β-cell mitochondria in diabetes mellitus: a missing puzzle piece in the generation of hPSC-derived pancreatic β-cells?

<div><p>Diabetes mellitus (DM), currently affecting 463 million people worldwide is a chronic disease characterized by impaired glucose metabolism resulting from the loss or dysfunction of pancreatic β-cells with the former preponderating in type 1 diabetes (T1DM) and the latter in type...

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Main Author: Abdoulaye Diane (14152749) (author)
Other Authors: Noora Ali Al-Shukri (18372429) (author), Razik Bin Abdul Mu-u-min (18372432) (author), Heba H. Al-Siddiqi (17733783) (author)
Published: 2022
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_version_ 1864513519231172608
author Abdoulaye Diane (14152749)
author2 Noora Ali Al-Shukri (18372429)
Razik Bin Abdul Mu-u-min (18372432)
Heba H. Al-Siddiqi (17733783)
author2_role author
author
author
author_facet Abdoulaye Diane (14152749)
Noora Ali Al-Shukri (18372429)
Razik Bin Abdul Mu-u-min (18372432)
Heba H. Al-Siddiqi (17733783)
author_role author
dc.creator.none.fl_str_mv Abdoulaye Diane (14152749)
Noora Ali Al-Shukri (18372429)
Razik Bin Abdul Mu-u-min (18372432)
Heba H. Al-Siddiqi (17733783)
dc.date.none.fl_str_mv 2022-04-09T03:00:00Z
dc.identifier.none.fl_str_mv 10.1186/s12967-022-03327-5
dc.relation.none.fl_str_mv https://figshare.com/articles/journal_contribution/_-cell_mitochondria_in_diabetes_mellitus_a_missing_puzzle_piece_in_the_generation_of_hPSC-derived_pancreatic_-cells_/25609440
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Biological sciences
Biochemistry and cell biology
Genetics
β-cell
Pancreas
Mitochondria
Diabetes mellitus
hPSC-derived β-cells
Differentiation
Glucose-stimulated insulin secretion (GSIS)
dc.title.none.fl_str_mv β-cell mitochondria in diabetes mellitus: a missing puzzle piece in the generation of hPSC-derived pancreatic β-cells?
dc.type.none.fl_str_mv Text
Journal contribution
info:eu-repo/semantics/publishedVersion
text
contribution to journal
description <div><p>Diabetes mellitus (DM), currently affecting 463 million people worldwide is a chronic disease characterized by impaired glucose metabolism resulting from the loss or dysfunction of pancreatic β-cells with the former preponderating in type 1 diabetes (T1DM) and the latter in type 2 diabetes (T2DM). Because impaired insulin secretion due to dysfunction or loss of pancreatic β-cells underlies different types of diabetes, research has focused its effort towards the generation of pancreatic β-cells from human pluripotent stem cell (hPSC) as a potential source of cells to compensate for insulin deficiency. However, many protocols developed to differentiate hPSCs into insulin-expressing β-cells in vitro have generated hPSC-derived β-cells with either immature phenotype such as impaired glucose-stimulated insulin secretion (GSIS) or a weaker response to GSIS than cadaveric islets. In pancreatic β-cells, mitochondria play a central role in coupling glucose metabolism to insulin exocytosis, thereby ensuring refined control of GSIS. Defects in β-cell mitochondrial metabolism and function impair this metabolic coupling. In the present review, we highlight the role of mitochondria in metabolism secretion coupling in the β-cells and summarize the evidence accumulated for the implication of mitochondria in β-cell dysfunction in DM and consequently, how targeting mitochondria function might be a new and interesting strategy to further perfect the differentiation protocol for generation of mature and functional hPSC-derived β-cells with GSIS profile similar to human cadaveric islets for drug screening or potentially for cell therapy.</p><p> </p></div><h2>Other Information</h2> <p> Published in: Journal of Translational Medicine<br> License: <a href="https://creativecommons.org/licenses/by/4.0" target="_blank">https://creativecommons.org/licenses/by/4.0</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.1186/s12967-022-03327-5" target="_blank">https://dx.doi.org/10.1186/s12967-022-03327-5</a></p>
eu_rights_str_mv openAccess
id Manara2_cb6e727a75c422a0ef3832290d7bc2c6
identifier_str_mv 10.1186/s12967-022-03327-5
network_acronym_str Manara2
network_name_str Manara2
oai_identifier_str oai:figshare.com:article/25609440
publishDate 2022
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rights_invalid_str_mv CC BY 4.0
spelling β-cell mitochondria in diabetes mellitus: a missing puzzle piece in the generation of hPSC-derived pancreatic β-cells?Abdoulaye Diane (14152749)Noora Ali Al-Shukri (18372429)Razik Bin Abdul Mu-u-min (18372432)Heba H. Al-Siddiqi (17733783)Biological sciencesBiochemistry and cell biologyGeneticsβ-cellPancreasMitochondriaDiabetes mellitushPSC-derived β-cellsDifferentiationGlucose-stimulated insulin secretion (GSIS)<div><p>Diabetes mellitus (DM), currently affecting 463 million people worldwide is a chronic disease characterized by impaired glucose metabolism resulting from the loss or dysfunction of pancreatic β-cells with the former preponderating in type 1 diabetes (T1DM) and the latter in type 2 diabetes (T2DM). Because impaired insulin secretion due to dysfunction or loss of pancreatic β-cells underlies different types of diabetes, research has focused its effort towards the generation of pancreatic β-cells from human pluripotent stem cell (hPSC) as a potential source of cells to compensate for insulin deficiency. However, many protocols developed to differentiate hPSCs into insulin-expressing β-cells in vitro have generated hPSC-derived β-cells with either immature phenotype such as impaired glucose-stimulated insulin secretion (GSIS) or a weaker response to GSIS than cadaveric islets. In pancreatic β-cells, mitochondria play a central role in coupling glucose metabolism to insulin exocytosis, thereby ensuring refined control of GSIS. Defects in β-cell mitochondrial metabolism and function impair this metabolic coupling. In the present review, we highlight the role of mitochondria in metabolism secretion coupling in the β-cells and summarize the evidence accumulated for the implication of mitochondria in β-cell dysfunction in DM and consequently, how targeting mitochondria function might be a new and interesting strategy to further perfect the differentiation protocol for generation of mature and functional hPSC-derived β-cells with GSIS profile similar to human cadaveric islets for drug screening or potentially for cell therapy.</p><p> </p></div><h2>Other Information</h2> <p> Published in: Journal of Translational Medicine<br> License: <a href="https://creativecommons.org/licenses/by/4.0" target="_blank">https://creativecommons.org/licenses/by/4.0</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.1186/s12967-022-03327-5" target="_blank">https://dx.doi.org/10.1186/s12967-022-03327-5</a></p>2022-04-09T03:00:00ZTextJournal contributioninfo:eu-repo/semantics/publishedVersiontextcontribution to journal10.1186/s12967-022-03327-5https://figshare.com/articles/journal_contribution/_-cell_mitochondria_in_diabetes_mellitus_a_missing_puzzle_piece_in_the_generation_of_hPSC-derived_pancreatic_-cells_/25609440CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/256094402022-04-09T03:00:00Z
spellingShingle β-cell mitochondria in diabetes mellitus: a missing puzzle piece in the generation of hPSC-derived pancreatic β-cells?
Abdoulaye Diane (14152749)
Biological sciences
Biochemistry and cell biology
Genetics
β-cell
Pancreas
Mitochondria
Diabetes mellitus
hPSC-derived β-cells
Differentiation
Glucose-stimulated insulin secretion (GSIS)
status_str publishedVersion
title β-cell mitochondria in diabetes mellitus: a missing puzzle piece in the generation of hPSC-derived pancreatic β-cells?
title_full β-cell mitochondria in diabetes mellitus: a missing puzzle piece in the generation of hPSC-derived pancreatic β-cells?
title_fullStr β-cell mitochondria in diabetes mellitus: a missing puzzle piece in the generation of hPSC-derived pancreatic β-cells?
title_full_unstemmed β-cell mitochondria in diabetes mellitus: a missing puzzle piece in the generation of hPSC-derived pancreatic β-cells?
title_short β-cell mitochondria in diabetes mellitus: a missing puzzle piece in the generation of hPSC-derived pancreatic β-cells?
title_sort β-cell mitochondria in diabetes mellitus: a missing puzzle piece in the generation of hPSC-derived pancreatic β-cells?
topic Biological sciences
Biochemistry and cell biology
Genetics
β-cell
Pancreas
Mitochondria
Diabetes mellitus
hPSC-derived β-cells
Differentiation
Glucose-stimulated insulin secretion (GSIS)