Novel thiazolones for the simultaneous modulation of PPARγ, COX-2 and 15-LOX to address metabolic disease-associated portal inflammation

<p dir="ltr">A hybrid pharmacophore model, based on structural motifs previously identified by our team, was employed to generate ligands that simultaneously target COX-2, 15-LOX, and PPARγ in the context of metabolic dysfunction-associated fatty liver disease (MAFLD). Notable COX-2...

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محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: Mai S. El-Shoukrofy (19646815) (author)
مؤلفون آخرون: Azza Ismail (10180577) (author), Reem H. Elhamammy (20448346) (author), Sherien A. Abdelhady (20837720) (author), Rasha Nassra (10180574) (author), Monica S. Makkar (20837723) (author), Mahmoud A. Agami (20837726) (author), Ahmed Wahid (15599036) (author), Hisham A. Nematalla (14798362) (author), Minh Sai (17049279) (author), Daniel Merk (1373682) (author), Ahmed F. El-Yazbi (10180589) (author), Ahmed S.F. Belal (20837729) (author), Ali H. Eid (5461829) (author), Perihan A. Elzahhar (10180568) (author)
منشور في: 2025
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author Mai S. El-Shoukrofy (19646815)
author2 Azza Ismail (10180577)
Reem H. Elhamammy (20448346)
Sherien A. Abdelhady (20837720)
Rasha Nassra (10180574)
Monica S. Makkar (20837723)
Mahmoud A. Agami (20837726)
Ahmed Wahid (15599036)
Hisham A. Nematalla (14798362)
Minh Sai (17049279)
Daniel Merk (1373682)
Ahmed F. El-Yazbi (10180589)
Ahmed S.F. Belal (20837729)
Ali H. Eid (5461829)
Perihan A. Elzahhar (10180568)
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author_facet Mai S. El-Shoukrofy (19646815)
Azza Ismail (10180577)
Reem H. Elhamammy (20448346)
Sherien A. Abdelhady (20837720)
Rasha Nassra (10180574)
Monica S. Makkar (20837723)
Mahmoud A. Agami (20837726)
Ahmed Wahid (15599036)
Hisham A. Nematalla (14798362)
Minh Sai (17049279)
Daniel Merk (1373682)
Ahmed F. El-Yazbi (10180589)
Ahmed S.F. Belal (20837729)
Ali H. Eid (5461829)
Perihan A. Elzahhar (10180568)
author_role author
dc.creator.none.fl_str_mv Mai S. El-Shoukrofy (19646815)
Azza Ismail (10180577)
Reem H. Elhamammy (20448346)
Sherien A. Abdelhady (20837720)
Rasha Nassra (10180574)
Monica S. Makkar (20837723)
Mahmoud A. Agami (20837726)
Ahmed Wahid (15599036)
Hisham A. Nematalla (14798362)
Minh Sai (17049279)
Daniel Merk (1373682)
Ahmed F. El-Yazbi (10180589)
Ahmed S.F. Belal (20837729)
Ali H. Eid (5461829)
Perihan A. Elzahhar (10180568)
dc.date.none.fl_str_mv 2025-02-28T12:00:00Z
dc.identifier.none.fl_str_mv 10.1016/j.ejmech.2025.117415
dc.relation.none.fl_str_mv https://figshare.com/articles/journal_contribution/Novel_thiazolones_for_the_simultaneous_modulation_of_PPAR_COX-2_and_15-LOX_to_address_metabolic_disease-associated_portal_inflammation/28546508
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Biomedical and clinical sciences
Medical biochemistry and metabolomics
Pharmacology and pharmaceutical sciences
Thiazolones
PPARγ
COX-2
15-LOX
Multi-targeting
Liver inflammation
Metabolic diseases
dc.title.none.fl_str_mv Novel thiazolones for the simultaneous modulation of PPARγ, COX-2 and 15-LOX to address metabolic disease-associated portal inflammation
dc.type.none.fl_str_mv Text
Journal contribution
info:eu-repo/semantics/publishedVersion
text
contribution to journal
description <p dir="ltr">A hybrid pharmacophore model, based on structural motifs previously identified by our team, was employed to generate ligands that simultaneously target COX-2, 15-LOX, and PPARγ in the context of metabolic dysfunction-associated fatty liver disease (MAFLD). Notable COX-2 inhibitory activities (IC<sub>50</sub> = 0.065–0.24 μM) were observed relative to celecoxib (IC<sub>50</sub> = 0.049 μM). The two most effective 15-LOX inhibitors, <b>2a</b> and <b>2b</b>, exhibited 69 % and 57% of quercetin's action, respectively. Utilizing the rat hemi-diaphragm model to assess <i>in vitro</i> glucose uptake capacity, compounds <b>2a</b> and <b>2b</b> demonstrated significant glucose uptake potential in the absence of insulin, surpassing that of pioglitazone. Compound 2a activated PPARγ with an EC<sub>50</sub> value of 3.4 μM in a Gal4-hybrid reporter gene assay, indicating partial agonistic action. Interesting binding interactions with targets of interest were identified by molecular docking studies. As well, the expression levels of 20-HETE, Il-1β and TNF-α were decreased in LPS-challenged RAW264.7 macrophages upon treatment with compound <b>2a</b>. The pharmacokinetic analysis of <b>2a</b> and assessment of its in vivo efficacy in addressing hepatic impairment in rat models of diabetes and pre-diabetes were carried out. Together, these findings may offer preliminary insights into the potential of these compounds for further refinement in the existing therapeutic arsenals for metabolic diseases.</p><h2>Other Information</h2><p dir="ltr">Published in: European Journal of Medicinal Chemistry<br>License: <a href="http://creativecommons.org/licenses/by/4.0/" target="_blank">http://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.1016/j.ejmech.2025.117415" target="_blank">https://dx.doi.org/10.1016/j.ejmech.2025.117415</a></p>
eu_rights_str_mv openAccess
id Manara2_d0e7a87c16680e4c79d48b0f5e7f0ac2
identifier_str_mv 10.1016/j.ejmech.2025.117415
network_acronym_str Manara2
network_name_str Manara2
oai_identifier_str oai:figshare.com:article/28546508
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spelling Novel thiazolones for the simultaneous modulation of PPARγ, COX-2 and 15-LOX to address metabolic disease-associated portal inflammationMai S. El-Shoukrofy (19646815)Azza Ismail (10180577)Reem H. Elhamammy (20448346)Sherien A. Abdelhady (20837720)Rasha Nassra (10180574)Monica S. Makkar (20837723)Mahmoud A. Agami (20837726)Ahmed Wahid (15599036)Hisham A. Nematalla (14798362)Minh Sai (17049279)Daniel Merk (1373682)Ahmed F. El-Yazbi (10180589)Ahmed S.F. Belal (20837729)Ali H. Eid (5461829)Perihan A. Elzahhar (10180568)Biomedical and clinical sciencesMedical biochemistry and metabolomicsPharmacology and pharmaceutical sciencesThiazolonesPPARγCOX-215-LOXMulti-targetingLiver inflammationMetabolic diseases<p dir="ltr">A hybrid pharmacophore model, based on structural motifs previously identified by our team, was employed to generate ligands that simultaneously target COX-2, 15-LOX, and PPARγ in the context of metabolic dysfunction-associated fatty liver disease (MAFLD). Notable COX-2 inhibitory activities (IC<sub>50</sub> = 0.065–0.24 μM) were observed relative to celecoxib (IC<sub>50</sub> = 0.049 μM). The two most effective 15-LOX inhibitors, <b>2a</b> and <b>2b</b>, exhibited 69 % and 57% of quercetin's action, respectively. Utilizing the rat hemi-diaphragm model to assess <i>in vitro</i> glucose uptake capacity, compounds <b>2a</b> and <b>2b</b> demonstrated significant glucose uptake potential in the absence of insulin, surpassing that of pioglitazone. Compound 2a activated PPARγ with an EC<sub>50</sub> value of 3.4 μM in a Gal4-hybrid reporter gene assay, indicating partial agonistic action. Interesting binding interactions with targets of interest were identified by molecular docking studies. As well, the expression levels of 20-HETE, Il-1β and TNF-α were decreased in LPS-challenged RAW264.7 macrophages upon treatment with compound <b>2a</b>. The pharmacokinetic analysis of <b>2a</b> and assessment of its in vivo efficacy in addressing hepatic impairment in rat models of diabetes and pre-diabetes were carried out. Together, these findings may offer preliminary insights into the potential of these compounds for further refinement in the existing therapeutic arsenals for metabolic diseases.</p><h2>Other Information</h2><p dir="ltr">Published in: European Journal of Medicinal Chemistry<br>License: <a href="http://creativecommons.org/licenses/by/4.0/" target="_blank">http://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.1016/j.ejmech.2025.117415" target="_blank">https://dx.doi.org/10.1016/j.ejmech.2025.117415</a></p>2025-02-28T12:00:00ZTextJournal contributioninfo:eu-repo/semantics/publishedVersiontextcontribution to journal10.1016/j.ejmech.2025.117415https://figshare.com/articles/journal_contribution/Novel_thiazolones_for_the_simultaneous_modulation_of_PPAR_COX-2_and_15-LOX_to_address_metabolic_disease-associated_portal_inflammation/28546508CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/285465082025-02-28T12:00:00Z
spellingShingle Novel thiazolones for the simultaneous modulation of PPARγ, COX-2 and 15-LOX to address metabolic disease-associated portal inflammation
Mai S. El-Shoukrofy (19646815)
Biomedical and clinical sciences
Medical biochemistry and metabolomics
Pharmacology and pharmaceutical sciences
Thiazolones
PPARγ
COX-2
15-LOX
Multi-targeting
Liver inflammation
Metabolic diseases
status_str publishedVersion
title Novel thiazolones for the simultaneous modulation of PPARγ, COX-2 and 15-LOX to address metabolic disease-associated portal inflammation
title_full Novel thiazolones for the simultaneous modulation of PPARγ, COX-2 and 15-LOX to address metabolic disease-associated portal inflammation
title_fullStr Novel thiazolones for the simultaneous modulation of PPARγ, COX-2 and 15-LOX to address metabolic disease-associated portal inflammation
title_full_unstemmed Novel thiazolones for the simultaneous modulation of PPARγ, COX-2 and 15-LOX to address metabolic disease-associated portal inflammation
title_short Novel thiazolones for the simultaneous modulation of PPARγ, COX-2 and 15-LOX to address metabolic disease-associated portal inflammation
title_sort Novel thiazolones for the simultaneous modulation of PPARγ, COX-2 and 15-LOX to address metabolic disease-associated portal inflammation
topic Biomedical and clinical sciences
Medical biochemistry and metabolomics
Pharmacology and pharmaceutical sciences
Thiazolones
PPARγ
COX-2
15-LOX
Multi-targeting
Liver inflammation
Metabolic diseases