Prenatal psychological distress and 11β-HSD2 gene expression in human placentas: Systematic review and meta-analysis

<h3>Background</h3><p dir="ltr">The placenta acts as a buffer to regulate the degree of fetal exposure to maternal cortisol through the 11-Beta Hydroxysteroid Dehydrogenase isoenzyme type 2 (<i>11-β HSD2</i>) enzyme. We conducted a systematic review and meta-a...

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محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: Angham Ibrahim Tartour (19756953) (author)
مؤلفون آخرون: Tawanda Chivese (801864) (author), Safa Eltayeb (19756956) (author), Fatima M. Elamin (19756959) (author), Eleni Fthenou (216980) (author), Mohammed Seed Ahmed (798933) (author), Giridhara Rathnaiah Babu (17933762) (author)
منشور في: 2024
الموضوعات:
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author Angham Ibrahim Tartour (19756953)
author2 Tawanda Chivese (801864)
Safa Eltayeb (19756956)
Fatima M. Elamin (19756959)
Eleni Fthenou (216980)
Mohammed Seed Ahmed (798933)
Giridhara Rathnaiah Babu (17933762)
author2_role author
author
author
author
author
author
author_facet Angham Ibrahim Tartour (19756953)
Tawanda Chivese (801864)
Safa Eltayeb (19756956)
Fatima M. Elamin (19756959)
Eleni Fthenou (216980)
Mohammed Seed Ahmed (798933)
Giridhara Rathnaiah Babu (17933762)
author_role author
dc.creator.none.fl_str_mv Angham Ibrahim Tartour (19756953)
Tawanda Chivese (801864)
Safa Eltayeb (19756956)
Fatima M. Elamin (19756959)
Eleni Fthenou (216980)
Mohammed Seed Ahmed (798933)
Giridhara Rathnaiah Babu (17933762)
dc.date.none.fl_str_mv 2024-08-01T00:00:00Z
dc.identifier.none.fl_str_mv 10.1016/j.psyneuen.2024.107060
dc.relation.none.fl_str_mv https://figshare.com/articles/journal_contribution/Prenatal_psychological_distress_and_11_-HSD2_gene_expression_in_human_placentas_Systematic_review_and_meta-analysis/27130053
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Biological sciences
Genetics
Biomedical and clinical sciences
Clinical sciences
Neurosciences
Reproductive medicine
Health sciences
Public health
Prenatal psychological distress
11β-Hydroxysteroid Dehydrogenase Type 2
Gene expression
Fetal reprogramming
Placenta
Early-life exposures
dc.title.none.fl_str_mv Prenatal psychological distress and 11β-HSD2 gene expression in human placentas: Systematic review and meta-analysis
dc.type.none.fl_str_mv Text
Journal contribution
info:eu-repo/semantics/publishedVersion
text
contribution to journal
description <h3>Background</h3><p dir="ltr">The placenta acts as a buffer to regulate the degree of fetal exposure to maternal cortisol through the 11-Beta Hydroxysteroid Dehydrogenase isoenzyme type 2 (<i>11-β HSD2</i>) enzyme. We conducted a systematic review and meta-analysis to assess the effect of prenatal psychological distress (PPD) on placental <i>11-β HSD2</i> gene expression and explore the related mechanistic pathways involved in fetal neurodevelopment.</p><h3>Methods</h3><p dir="ltr">We searched PubMed, Embase, Scopus, APA PsycInfo®, and ProQuest Dissertations for observational studies assessing the association between PPD and <i>11-β HSD2</i> expression in human placentas. Adjusted regression coefficients (β) and corresponding 95% confidence intervals (CIs) were pooled based on three contextual PPD exposure groups: prenatal depression, anxiety symptoms, and perceived stress.</p><h3>Results</h3><p dir="ltr">Of 3159 retrieved records, sixteen longitudinal studies involving 1869 participants across seven countries were included. Overall, exposure to PPD disorders showed weak negative associations with the placental <i>11-β HSD2</i> gene expression as follows: prenatal depression (β −0.01, 95% CI 0.05–0.02, I2=0%), anxiety symptoms (β −0.02, 95% CI 0.06–0.01, I2=0%), and perceived stress (β −0.01 95% CI 0.06–0.04, I2=62.8%). Third-trimester PPD exposure was more frequently associated with lower placental <i>11-β HSD2</i> levels. PPD and placental <i>11-β HSD2</i> were associated with changes in cortisol reactivity and the development of adverse health outcomes in mothers and children. Female-offspring were more vulnerable to PPD exposures.</p><h3>Conclusion</h3><p dir="ltr">The study presents evidence of a modest role of prenatal psychological distress in regulating placental 11-β HSD2 gene expression. Future prospective cohorts utilizing larger sample sizes or advanced statistical methods to enhance the detection of small effect sizes should be planned. Additionally, controlling for key predictors such as the mother’s ethnicity, trimester of PPD exposure, mode of delivery, and infant sex is crucial for valid exploration of PPD effects on fetal programming.</p><h2>Other Information</h2><p dir="ltr">Published in: Psychoneuroendocrinology<br>License: <a href="http://creativecommons.org/licenses/by/4.0/" target="_blank">http://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.1016/j.psyneuen.2024.107060" target="_blank">https://dx.doi.org/10.1016/j.psyneuen.2024.107060</a></p>
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spelling Prenatal psychological distress and 11β-HSD2 gene expression in human placentas: Systematic review and meta-analysisAngham Ibrahim Tartour (19756953)Tawanda Chivese (801864)Safa Eltayeb (19756956)Fatima M. Elamin (19756959)Eleni Fthenou (216980)Mohammed Seed Ahmed (798933)Giridhara Rathnaiah Babu (17933762)Biological sciencesGeneticsBiomedical and clinical sciencesClinical sciencesNeurosciencesReproductive medicineHealth sciencesPublic healthPrenatal psychological distress11β-Hydroxysteroid Dehydrogenase Type 2Gene expressionFetal reprogrammingPlacentaEarly-life exposures<h3>Background</h3><p dir="ltr">The placenta acts as a buffer to regulate the degree of fetal exposure to maternal cortisol through the 11-Beta Hydroxysteroid Dehydrogenase isoenzyme type 2 (<i>11-β HSD2</i>) enzyme. We conducted a systematic review and meta-analysis to assess the effect of prenatal psychological distress (PPD) on placental <i>11-β HSD2</i> gene expression and explore the related mechanistic pathways involved in fetal neurodevelopment.</p><h3>Methods</h3><p dir="ltr">We searched PubMed, Embase, Scopus, APA PsycInfo®, and ProQuest Dissertations for observational studies assessing the association between PPD and <i>11-β HSD2</i> expression in human placentas. Adjusted regression coefficients (β) and corresponding 95% confidence intervals (CIs) were pooled based on three contextual PPD exposure groups: prenatal depression, anxiety symptoms, and perceived stress.</p><h3>Results</h3><p dir="ltr">Of 3159 retrieved records, sixteen longitudinal studies involving 1869 participants across seven countries were included. Overall, exposure to PPD disorders showed weak negative associations with the placental <i>11-β HSD2</i> gene expression as follows: prenatal depression (β −0.01, 95% CI 0.05–0.02, I2=0%), anxiety symptoms (β −0.02, 95% CI 0.06–0.01, I2=0%), and perceived stress (β −0.01 95% CI 0.06–0.04, I2=62.8%). Third-trimester PPD exposure was more frequently associated with lower placental <i>11-β HSD2</i> levels. PPD and placental <i>11-β HSD2</i> were associated with changes in cortisol reactivity and the development of adverse health outcomes in mothers and children. Female-offspring were more vulnerable to PPD exposures.</p><h3>Conclusion</h3><p dir="ltr">The study presents evidence of a modest role of prenatal psychological distress in regulating placental 11-β HSD2 gene expression. Future prospective cohorts utilizing larger sample sizes or advanced statistical methods to enhance the detection of small effect sizes should be planned. Additionally, controlling for key predictors such as the mother’s ethnicity, trimester of PPD exposure, mode of delivery, and infant sex is crucial for valid exploration of PPD effects on fetal programming.</p><h2>Other Information</h2><p dir="ltr">Published in: Psychoneuroendocrinology<br>License: <a href="http://creativecommons.org/licenses/by/4.0/" target="_blank">http://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.1016/j.psyneuen.2024.107060" target="_blank">https://dx.doi.org/10.1016/j.psyneuen.2024.107060</a></p>2024-08-01T00:00:00ZTextJournal contributioninfo:eu-repo/semantics/publishedVersiontextcontribution to journal10.1016/j.psyneuen.2024.107060https://figshare.com/articles/journal_contribution/Prenatal_psychological_distress_and_11_-HSD2_gene_expression_in_human_placentas_Systematic_review_and_meta-analysis/27130053CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/271300532024-08-01T00:00:00Z
spellingShingle Prenatal psychological distress and 11β-HSD2 gene expression in human placentas: Systematic review and meta-analysis
Angham Ibrahim Tartour (19756953)
Biological sciences
Genetics
Biomedical and clinical sciences
Clinical sciences
Neurosciences
Reproductive medicine
Health sciences
Public health
Prenatal psychological distress
11β-Hydroxysteroid Dehydrogenase Type 2
Gene expression
Fetal reprogramming
Placenta
Early-life exposures
status_str publishedVersion
title Prenatal psychological distress and 11β-HSD2 gene expression in human placentas: Systematic review and meta-analysis
title_full Prenatal psychological distress and 11β-HSD2 gene expression in human placentas: Systematic review and meta-analysis
title_fullStr Prenatal psychological distress and 11β-HSD2 gene expression in human placentas: Systematic review and meta-analysis
title_full_unstemmed Prenatal psychological distress and 11β-HSD2 gene expression in human placentas: Systematic review and meta-analysis
title_short Prenatal psychological distress and 11β-HSD2 gene expression in human placentas: Systematic review and meta-analysis
title_sort Prenatal psychological distress and 11β-HSD2 gene expression in human placentas: Systematic review and meta-analysis
topic Biological sciences
Genetics
Biomedical and clinical sciences
Clinical sciences
Neurosciences
Reproductive medicine
Health sciences
Public health
Prenatal psychological distress
11β-Hydroxysteroid Dehydrogenase Type 2
Gene expression
Fetal reprogramming
Placenta
Early-life exposures