Peroxynitrite appears to be the main source of ROS in YFV-17D infection.

(A) ELISA quantification of IFNβ secreted by HepG2 cells infected with YFV-17D and treated with NAC [10nM] at 0hpi or 24hpi. (B) Flow cytometric quantification of peroxynitrite staining in mock, YFV-17D, and DENV2 infected HepG2 cells treated with MnTBA...

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Main Author:	Samantha G. Muccilli (21156443) (author)
Other Authors:	Benjamin Schwarz (1527106) (author), Byron Shue (14774425) (author), Forrest Jessop (2902226) (author), Jeffrey G. Shannon (21156446) (author), Charles L. Larson (21156449) (author), Adam Hage (12519597) (author), Seon-Hui Hong (15016838) (author), Eric Bohrnsen (12206821) (author), Thomas Hsu (21156452) (author), Alison W. Ashbrook (9444069) (author), Gail L. Sturdevant (11784711) (author), Shelly J. Robertson (3826663) (author), Joseph W. Guarnieri (12549292) (author), Justin Lack (3774427) (author), Douglas C. Wallace (57574) (author), Catharine M. Bosio (178161) (author), Margaret R. MacDonald (9444081) (author), Charles M. Rice (9110743) (author), Jonathan W. Yewdell (17937527) (author), Sonja M. Best (10005261) (author)
Published:	2025
Subjects:	Medicine Microbiology Cell Biology Genetics Molecular Biology Immunology Infectious Diseases Plant Biology Virology Biological Sciences not elsewhere classified meet energy demands innate immunity essential innate immune responses high antiviral immunity fuel atp synthase like receptor signaling yellow fever virus major metabolic perturbations live attenuated vaccine ifn signaling pathways abrogated mavs oligomerization blunted ifn response signaling mechanisms ifn expression parental virus metabolic stress 17d live xlink "> molecular understanding mitochondrial hyperactivity maximal activation infected cells including peroxynitrite including hyperactivation electron transport critical messengers considered one
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Peroxynitrite appears to be the main source of ROS in YFV-17D infection.

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