Heart bacterial burden in mice infected with MRSA wild-type, Δ<i>yqhG</i>, and complemented strains.

<p>Heart bacterial burden in mice infected with MRSA wild-type, Δ<i>yqhG</i>, and complemented strains.</p>

Wedi'i Gadw mewn:
Manylion Llyfryddiaeth
Prif Awdur: Jianhua Liao (1393228) (author)
Awduron Eraill: Jun Cheng (194158) (author), Baoqing Liu (1508821) (author), Yuzhi Shao (22683349) (author), Chunyan Meng (5993228) (author)
Cyhoeddwyd: 2025
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author Jianhua Liao (1393228)
author2 Jun Cheng (194158)
Baoqing Liu (1508821)
Yuzhi Shao (22683349)
Chunyan Meng (5993228)
author2_role author
author
author
author
author_facet Jianhua Liao (1393228)
Jun Cheng (194158)
Baoqing Liu (1508821)
Yuzhi Shao (22683349)
Chunyan Meng (5993228)
author_role author
dc.creator.none.fl_str_mv Jianhua Liao (1393228)
Jun Cheng (194158)
Baoqing Liu (1508821)
Yuzhi Shao (22683349)
Chunyan Meng (5993228)
dc.date.none.fl_str_mv 2025-11-25T18:27:50Z
dc.identifier.none.fl_str_mv 10.1371/journal.pone.0337292.s003
dc.relation.none.fl_str_mv https://figshare.com/articles/dataset/Heart_bacterial_burden_in_mice_infected_with_MRSA_wild-type_i_yqhG_i_and_complemented_strains_/30713716
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Medicine
Microbiology
Cell Biology
Biotechnology
Evolutionary Biology
Ecology
Science Policy
Infectious Diseases
Virology
Environmental Sciences not elsewhere classified
Biological Sciences not elsewhere classified
therapeutic strategies aimed
novel therapeutic approaches
murine infection model
div >< p
defense mechanisms employed
conserved gene encoding
compromising membrane stability
affect biofilm formation
staphylococcus aureus </
reduced bacterial burden
improved host survival
maintaining mrsa ’
oxidative stress resistance
yqhg </
withstand host
reduced motility
oxidative stress
increasing resistance
stress adaptation
potential target
periplasmic protein
mrsa virulence
mrsa pathogenesis
key determinant
infected organs
increased sensitivity
imposed stresses
growing prevalence
findings highlight
existing antibiotics
critical need
attenuates virulence
dc.title.none.fl_str_mv Heart bacterial burden in mice infected with MRSA wild-type, Δ<i>yqhG</i>, and complemented strains.
dc.type.none.fl_str_mv Dataset
info:eu-repo/semantics/publishedVersion
dataset
description <p>Heart bacterial burden in mice infected with MRSA wild-type, Δ<i>yqhG</i>, and complemented strains.</p>
eu_rights_str_mv openAccess
id Manara_6975aaf734f5744804a9f240da846f3e
identifier_str_mv 10.1371/journal.pone.0337292.s003
network_acronym_str Manara
network_name_str ManaraRepo
oai_identifier_str oai:figshare.com:article/30713716
publishDate 2025
repository.mail.fl_str_mv
repository.name.fl_str_mv
repository_id_str
rights_invalid_str_mv CC BY 4.0
spelling Heart bacterial burden in mice infected with MRSA wild-type, Δ<i>yqhG</i>, and complemented strains.Jianhua Liao (1393228)Jun Cheng (194158)Baoqing Liu (1508821)Yuzhi Shao (22683349)Chunyan Meng (5993228)MedicineMicrobiologyCell BiologyBiotechnologyEvolutionary BiologyEcologyScience PolicyInfectious DiseasesVirologyEnvironmental Sciences not elsewhere classifiedBiological Sciences not elsewhere classifiedtherapeutic strategies aimednovel therapeutic approachesmurine infection modeldiv >< pdefense mechanisms employedconserved gene encodingcompromising membrane stabilityaffect biofilm formationstaphylococcus aureus </reduced bacterial burdenimproved host survivalmaintaining mrsa ’oxidative stress resistanceyqhg </withstand hostreduced motilityoxidative stressincreasing resistancestress adaptationpotential targetperiplasmic proteinmrsa virulencemrsa pathogenesiskey determinantinfected organsincreased sensitivityimposed stressesgrowing prevalencefindings highlightexisting antibioticscritical needattenuates virulence<p>Heart bacterial burden in mice infected with MRSA wild-type, Δ<i>yqhG</i>, and complemented strains.</p>2025-11-25T18:27:50ZDatasetinfo:eu-repo/semantics/publishedVersiondataset10.1371/journal.pone.0337292.s003https://figshare.com/articles/dataset/Heart_bacterial_burden_in_mice_infected_with_MRSA_wild-type_i_yqhG_i_and_complemented_strains_/30713716CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/307137162025-11-25T18:27:50Z
spellingShingle Heart bacterial burden in mice infected with MRSA wild-type, Δ<i>yqhG</i>, and complemented strains.
Jianhua Liao (1393228)
Medicine
Microbiology
Cell Biology
Biotechnology
Evolutionary Biology
Ecology
Science Policy
Infectious Diseases
Virology
Environmental Sciences not elsewhere classified
Biological Sciences not elsewhere classified
therapeutic strategies aimed
novel therapeutic approaches
murine infection model
div >< p
defense mechanisms employed
conserved gene encoding
compromising membrane stability
affect biofilm formation
staphylococcus aureus </
reduced bacterial burden
improved host survival
maintaining mrsa ’
oxidative stress resistance
yqhg </
withstand host
reduced motility
oxidative stress
increasing resistance
stress adaptation
potential target
periplasmic protein
mrsa virulence
mrsa pathogenesis
key determinant
infected organs
increased sensitivity
imposed stresses
growing prevalence
findings highlight
existing antibiotics
critical need
attenuates virulence
status_str publishedVersion
title Heart bacterial burden in mice infected with MRSA wild-type, Δ<i>yqhG</i>, and complemented strains.
title_full Heart bacterial burden in mice infected with MRSA wild-type, Δ<i>yqhG</i>, and complemented strains.
title_fullStr Heart bacterial burden in mice infected with MRSA wild-type, Δ<i>yqhG</i>, and complemented strains.
title_full_unstemmed Heart bacterial burden in mice infected with MRSA wild-type, Δ<i>yqhG</i>, and complemented strains.
title_short Heart bacterial burden in mice infected with MRSA wild-type, Δ<i>yqhG</i>, and complemented strains.
title_sort Heart bacterial burden in mice infected with MRSA wild-type, Δ<i>yqhG</i>, and complemented strains.
topic Medicine
Microbiology
Cell Biology
Biotechnology
Evolutionary Biology
Ecology
Science Policy
Infectious Diseases
Virology
Environmental Sciences not elsewhere classified
Biological Sciences not elsewhere classified
therapeutic strategies aimed
novel therapeutic approaches
murine infection model
div >< p
defense mechanisms employed
conserved gene encoding
compromising membrane stability
affect biofilm formation
staphylococcus aureus </
reduced bacterial burden
improved host survival
maintaining mrsa ’
oxidative stress resistance
yqhg </
withstand host
reduced motility
oxidative stress
increasing resistance
stress adaptation
potential target
periplasmic protein
mrsa virulence
mrsa pathogenesis
key determinant
infected organs
increased sensitivity
imposed stresses
growing prevalence
findings highlight
existing antibiotics
critical need
attenuates virulence