Image 1_SLC16A3 drives lung adenocarcinoma progression and gefitinib resistance through coordinated regulation of ferroptosis and lactate metabolism.jpg
Background<p>Ferroptosis is an iron-dependent form of regulated cell death that plays a critical role in tumor suppression and therapeutic response. However, the metabolic mechanisms that drive ferroptosis resistance in lung adenocarcinoma (LUAD), particularly in the context of EGFR-TKI tolera...
محفوظ في:
| المؤلف الرئيسي: | Wenhan Cai (12693371) (author) |
|---|---|
| مؤلفون آخرون: | Yiming Liu (765457) (author), Kai Zhao (53042) (author), Zirui Zhu (15171841) (author), Jiamei Jin (18827032) (author), Jiaxin Wen (5531090) (author), Zhiqiang Xue (5531093) (author) |
| منشور في: |
2025
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| الموضوعات: | |
| الوسوم: |
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مواد مشابهة
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Image 2_SLC16A3 drives lung adenocarcinoma progression and gefitinib resistance through coordinated regulation of ferroptosis and lactate metabolism.jpeg
حسب: Wenhan Cai (12693371)
منشور في: (2025) -
Table 1_SLC16A3 drives lung adenocarcinoma progression and gefitinib resistance through coordinated regulation of ferroptosis and lactate metabolism.docx
حسب: Wenhan Cai (12693371)
منشور في: (2025) -
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منشور في: (2025) -
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Data Sheet 1_Case Report: Gefitinib in EGFR 19del recurrent aggressive fibromatosis.docx
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