Neoplastic Transformation of Human Mesenchymal Stromal Cells Mediated via <i>LIN28B</i>

<p dir="ltr">Bone marrow stromal (Mesenchymal) stem cells (MSCs) are multipotent bone cells capable of differentiating into mesoderm-type cells, such as osteoblasts and adipocytes. Existing evidence suggests that transformation of MSCs gives rise to sarcoma. In order to identify the...

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Main Author: Radhakrishnan Vishnubalaji (3563306) (author)
Other Authors: Ramesh Elango (7542068) (author), Mashael Al-Toub (14152764) (author), Muthurangan Manikandan (6002243) (author), Ammar Al-Rikabi (5213753) (author), Linda Harkness (101821) (author), Nicholas Ditzel (127179) (author), Muhammad Atteya (6002246) (author), Rimi Hamam (3563312) (author), Musaad Alfayez (3571736) (author), Abdullah Aldahmash (3563300) (author), Moustapha Kassem (101848) (author), Nehad M. Alajez (7397276) (author)
Published: 2019
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author Radhakrishnan Vishnubalaji (3563306)
author2 Ramesh Elango (7542068)
Mashael Al-Toub (14152764)
Muthurangan Manikandan (6002243)
Ammar Al-Rikabi (5213753)
Linda Harkness (101821)
Nicholas Ditzel (127179)
Muhammad Atteya (6002246)
Rimi Hamam (3563312)
Musaad Alfayez (3571736)
Abdullah Aldahmash (3563300)
Moustapha Kassem (101848)
Nehad M. Alajez (7397276)
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author_facet Radhakrishnan Vishnubalaji (3563306)
Ramesh Elango (7542068)
Mashael Al-Toub (14152764)
Muthurangan Manikandan (6002243)
Ammar Al-Rikabi (5213753)
Linda Harkness (101821)
Nicholas Ditzel (127179)
Muhammad Atteya (6002246)
Rimi Hamam (3563312)
Musaad Alfayez (3571736)
Abdullah Aldahmash (3563300)
Moustapha Kassem (101848)
Nehad M. Alajez (7397276)
author_role author
dc.creator.none.fl_str_mv Radhakrishnan Vishnubalaji (3563306)
Ramesh Elango (7542068)
Mashael Al-Toub (14152764)
Muthurangan Manikandan (6002243)
Ammar Al-Rikabi (5213753)
Linda Harkness (101821)
Nicholas Ditzel (127179)
Muhammad Atteya (6002246)
Rimi Hamam (3563312)
Musaad Alfayez (3571736)
Abdullah Aldahmash (3563300)
Moustapha Kassem (101848)
Nehad M. Alajez (7397276)
dc.date.none.fl_str_mv 2019-05-30T00:00:00Z
dc.identifier.none.fl_str_mv 10.1038/s41598-019-44536-1
dc.relation.none.fl_str_mv https://figshare.com/articles/journal_contribution/Neoplastic_Transformation_of_Human_Mesenchymal_Stromal_Cells_Mediated_via_LIN28B/21598119
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Biomedical and clinical sciences
Clinical sciences
Oncology and carcinogenesis
Bone Marrow Stromal Cells
Sarcoma
Cell Transformation
Tumorigenesis
Colony Formation
Cancer Therapeutics
dc.title.none.fl_str_mv Neoplastic Transformation of Human Mesenchymal Stromal Cells Mediated via <i>LIN28B</i>
dc.type.none.fl_str_mv Text
Journal contribution
info:eu-repo/semantics/publishedVersion
text
contribution to journal
description <p dir="ltr">Bone marrow stromal (Mesenchymal) stem cells (MSCs) are multipotent bone cells capable of differentiating into mesoderm-type cells, such as osteoblasts and adipocytes. Existing evidence suggests that transformation of MSCs gives rise to sarcoma. In order to identify the molecular mechanism leading to spontaneous transformation of human bone marrow MSCs (hBMSCs), we performed comprehensive microRNA (miRNA) and mRNA profiling in the transformed hBMSC-Tum line compared to the parental clone. As a result, we identified multiple dysregulated molecular networks associated with the hBMSC transformed phenotype. <i>LIN28B</i> was upregulated 177.0-fold in hBMSC-Tum, which was associated with marked reduction in <i>LET-7</i> expression and upregulated expression of its target <i>HMGA2</i>. Targeted depletion of <i>LIN28B</i> or exogenous expression of <i>LET-7</i>b suppressed hBMSC-Tum proliferation, colony formation, and migration. On the other hand, forced expression of LIN28B promoted malignant transformation of parental hBMSC cells as shown by enhanced in vitro colony formation, doxorubicin resistance, and in vivo tumor formation in immunocompromised mice. Analysis of <i>LIN28B</i> and <i>HMGA2 </i>expression levels in cohorts from The Cancer Genome Atlas sarcoma dataset revealed a strong inverse-relationship between elevated expression and overall survival (OS) in 260 patients (p = 0.005) and disease-free survival (DFS) in 231 patients (p = 0.02), suggesting <i>LIN28B</i> and <i>HMGA2</i> are important regulators of sarcoma biology. Our results highlight an important role for the <i>LIN28B/LET-7</i> axis in human sarcoma pathogenesis and suggest that the therapeutic targeting of <i>LIN28B</i> may be relevant for patients with sarcoma.</p><h2>Other Information</h2><p dir="ltr">Published in: Scientific Reports<br>License: <a href="https://creativecommons.org/licenses/by/4.0" target="_blank">https://creativecommons.org/licenses/by/4.0</a><br>See article on publisher's website: <a href="http://dx.doi.org/10.1038/s41598-019-44536-1" target="_blank">http://dx.doi.org/10.1038/s41598-019-44536-1</a></p>
eu_rights_str_mv openAccess
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identifier_str_mv 10.1038/s41598-019-44536-1
network_acronym_str Manara2
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spelling Neoplastic Transformation of Human Mesenchymal Stromal Cells Mediated via <i>LIN28B</i>Radhakrishnan Vishnubalaji (3563306)Ramesh Elango (7542068)Mashael Al-Toub (14152764)Muthurangan Manikandan (6002243)Ammar Al-Rikabi (5213753)Linda Harkness (101821)Nicholas Ditzel (127179)Muhammad Atteya (6002246)Rimi Hamam (3563312)Musaad Alfayez (3571736)Abdullah Aldahmash (3563300)Moustapha Kassem (101848)Nehad M. Alajez (7397276)Biomedical and clinical sciencesClinical sciencesOncology and carcinogenesisBone Marrow Stromal CellsSarcomaCell TransformationTumorigenesisColony FormationCancer Therapeutics<p dir="ltr">Bone marrow stromal (Mesenchymal) stem cells (MSCs) are multipotent bone cells capable of differentiating into mesoderm-type cells, such as osteoblasts and adipocytes. Existing evidence suggests that transformation of MSCs gives rise to sarcoma. In order to identify the molecular mechanism leading to spontaneous transformation of human bone marrow MSCs (hBMSCs), we performed comprehensive microRNA (miRNA) and mRNA profiling in the transformed hBMSC-Tum line compared to the parental clone. As a result, we identified multiple dysregulated molecular networks associated with the hBMSC transformed phenotype. <i>LIN28B</i> was upregulated 177.0-fold in hBMSC-Tum, which was associated with marked reduction in <i>LET-7</i> expression and upregulated expression of its target <i>HMGA2</i>. Targeted depletion of <i>LIN28B</i> or exogenous expression of <i>LET-7</i>b suppressed hBMSC-Tum proliferation, colony formation, and migration. On the other hand, forced expression of LIN28B promoted malignant transformation of parental hBMSC cells as shown by enhanced in vitro colony formation, doxorubicin resistance, and in vivo tumor formation in immunocompromised mice. Analysis of <i>LIN28B</i> and <i>HMGA2 </i>expression levels in cohorts from The Cancer Genome Atlas sarcoma dataset revealed a strong inverse-relationship between elevated expression and overall survival (OS) in 260 patients (p = 0.005) and disease-free survival (DFS) in 231 patients (p = 0.02), suggesting <i>LIN28B</i> and <i>HMGA2</i> are important regulators of sarcoma biology. Our results highlight an important role for the <i>LIN28B/LET-7</i> axis in human sarcoma pathogenesis and suggest that the therapeutic targeting of <i>LIN28B</i> may be relevant for patients with sarcoma.</p><h2>Other Information</h2><p dir="ltr">Published in: Scientific Reports<br>License: <a href="https://creativecommons.org/licenses/by/4.0" target="_blank">https://creativecommons.org/licenses/by/4.0</a><br>See article on publisher's website: <a href="http://dx.doi.org/10.1038/s41598-019-44536-1" target="_blank">http://dx.doi.org/10.1038/s41598-019-44536-1</a></p>2019-05-30T00:00:00ZTextJournal contributioninfo:eu-repo/semantics/publishedVersiontextcontribution to journal10.1038/s41598-019-44536-1https://figshare.com/articles/journal_contribution/Neoplastic_Transformation_of_Human_Mesenchymal_Stromal_Cells_Mediated_via_LIN28B/21598119CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/215981192019-05-30T00:00:00Z
spellingShingle Neoplastic Transformation of Human Mesenchymal Stromal Cells Mediated via <i>LIN28B</i>
Radhakrishnan Vishnubalaji (3563306)
Biomedical and clinical sciences
Clinical sciences
Oncology and carcinogenesis
Bone Marrow Stromal Cells
Sarcoma
Cell Transformation
Tumorigenesis
Colony Formation
Cancer Therapeutics
status_str publishedVersion
title Neoplastic Transformation of Human Mesenchymal Stromal Cells Mediated via <i>LIN28B</i>
title_full Neoplastic Transformation of Human Mesenchymal Stromal Cells Mediated via <i>LIN28B</i>
title_fullStr Neoplastic Transformation of Human Mesenchymal Stromal Cells Mediated via <i>LIN28B</i>
title_full_unstemmed Neoplastic Transformation of Human Mesenchymal Stromal Cells Mediated via <i>LIN28B</i>
title_short Neoplastic Transformation of Human Mesenchymal Stromal Cells Mediated via <i>LIN28B</i>
title_sort Neoplastic Transformation of Human Mesenchymal Stromal Cells Mediated via <i>LIN28B</i>
topic Biomedical and clinical sciences
Clinical sciences
Oncology and carcinogenesis
Bone Marrow Stromal Cells
Sarcoma
Cell Transformation
Tumorigenesis
Colony Formation
Cancer Therapeutics