$S3 Fig - PPARγ inhibitors enhance the efficacy of statin therapy for steroid-induced osteonecrosis of the femoral head by directly inhibiting apoptosis and indirectly modulating lipoprotein subfractions$

S3 Fig - PPARγ inhibitors enhance the efficacy of statin therapy for steroid-induced osteonecrosis of the femoral head by directly inhibiting apoptosis and indirectly modulating lipoprotein subfractions

S3 Fig - <p>PPARγ inhibitors enhance the efficacy of statin therapy for steroid-induced osteonecrosis of the femoral head by directly inhibiting apoptosis and indirectly modulating lipoprotein subfractions</p>

محفوظ في:

التفاصيل البيبلوغرافية
المؤلف الرئيسي:	Kai-yun Chen (21578641) (author)
مؤلفون آخرون:	Xu-huan Li (21578644) (author), Dan Chen (32468) (author), Shi-da Qian (21578647) (author), Run-hong Mei (21578650) (author), Qian Li (114642) (author), Xue-feng Yu (14523553) (author), Xi-jing He (21578653) (author)
منشور في:	2025
الموضوعات:	Biochemistry Medicine Cell Biology Pharmacology Biotechnology Marine Biology Cancer Infectious Diseases Virology Chemical Sciences not elsewhere classified vap analysis showed ppar &# 947 pathological results showed disturbed lipoprotein subfractions assessed cell proliferation term glucocorticoid therapy directly inhibiting apoptosis apoptosis using cck inhibitors significantly enhance inhibitors may enhance combination therapy may vitro experiments showed sonfh remains limited improving lipid metabolism first treated osteoblasts combination therapy group xlink "> steroid inhibitors enhance xlink "> lipid metabolism statin therapy induced apoptosis lovastatin group control group western blotting study aims statin lovastatin sonfh environment several mechanisms randomly divided performed hematoxylin induced osteonecrosis high concentrations genes related fourth week flow cytometry femoral head eosin staining clinical management animal studies 4 weeks
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S3 Fig - PPARγ inhibitors enhance the efficacy of statin therapy for steroid-induced osteonecrosis of the femoral head by directly inhibiting apoptosis and indirectly modulating lipoprotein subfractions

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