WNT signaling at the intersection between neurogenesis and brain tumorigenesis

<div><p>Neurogenesis and tumorigenesis share signaling molecules/pathways involved in cell proliferation, differentiation, migration, and death. Self-renewal of neural stem cells is a tightly regulated process that secures the accuracy of cell division and eliminates cells that undergo m...

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Main Author: Maisa I. Alkailani (15070764) (author)
Other Authors: Mohamed Aittaleb (18007087) (author), Fadel Tissir (245153) (author)
Published: 2022
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author Maisa I. Alkailani (15070764)
author2 Mohamed Aittaleb (18007087)
Fadel Tissir (245153)
author2_role author
author
author_facet Maisa I. Alkailani (15070764)
Mohamed Aittaleb (18007087)
Fadel Tissir (245153)
author_role author
dc.creator.none.fl_str_mv Maisa I. Alkailani (15070764)
Mohamed Aittaleb (18007087)
Fadel Tissir (245153)
dc.date.none.fl_str_mv 2022-10-04T03:00:00Z
dc.identifier.none.fl_str_mv 10.3389/fnmol.2022.1017568
dc.relation.none.fl_str_mv https://figshare.com/articles/journal_contribution/WNT_signaling_at_the_intersection_between_neurogenesis_and_brain_tumorigenesis/25659090
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Biomedical and clinical sciences
Neurosciences
Wnt/β-catenin
Wnt/PCP
WNT/calcium
neural progenitor cells
neurogenesis
glioma
glioblastoma therapy
dc.title.none.fl_str_mv WNT signaling at the intersection between neurogenesis and brain tumorigenesis
dc.type.none.fl_str_mv Text
Journal contribution
info:eu-repo/semantics/publishedVersion
text
contribution to journal
description <div><p>Neurogenesis and tumorigenesis share signaling molecules/pathways involved in cell proliferation, differentiation, migration, and death. Self-renewal of neural stem cells is a tightly regulated process that secures the accuracy of cell division and eliminates cells that undergo mitotic errors. Abnormalities in the molecular mechanisms controlling this process can trigger aneuploidy and genome instability, leading to neoplastic transformation. Mutations that affect cell adhesion, polarity, or migration enhance the invasive potential and favor the progression of tumors. Here, we review recent evidence of the WNT pathway’s involvement in both neurogenesis and tumorigenesis and discuss the experimental progress on therapeutic opportunities targeting components of this pathway.</p><p> </p></div><h2>Other Information</h2> <p> Published in: Frontiers in Molecular Neuroscience<br> License: <a href="https://creativecommons.org/licenses/by/4.0/" target="_blank">https://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.3389/fnmol.2022.1017568" target="_blank">https://dx.doi.org/10.3389/fnmol.2022.1017568</a></p>
eu_rights_str_mv openAccess
id Manara2_e8967e446538a8a6a62a21e692f4addc
identifier_str_mv 10.3389/fnmol.2022.1017568
network_acronym_str Manara2
network_name_str Manara2
oai_identifier_str oai:figshare.com:article/25659090
publishDate 2022
repository.mail.fl_str_mv
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rights_invalid_str_mv CC BY 4.0
spelling WNT signaling at the intersection between neurogenesis and brain tumorigenesisMaisa I. Alkailani (15070764)Mohamed Aittaleb (18007087)Fadel Tissir (245153)Biomedical and clinical sciencesNeurosciencesWnt/β-cateninWnt/PCPWNT/calciumneural progenitor cellsneurogenesisgliomaglioblastoma therapy<div><p>Neurogenesis and tumorigenesis share signaling molecules/pathways involved in cell proliferation, differentiation, migration, and death. Self-renewal of neural stem cells is a tightly regulated process that secures the accuracy of cell division and eliminates cells that undergo mitotic errors. Abnormalities in the molecular mechanisms controlling this process can trigger aneuploidy and genome instability, leading to neoplastic transformation. Mutations that affect cell adhesion, polarity, or migration enhance the invasive potential and favor the progression of tumors. Here, we review recent evidence of the WNT pathway’s involvement in both neurogenesis and tumorigenesis and discuss the experimental progress on therapeutic opportunities targeting components of this pathway.</p><p> </p></div><h2>Other Information</h2> <p> Published in: Frontiers in Molecular Neuroscience<br> License: <a href="https://creativecommons.org/licenses/by/4.0/" target="_blank">https://creativecommons.org/licenses/by/4.0/</a><br>See article on publisher's website: <a href="https://dx.doi.org/10.3389/fnmol.2022.1017568" target="_blank">https://dx.doi.org/10.3389/fnmol.2022.1017568</a></p>2022-10-04T03:00:00ZTextJournal contributioninfo:eu-repo/semantics/publishedVersiontextcontribution to journal10.3389/fnmol.2022.1017568https://figshare.com/articles/journal_contribution/WNT_signaling_at_the_intersection_between_neurogenesis_and_brain_tumorigenesis/25659090CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/256590902022-10-04T03:00:00Z
spellingShingle WNT signaling at the intersection between neurogenesis and brain tumorigenesis
Maisa I. Alkailani (15070764)
Biomedical and clinical sciences
Neurosciences
Wnt/β-catenin
Wnt/PCP
WNT/calcium
neural progenitor cells
neurogenesis
glioma
glioblastoma therapy
status_str publishedVersion
title WNT signaling at the intersection between neurogenesis and brain tumorigenesis
title_full WNT signaling at the intersection between neurogenesis and brain tumorigenesis
title_fullStr WNT signaling at the intersection between neurogenesis and brain tumorigenesis
title_full_unstemmed WNT signaling at the intersection between neurogenesis and brain tumorigenesis
title_short WNT signaling at the intersection between neurogenesis and brain tumorigenesis
title_sort WNT signaling at the intersection between neurogenesis and brain tumorigenesis
topic Biomedical and clinical sciences
Neurosciences
Wnt/β-catenin
Wnt/PCP
WNT/calcium
neural progenitor cells
neurogenesis
glioma
glioblastoma therapy