموضوعات:
الوسوم: إضافة وسم
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author Muneera Vakayil (12037693)
author2 Aisha Madani (15430957)
Yasser Majeed (8779407)
Nayef Mazloum (1275666)
author2_role author
author
author
author_facet Muneera Vakayil (12037693)
Aisha Madani (15430957)
Yasser Majeed (8779407)
Nayef Mazloum (1275666)
author_role author
dc.creator.none.fl_str_mv Muneera Vakayil (12037693)
Aisha Madani (15430957)
Yasser Majeed (8779407)
Nayef Mazloum (1275666)
dc.date.none.fl_str_mv 2023-05-17T12:01:58Z
dc.identifier.none.fl_str_mv 10.57945/manara.22785659.v1
dc.relation.none.fl_str_mv https://figshare.com/articles/poster/Uhrf1_regulates_adipogenesis_through_GPNMB_mediated_TGFBeta_axis/22785659
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Biomedical and clinical sciences
Medical biotechnology
Uhrf1
Adipogenesis
Obesity
Inflammation
Type 2 Diabetes Mellitus
dc.title.none.fl_str_mv Uhrf1 regulates adipogenesis through GPNMB mediated TGFBeta axis
dc.type.none.fl_str_mv Image
Poster
info:eu-repo/semantics/publishedVersion
image
description <p><strong>Poster by Muneera Vakayil (Weill Cornell Medicine - Qatar, Hamad Bin Khalifa University), Aisha Madani (Weill Cornell Medicine - Qatar), Yasser Majeed (Weill Cornell Medicine - Qatar), and Nayef Mazloum (Weill Cornell Medicine - Qatar)</strong></p> <p>Background: The increased prevalence of obesity has led to a concomitant increase in the incidence of type 2 diabetes (T2D). Obesity-linked inflammation is the major cause of insulin resistance, contributing to the onset of T2D. Therefore, understanding the cellular mechanisms responsible for adipogenesis, the conversion of preadipocytes to adipocytes, is critical for identifying anti-diabetic therapeutic targets. Uhrf1 is a key epigenetic regulator that coordinates DNA methylation and histone modifications; however, the function of Uhrf1 in metabolic diseases is not known. </p> <p>Objective: Therefore, here, we aimed to understand the role of Uhrf1 in adipogenesis by studying Uhrf1 loss of function cells.</p> <p>Methods: We performed CRISPR/ Cas9 and shRNA-mediated gene silencing in addition to whole transcriptomics analysis on Uhrf1 KO cells.</p> <p>Results: We identified altered molecular and cellular pathways indicating that Uhrf1 is a critical factor required for adipocyte metabolism, and in its absence, cells exhibit adipocyte dysfunctions with higher levels of inflammatory markers.</p> <p>Conclusion: Our results highlight the possibility of Uhrf1 and its targets to have clinical applications in obesity and T2D treatment.</p>
eu_rights_str_mv openAccess
id Manara2_5ea9d0cdb609cd7e8e66f4a7ce1c9c13
identifier_str_mv 10.57945/manara.22785659.v1
network_acronym_str Manara2
network_name_str Manara2
oai_identifier_str oai:figshare.com:article/22785659
publishDate 2023
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repository.name.fl_str_mv
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rights_invalid_str_mv CC BY 4.0
spelling Uhrf1 regulates adipogenesis through GPNMB mediated TGFBeta axisMuneera Vakayil (12037693)Aisha Madani (15430957)Yasser Majeed (8779407)Nayef Mazloum (1275666)Biomedical and clinical sciencesMedical biotechnologyUhrf1AdipogenesisObesityInflammationType 2 Diabetes Mellitus<p><strong>Poster by Muneera Vakayil (Weill Cornell Medicine - Qatar, Hamad Bin Khalifa University), Aisha Madani (Weill Cornell Medicine - Qatar), Yasser Majeed (Weill Cornell Medicine - Qatar), and Nayef Mazloum (Weill Cornell Medicine - Qatar)</strong></p> <p>Background: The increased prevalence of obesity has led to a concomitant increase in the incidence of type 2 diabetes (T2D). Obesity-linked inflammation is the major cause of insulin resistance, contributing to the onset of T2D. Therefore, understanding the cellular mechanisms responsible for adipogenesis, the conversion of preadipocytes to adipocytes, is critical for identifying anti-diabetic therapeutic targets. Uhrf1 is a key epigenetic regulator that coordinates DNA methylation and histone modifications; however, the function of Uhrf1 in metabolic diseases is not known. </p> <p>Objective: Therefore, here, we aimed to understand the role of Uhrf1 in adipogenesis by studying Uhrf1 loss of function cells.</p> <p>Methods: We performed CRISPR/ Cas9 and shRNA-mediated gene silencing in addition to whole transcriptomics analysis on Uhrf1 KO cells.</p> <p>Results: We identified altered molecular and cellular pathways indicating that Uhrf1 is a critical factor required for adipocyte metabolism, and in its absence, cells exhibit adipocyte dysfunctions with higher levels of inflammatory markers.</p> <p>Conclusion: Our results highlight the possibility of Uhrf1 and its targets to have clinical applications in obesity and T2D treatment.</p>2023-05-17T12:01:58ZImagePosterinfo:eu-repo/semantics/publishedVersionimage10.57945/manara.22785659.v1https://figshare.com/articles/poster/Uhrf1_regulates_adipogenesis_through_GPNMB_mediated_TGFBeta_axis/22785659CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/227856592023-05-17T12:01:58Z
spellingShingle Uhrf1 regulates adipogenesis through GPNMB mediated TGFBeta axis
Muneera Vakayil (12037693)
Biomedical and clinical sciences
Medical biotechnology
Uhrf1
Adipogenesis
Obesity
Inflammation
Type 2 Diabetes Mellitus
status_str publishedVersion
title Uhrf1 regulates adipogenesis through GPNMB mediated TGFBeta axis
title_full Uhrf1 regulates adipogenesis through GPNMB mediated TGFBeta axis
title_fullStr Uhrf1 regulates adipogenesis through GPNMB mediated TGFBeta axis
title_full_unstemmed Uhrf1 regulates adipogenesis through GPNMB mediated TGFBeta axis
title_short Uhrf1 regulates adipogenesis through GPNMB mediated TGFBeta axis
title_sort Uhrf1 regulates adipogenesis through GPNMB mediated TGFBeta axis
topic Biomedical and clinical sciences
Medical biotechnology
Uhrf1
Adipogenesis
Obesity
Inflammation
Type 2 Diabetes Mellitus