Loss of the yeast transporter Agp2 upregulates the pleiotropic drug-resistant pump Pdr5 and confers resistance to the protein synthesis inhibitor cycloheximide

<p dir="ltr">The transmembrane protein Agp2, initially shown as a transporter of L-carnitine, mediates the high-affinity transport of polyamines and the anticancer drug bleomycin-A5. Cells lacking Agp2 are hyper-resistant to polyamine and bleomycin-A5. In these earlier studies, we sh...

وصف كامل

محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: Yusra Manzoor (18599180) (author)
مؤلفون آخرون: Mustapha Aouida (417652) (author), Ramya Ramadoss (16003318) (author), Balasubramanian Moovarkumudalvan (18300811) (author), Nisar Ahmed (584617) (author), Abdallah Alhaj Sulaiman (17777421) (author), Ashima Mohanty (18599183) (author), Reem Ali (9913494) (author), Borbala Mifsud (3907267) (author), Dindial Ramotar (208416) (author)
منشور في: 2024
الموضوعات:
الوسوم: إضافة وسم
لا توجد وسوم, كن أول من يضع وسما على هذه التسجيلة!
_version_ 1864513510668500992
author Yusra Manzoor (18599180)
author2 Mustapha Aouida (417652)
Ramya Ramadoss (16003318)
Balasubramanian Moovarkumudalvan (18300811)
Nisar Ahmed (584617)
Abdallah Alhaj Sulaiman (17777421)
Ashima Mohanty (18599183)
Reem Ali (9913494)
Borbala Mifsud (3907267)
Dindial Ramotar (208416)
author2_role author
author
author
author
author
author
author
author
author
author_facet Yusra Manzoor (18599180)
Mustapha Aouida (417652)
Ramya Ramadoss (16003318)
Balasubramanian Moovarkumudalvan (18300811)
Nisar Ahmed (584617)
Abdallah Alhaj Sulaiman (17777421)
Ashima Mohanty (18599183)
Reem Ali (9913494)
Borbala Mifsud (3907267)
Dindial Ramotar (208416)
author_role author
dc.creator.none.fl_str_mv Yusra Manzoor (18599180)
Mustapha Aouida (417652)
Ramya Ramadoss (16003318)
Balasubramanian Moovarkumudalvan (18300811)
Nisar Ahmed (584617)
Abdallah Alhaj Sulaiman (17777421)
Ashima Mohanty (18599183)
Reem Ali (9913494)
Borbala Mifsud (3907267)
Dindial Ramotar (208416)
dc.date.none.fl_str_mv 2024-05-22T09:00:00Z
dc.identifier.none.fl_str_mv 10.1371/journal.pone.0303747
dc.relation.none.fl_str_mv https://figshare.com/articles/journal_contribution/Loss_of_the_yeast_transporter_Agp2_upregulates_the_pleiotropic_drug-resistant_pump_Pdr5_and_confers_resistance_to_the_protein_synthesis_inhibitor_cycloheximide/26325034
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Biological sciences
Biochemistry and cell biology
Genetics
Biomedical and clinical sciences
Medical biochemistry and metabolomics
Mitochondria
Gene expression
Membrane proteins
Protein expression
Cell membranes
Yeast
Saccharomyces cerevisiae
RNA analysis
dc.title.none.fl_str_mv Loss of the yeast transporter Agp2 upregulates the pleiotropic drug-resistant pump Pdr5 and confers resistance to the protein synthesis inhibitor cycloheximide
dc.type.none.fl_str_mv Text
Journal contribution
info:eu-repo/semantics/publishedVersion
text
contribution to journal
description <p dir="ltr">The transmembrane protein Agp2, initially shown as a transporter of L-carnitine, mediates the high-affinity transport of polyamines and the anticancer drug bleomycin-A5. Cells lacking Agp2 are hyper-resistant to polyamine and bleomycin-A5. In these earlier studies, we showed that the protein synthesis inhibitor cycloheximide blocked the uptake of bleomycin-A5 into the cells suggesting that the drug uptake system may require de <i>novo</i> synthesis. However, our recent findings demonstrated that cycloheximide, instead, induced rapid degradation of Agp2, and in the absence of Agp2 cells are resistant to cycloheximide. These observations raised the possibility that the degradation of Agp2 may allow the cell to alter its drug resistance network to combat the toxic effects of cycloheximide. In this study, we show that membrane extracts from<i> </i>agp2Δ mutants accentuated several proteins that were differentially expressed in comparison to the parent. Mass spectrometry analysis of the membrane extracts uncovered the pleiotropic drug efflux pump, Pdr5, involved in the efflux of cycloheximide, as a key protein upregulated in the <i>agp2Δ</i> mutant. Moreover, a global gene expression analysis revealed that 322 genes were differentially affected in the <i>agp2Δ</i> mutant versus the parent, including the prominent <i>PDR5</i> gene and genes required for mitochondrial function. We further show that <i>Agp2</i> is associated with the upstream region of the<i> PDR5</i> gene, leading to the hypothesis that cycloheximide resistance displayed by the <i>agp2Δ</i> mutant is due to the derepression of the <i>PDR5</i> gene.</p><h2>Other Information</h2><p dir="ltr">Published in: PLOS ONE<br>License: <a href="http://creativecommons.org/licenses/by/4.0/" target="_blank">http://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.1371/journal.pone.0303747" target="_blank">https://dx.doi.org/10.1371/journal.pone.0303747</a></p>
eu_rights_str_mv openAccess
id Manara2_a88e72d616a209b26e516b2dbf8a44db
identifier_str_mv 10.1371/journal.pone.0303747
network_acronym_str Manara2
network_name_str Manara2
oai_identifier_str oai:figshare.com:article/26325034
publishDate 2024
repository.mail.fl_str_mv
repository.name.fl_str_mv
repository_id_str
rights_invalid_str_mv CC BY 4.0
spelling Loss of the yeast transporter Agp2 upregulates the pleiotropic drug-resistant pump Pdr5 and confers resistance to the protein synthesis inhibitor cycloheximideYusra Manzoor (18599180)Mustapha Aouida (417652)Ramya Ramadoss (16003318)Balasubramanian Moovarkumudalvan (18300811)Nisar Ahmed (584617)Abdallah Alhaj Sulaiman (17777421)Ashima Mohanty (18599183)Reem Ali (9913494)Borbala Mifsud (3907267)Dindial Ramotar (208416)Biological sciencesBiochemistry and cell biologyGeneticsBiomedical and clinical sciencesMedical biochemistry and metabolomicsMitochondriaGene expressionMembrane proteinsProtein expressionCell membranesYeastSaccharomyces cerevisiaeRNA analysis<p dir="ltr">The transmembrane protein Agp2, initially shown as a transporter of L-carnitine, mediates the high-affinity transport of polyamines and the anticancer drug bleomycin-A5. Cells lacking Agp2 are hyper-resistant to polyamine and bleomycin-A5. In these earlier studies, we showed that the protein synthesis inhibitor cycloheximide blocked the uptake of bleomycin-A5 into the cells suggesting that the drug uptake system may require de <i>novo</i> synthesis. However, our recent findings demonstrated that cycloheximide, instead, induced rapid degradation of Agp2, and in the absence of Agp2 cells are resistant to cycloheximide. These observations raised the possibility that the degradation of Agp2 may allow the cell to alter its drug resistance network to combat the toxic effects of cycloheximide. In this study, we show that membrane extracts from<i> </i>agp2Δ mutants accentuated several proteins that were differentially expressed in comparison to the parent. Mass spectrometry analysis of the membrane extracts uncovered the pleiotropic drug efflux pump, Pdr5, involved in the efflux of cycloheximide, as a key protein upregulated in the <i>agp2Δ</i> mutant. Moreover, a global gene expression analysis revealed that 322 genes were differentially affected in the <i>agp2Δ</i> mutant versus the parent, including the prominent <i>PDR5</i> gene and genes required for mitochondrial function. We further show that <i>Agp2</i> is associated with the upstream region of the<i> PDR5</i> gene, leading to the hypothesis that cycloheximide resistance displayed by the <i>agp2Δ</i> mutant is due to the derepression of the <i>PDR5</i> gene.</p><h2>Other Information</h2><p dir="ltr">Published in: PLOS ONE<br>License: <a href="http://creativecommons.org/licenses/by/4.0/" target="_blank">http://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.1371/journal.pone.0303747" target="_blank">https://dx.doi.org/10.1371/journal.pone.0303747</a></p>2024-05-22T09:00:00ZTextJournal contributioninfo:eu-repo/semantics/publishedVersiontextcontribution to journal10.1371/journal.pone.0303747https://figshare.com/articles/journal_contribution/Loss_of_the_yeast_transporter_Agp2_upregulates_the_pleiotropic_drug-resistant_pump_Pdr5_and_confers_resistance_to_the_protein_synthesis_inhibitor_cycloheximide/26325034CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/263250342024-05-22T09:00:00Z
spellingShingle Loss of the yeast transporter Agp2 upregulates the pleiotropic drug-resistant pump Pdr5 and confers resistance to the protein synthesis inhibitor cycloheximide
Yusra Manzoor (18599180)
Biological sciences
Biochemistry and cell biology
Genetics
Biomedical and clinical sciences
Medical biochemistry and metabolomics
Mitochondria
Gene expression
Membrane proteins
Protein expression
Cell membranes
Yeast
Saccharomyces cerevisiae
RNA analysis
status_str publishedVersion
title Loss of the yeast transporter Agp2 upregulates the pleiotropic drug-resistant pump Pdr5 and confers resistance to the protein synthesis inhibitor cycloheximide
title_full Loss of the yeast transporter Agp2 upregulates the pleiotropic drug-resistant pump Pdr5 and confers resistance to the protein synthesis inhibitor cycloheximide
title_fullStr Loss of the yeast transporter Agp2 upregulates the pleiotropic drug-resistant pump Pdr5 and confers resistance to the protein synthesis inhibitor cycloheximide
title_full_unstemmed Loss of the yeast transporter Agp2 upregulates the pleiotropic drug-resistant pump Pdr5 and confers resistance to the protein synthesis inhibitor cycloheximide
title_short Loss of the yeast transporter Agp2 upregulates the pleiotropic drug-resistant pump Pdr5 and confers resistance to the protein synthesis inhibitor cycloheximide
title_sort Loss of the yeast transporter Agp2 upregulates the pleiotropic drug-resistant pump Pdr5 and confers resistance to the protein synthesis inhibitor cycloheximide
topic Biological sciences
Biochemistry and cell biology
Genetics
Biomedical and clinical sciences
Medical biochemistry and metabolomics
Mitochondria
Gene expression
Membrane proteins
Protein expression
Cell membranes
Yeast
Saccharomyces cerevisiae
RNA analysis