Knockdown of BTB domain and CNC homolog 1 (BACH1) impedes Polo-like kinase 1 (PLK1)-induced phenotype in melanoma.

(A and B) Immunoblots for the expression of BACH1 in 2D-monolayer (A) and 3D-tumorsphere (B) of mouse melanoma cells (mMC) and mouse melanoma with Plk1 knock-in (mMPI) cells. (C) Immunoblots of BACH1 protein in nucleus and cytosol of mMC and mMPI cells....

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Bibliographische Detailangaben
1. Verfasser:	Fengyi Mao (15073242) (author)
Weitere Verfasser:	Sai Wu (6096290) (author), Derek B. Allison (15141675) (author), Daheng He (9189701) (author), Yifan Kong (4458616) (author), Chaohao Li (15141681) (author), Zhiguo Li (431917) (author), Yanquan Zhang (14930570) (author), Xinyi Wang (559030) (author), Qiongsi Zhang (2818111) (author), Chi Wang (131921) (author), Xiaoqi Liu (45630) (author)
Veröffentlicht:	2025
Schlagworte:	Cell Biology Genetics Molecular Biology Developmental Biology Cancer Hematology Biological Sciences not elsewhere classified suppress tumor growth like kinase 1 crucial transcription factor cnc homolog 1 clinical database analysis shortened mice survival >< sup >< div >< p cutaneous melanoma based v600e </ sup patient survival pten </ ca </ ></ sup synergistic efficiency novel function negatively correlates genes involved cell cycle cancer progression cancer metabolism bach1 ), >- deficiency
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Knockdown of BTB domain and CNC homolog 1 (BACH1) impedes Polo-like kinase 1 (PLK1)-induced phenotype in melanoma.

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