Figure 2 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival

<p>Knockdown SIRT3 caused ATF4 protein decrease via translation regulation. <b>A,</b> Western blots show ATF4 protein levels in different DLBCL cells with control or SIRT3 shRNAs. SIRT3 was blotted showing knockdown efficiency, and ACTB was used as reference protein control. <b&...

全面介绍

Saved in:
书目详细资料
主要作者: Meng Li (79487) (author)
其他作者: Matthew R. Teater (15129060) (author), Jun Young Hong (6588914) (author), Noel R. Park (15129063) (author), Cihangir Duy (15112006) (author), Hao Shen (195461) (author), Ling Wang (56577) (author), Zhengming Chen (385056) (author), Leandro Cerchietti (15091552) (author), Shawn M. Davidson (14911596) (author), Hening Lin (1306563) (author), Ari M. Melnick (15110132) (author)
出版: 2025
主题:
标签: 添加标签
没有标签, 成为第一个标记此记录!
_version_ 1849927640595562496
author Meng Li (79487)
author2 Matthew R. Teater (15129060)
Jun Young Hong (6588914)
Noel R. Park (15129063)
Cihangir Duy (15112006)
Hao Shen (195461)
Ling Wang (56577)
Zhengming Chen (385056)
Leandro Cerchietti (15091552)
Shawn M. Davidson (14911596)
Hening Lin (1306563)
Ari M. Melnick (15110132)
author2_role author
author
author
author
author
author
author
author
author
author
author
author_facet Meng Li (79487)
Matthew R. Teater (15129060)
Jun Young Hong (6588914)
Noel R. Park (15129063)
Cihangir Duy (15112006)
Hao Shen (195461)
Ling Wang (56577)
Zhengming Chen (385056)
Leandro Cerchietti (15091552)
Shawn M. Davidson (14911596)
Hening Lin (1306563)
Ari M. Melnick (15110132)
author_role author
dc.creator.none.fl_str_mv Meng Li (79487)
Matthew R. Teater (15129060)
Jun Young Hong (6588914)
Noel R. Park (15129063)
Cihangir Duy (15112006)
Hao Shen (195461)
Ling Wang (56577)
Zhengming Chen (385056)
Leandro Cerchietti (15091552)
Shawn M. Davidson (14911596)
Hening Lin (1306563)
Ari M. Melnick (15110132)
dc.date.none.fl_str_mv 2025-11-24T22:01:44Z
dc.identifier.none.fl_str_mv 10.1158/2643-3230.30698631
dc.relation.none.fl_str_mv https://figshare.com/articles/figure/Figure_2_from_Translational_Activation_of_ATF4_through_Mitochondrial_Anaplerotic_Metabolic_Pathways_Is_Required_for_DLBCL_Growth_and_Survival/30698631
dc.rights.none.fl_str_mv CC BY
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Cancer
Molecular and Cellular Biology
Cellular Stress Responses
Gene Regulation
Posttranscriptional and translational control
Hematological Cancers
Lymphomas
Metabolism
dc.title.none.fl_str_mv Figure 2 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
dc.type.none.fl_str_mv Image
Figure
info:eu-repo/semantics/publishedVersion
image
description <p>Knockdown SIRT3 caused ATF4 protein decrease via translation regulation. <b>A,</b> Western blots show ATF4 protein levels in different DLBCL cells with control or SIRT3 shRNAs. SIRT3 was blotted showing knockdown efficiency, and ACTB was used as reference protein control. <b>B,</b> qPCR results reflect the mRNA levels of ATF4 and PSAT1 in OCI-LY1 cells. Samples were harvested at day 4 after viral transduction. Results were normalized to the mRNA levels in control shRNA–transduced cells. <b>C,</b> qPCR results reflect the mRNA levels of ATF4 and PSAT1 in Karpas 422 cells. Samples were harvested at day 4 after viral transduction. Actin mRNA was used as reference, and results were normalized to the mRNA levels in control shRNA–transduced cells. <b>D,</b> qPCR results show the relative levels of ATF4 mRNAs in different cell lines at different time points after shRNA transduction. Samples were harvested at days 2, 3, 4, and 7 after viral transduction. Actin mRNA was used as reference, and results were normalized to the mRNA levels in control shRNA–transduced cells. <b>E,</b> Western blots show changes of phosphorylation of EIF2A and ATF4 protein levels in Karpas 422 cells with control or SIRT3 shRNAs. Total EIF2A and ACTB were blotted as loading controls. <b>F,</b> Western blots show GFP expression from the ATF4-5′UTR-GFP reporter and endogenous ATF4 protein levels in Karpas 422 cells with control or SIRT3 shRNAs. Tubulin and ACTB were blotted as loading controls. <b>G,</b> FCs of mean fluorescence intensity (MFI) of GFP expressed from the ATF4-5′UTR-GFP translation reporter in Karpas 422 cells with control or SIRT3 shRNAs. The data were collected from days 4 and 7 after viral transduction. MFI of normal yellow fluorescent protein (YFP) expression in control or SIRT3 knockdown cells were used for normalization to avoid background translation variations. <b>H,</b> FCs of MFI of GFP expressed from the ATF4-5′UTR-GFP translation reporter in OCI-LY1 cells with control or SIRT3 shRNAs. The data were collected from days 4 and 7 after viral transduction. MFI of normal YFP expression in control or SIRT3 knockdown cells were used for normalization to avoid background translation variations. Error bars represent the mean ± SD of three or more replicates.</p>
eu_rights_str_mv openAccess
id Manara_424a2d4703fea8a2ac52922eff732857
identifier_str_mv 10.1158/2643-3230.30698631
network_acronym_str Manara
network_name_str ManaraRepo
oai_identifier_str oai:figshare.com:article/30698631
publishDate 2025
repository.mail.fl_str_mv
repository.name.fl_str_mv
repository_id_str
rights_invalid_str_mv CC BY
spelling Figure 2 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and SurvivalMeng Li (79487)Matthew R. Teater (15129060)Jun Young Hong (6588914)Noel R. Park (15129063)Cihangir Duy (15112006)Hao Shen (195461)Ling Wang (56577)Zhengming Chen (385056)Leandro Cerchietti (15091552)Shawn M. Davidson (14911596)Hening Lin (1306563)Ari M. Melnick (15110132)CancerMolecular and Cellular BiologyCellular Stress ResponsesGene RegulationPosttranscriptional and translational controlHematological CancersLymphomasMetabolism<p>Knockdown SIRT3 caused ATF4 protein decrease via translation regulation. <b>A,</b> Western blots show ATF4 protein levels in different DLBCL cells with control or SIRT3 shRNAs. SIRT3 was blotted showing knockdown efficiency, and ACTB was used as reference protein control. <b>B,</b> qPCR results reflect the mRNA levels of ATF4 and PSAT1 in OCI-LY1 cells. Samples were harvested at day 4 after viral transduction. Results were normalized to the mRNA levels in control shRNA–transduced cells. <b>C,</b> qPCR results reflect the mRNA levels of ATF4 and PSAT1 in Karpas 422 cells. Samples were harvested at day 4 after viral transduction. Actin mRNA was used as reference, and results were normalized to the mRNA levels in control shRNA–transduced cells. <b>D,</b> qPCR results show the relative levels of ATF4 mRNAs in different cell lines at different time points after shRNA transduction. Samples were harvested at days 2, 3, 4, and 7 after viral transduction. Actin mRNA was used as reference, and results were normalized to the mRNA levels in control shRNA–transduced cells. <b>E,</b> Western blots show changes of phosphorylation of EIF2A and ATF4 protein levels in Karpas 422 cells with control or SIRT3 shRNAs. Total EIF2A and ACTB were blotted as loading controls. <b>F,</b> Western blots show GFP expression from the ATF4-5′UTR-GFP reporter and endogenous ATF4 protein levels in Karpas 422 cells with control or SIRT3 shRNAs. Tubulin and ACTB were blotted as loading controls. <b>G,</b> FCs of mean fluorescence intensity (MFI) of GFP expressed from the ATF4-5′UTR-GFP translation reporter in Karpas 422 cells with control or SIRT3 shRNAs. The data were collected from days 4 and 7 after viral transduction. MFI of normal yellow fluorescent protein (YFP) expression in control or SIRT3 knockdown cells were used for normalization to avoid background translation variations. <b>H,</b> FCs of MFI of GFP expressed from the ATF4-5′UTR-GFP translation reporter in OCI-LY1 cells with control or SIRT3 shRNAs. The data were collected from days 4 and 7 after viral transduction. MFI of normal YFP expression in control or SIRT3 knockdown cells were used for normalization to avoid background translation variations. Error bars represent the mean ± SD of three or more replicates.</p>2025-11-24T22:01:44ZImageFigureinfo:eu-repo/semantics/publishedVersionimage10.1158/2643-3230.30698631https://figshare.com/articles/figure/Figure_2_from_Translational_Activation_of_ATF4_through_Mitochondrial_Anaplerotic_Metabolic_Pathways_Is_Required_for_DLBCL_Growth_and_Survival/30698631CC BYinfo:eu-repo/semantics/openAccessoai:figshare.com:article/306986312025-11-24T22:01:44Z
spellingShingle Figure 2 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
Meng Li (79487)
Cancer
Molecular and Cellular Biology
Cellular Stress Responses
Gene Regulation
Posttranscriptional and translational control
Hematological Cancers
Lymphomas
Metabolism
status_str publishedVersion
title Figure 2 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
title_full Figure 2 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
title_fullStr Figure 2 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
title_full_unstemmed Figure 2 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
title_short Figure 2 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
title_sort Figure 2 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
topic Cancer
Molecular and Cellular Biology
Cellular Stress Responses
Gene Regulation
Posttranscriptional and translational control
Hematological Cancers
Lymphomas
Metabolism