Downregulation of <i>CYP17A1</i> by 20-hydroxyecdysone: plasma progesterone and its vasodilatory properties

<p dir="ltr">Aim: To investigate the effect of 20-hydroxyecdysone on steroidogenic pathway genes and plasma progesterone, and its potential impact on vascular functions. Methods: Chimeric mice with humanized liver were treated with 20-hydroxyecdysone for 3 days, and hepatic steroidog...

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محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: Maneera Y Aljaber (18278971) (author)
مؤلفون آخرون: Nelson N Orie (18278974) (author), Asmaa Raees (18278977) (author), Suhail Kraiem (18278980) (author), Mashael Al-Jaber (18278983) (author), Waseem Samsam (12204880) (author), Mostafa M Hamza (18278986) (author), David Abraham (36404) (author), Norman M Kneteman (18278989) (author), Alka Beotra (14780368) (author), Vidya Mohamed-Ali (12204895) (author), Mohammed Almaadheed (18278992) (author)
منشور في: 2022
الموضوعات:
الوسوم: إضافة وسم
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الوصف
الملخص:<p dir="ltr">Aim: To investigate the effect of 20-hydroxyecdysone on steroidogenic pathway genes and plasma progesterone, and its potential impact on vascular functions. Methods: Chimeric mice with humanized liver were treated with 20-hydroxyecdysone for 3 days, and hepatic steroidogenic pathway genes and plasma progesterone were measured by transcriptomics and GC–MS/MS, respectively. Direct effects on muscle and mesenteric arterioles were assessed by myography. Results: CYP17A1 was downregulated in 20-hydroxyecdysone-treated mice compared with untreated group (p = 0.04), with an insignificant increase in plasma progesterone. Progesterone caused vasorelaxation which was blocked by 60 mM KCl, but unaffected by nitric oxide synthase inhibition. Conclusion: In the short term, 20-hydroxyecdysone mediates CYP17A1 downregulation without a significant increase in plasma progesterone, which has a vasodilatory effect involving inhibition of voltage-dependent calcium channels, and the potential to enhance 20-hydroxyecdysone vasorelaxation.</p><h2>Other Information</h2><p dir="ltr">Published in: Future Science OA<br>License: <a href="https://creativecommons.org/licenses/by/4.0/" target="_blank">https://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.2144/fsoa-2022-0006" target="_blank">https://dx.doi.org/10.2144/fsoa-2022-0006</a></p>