SH2D5 regulates epithelial-mesenchymal transition (EMT) in lung adenocarcinoma (LUAD) cells.

<p>(A, B) Gene Set Enrichment Analysis (GSEA) was used to identified the potential regulatory pathways between high and low SH2D5 expression groups. NES, normalized enrichment score. (C, D) The expression of EMT‐related genes in LUAD cells with SH2D5 overexpression or knockdown determined by W...

وصف كامل

محفوظ في:

التفاصيل البيبلوغرافية
المؤلف الرئيسي:	Licheng Du (16469836) (author)
مؤلفون آخرون:	Wenjia Ren (16469833) (author), Linjun Liu (16469830) (author), Haojia Zhu (20485284) (author), Ke Xu (134879) (author), Yubai Zhou (3931427) (author)
منشور في:	2024
الموضوعات:	Biochemistry Medicine Cell Biology Molecular Biology Developmental Biology Cancer Biological Sciences not elsewhere classified uncharacterized scaffolding protein inhibitors partially reversed common histological subtype crucial potential target high mortality rate akt signaling pathway akt pathway activators sh2d5 positively regulates sh2d5 may serve luad remains unclear emt processes induced lung adenocarcinoma cells found aberrant expression potential mechanisms lung cancer high incidence high expression sh2d5 promotes sh2d5 influenced study demonstrated rescue experiments present study poor prognosis phosphorylation levels mesenchymal transition luad tissues luad patients luad cells late stages closely associated
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SH2D5 regulates epithelial-mesenchymal transition (EMT) in lung adenocarcinoma (LUAD) cells.

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