SRPK1 activity is required for HPV16 late gene expression.

<p>(A) Western blot analysis of levels of L1 protein in monolayer cultured and differentiated NIKS16 cells mock-treated or DMSO-treated or treated with 10 µM or 50 µM SRPIN340 for 48 hours. Protein extract from monolayer cultured and differentiated HPV-negative, NIKS cells was included on the...

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Bibliographic Details
Main Author:	Arwa A. A. Faizo (21030140) (author)
Other Authors:	Clare Bellward (21030143) (author), Hegel R. Hernandez-Lopez (21030146) (author), Andrew Stevenson (9143975) (author), Quan Gu (204269) (author), Sheila V. Graham (11864634) (author)
Published:	2025
Subjects:	Biochemistry Cell Biology Genetics Molecular Biology Immunology Developmental Biology Cancer Infectious Diseases Virology Biological Sciences not elsewhere classified pathway analysis showed novel antiviral therapies cause precancerous lesions cause benign lesions called &# 8220 positively regulate splicing phosphorylate sr proteins l1 late proteins sr protein kinases sr protein activity srpin340 treatment led since srpk1 controls hpv life cycle splicing factor kinase e2 viral replication viral replication protein family alternative splicing transcription factor late events viral mrnas inhibit replication three days therapeutic strategy srpin340 inhibits specific inhibitor sindbis virus rna processing nuclear entry may progress malignant tumours infect epithelia hpvs infecting hpv16 infection hpv e4 full suite epithelial barrier dna repair chromatin conformation cellular location arginine rich arginine domains anogenital region also observed 935 rnas
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SRPK1 activity is required for HPV16 late gene expression.

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