Supplementary Table from Ubiquitin Ligases Siah1a/2 Control Alveolar Macrophage Functions to Limit Carcinogen-Induced Lung Adenocarcinoma

Supplementary Table from Ubiquitin Ligases Siah1a/2 Control Alveolar Macrophage Functions to Limit Carcinogen-Induced Lung Adenocarcinoma

<p>Sup Table 3</p>

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Bibliografische gegevens
Hoofdauteur:	Marzia Scortegagna (16016032) (author)
Andere auteurs:	Yuanning Du (16016035) (author), Linda M. Bradley (16016038) (author), Kun Wang (16016041) (author), Alfredo Molinolo (16016044) (author), Eytan Ruppin (14941596) (author), Rabi Murad (16016047) (author), Ze'ev A. Ronai (15055921) (author)
Gepubliceerd in:	2025
Onderwerpen:	Cancer Tumor Biology Molecular and Cellular Biology Cell Signaling Gene Regulation Inducible gene expression Lung Cancer Tumor Microenvironment
Tags:	Voeg label toe Geen labels, Wees de eerste die dit record labelt!

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Supplementary Table from Ubiquitin Ligases Siah1a/2 Control Alveolar Macrophage Functions to Limit Carcinogen-Induced Lung Adenocarcinoma
door: Marzia Scortegagna (16016032)
Gepubliceerd in: (2025)

Supplementary Table from Ubiquitin Ligases Siah1a/2 Control Alveolar Macrophage Functions to Limit Carcinogen-Induced Lung Adenocarcinoma
door: Marzia Scortegagna (16016032)
Gepubliceerd in: (2025)

Supplementary Table from Ubiquitin Ligases Siah1a/2 Control Alveolar Macrophage Functions to Limit Carcinogen-Induced Lung Adenocarcinoma
door: Marzia Scortegagna (16016032)
Gepubliceerd in: (2025)

Supplementary video from Phagocytosis of Glioma Cells Enhances the Immunosuppressive Phenotype of Bone Marrow–Derived Macrophages
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Table S1 from Phagocytosis of Glioma Cells Enhances the Immunosuppressive Phenotype of Bone Marrow–Derived Macrophages
door: Min Wu (14962986)
Gepubliceerd in: (2025)

Table S2 from Phagocytosis of Glioma Cells Enhances the Immunosuppressive Phenotype of Bone Marrow–Derived Macrophages
door: Min Wu (14962986)
Gepubliceerd in: (2025)

Table S3 from Phagocytosis of Glioma Cells Enhances the Immunosuppressive Phenotype of Bone Marrow–Derived Macrophages
door: Min Wu (14962986)
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Supplementary Data from FOXA2 Cooperates with Mutant KRAS to Drive Invasive Mucinous Adenocarcinoma of the Lung
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Supplementary Table S3 from The Polyamine–Hypusine Circuit Controls an Oncogenic Translational Program Essential for Malignant Conversion in MYC-Driven Lymphoma
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Supplementary Table S4 from The Polyamine–Hypusine Circuit Controls an Oncogenic Translational Program Essential for Malignant Conversion in MYC-Driven Lymphoma
door: Shima Nakanishi (16336496)
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Supplementary Table 1 from N-myc–Mediated Translation Control Is a Therapeutic Vulnerability in Medulloblastoma
door: Duygu Kuzuoglu-Ozturk (14960213)
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Supplementary Table 2 from N-myc–Mediated Translation Control Is a Therapeutic Vulnerability in Medulloblastoma
door: Duygu Kuzuoglu-Ozturk (14960213)
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Supplementary Table 3 from N-myc–Mediated Translation Control Is a Therapeutic Vulnerability in Medulloblastoma
door: Duygu Kuzuoglu-Ozturk (14960213)
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Supplementary table S5 from HDAC5 Loss Enhances Phospholipid-Derived Arachidonic Acid Generation and Confers Sensitivity to cPLA2 Inhibition in Pancreatic Cancer
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Supplementary Table from Functional Analysis of <i>MET</i> Exon 14 Skipping Alteration in Cancer Invasion and Metastatic Dissemination
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Supplementary Table from Functional Analysis of <i>MET</i> Exon 14 Skipping Alteration in Cancer Invasion and Metastatic Dissemination
door: Feng Wang (44414)
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Supplementary Table from Functional Analysis of <i>MET</i> Exon 14 Skipping Alteration in Cancer Invasion and Metastatic Dissemination
door: Feng Wang (44414)
Gepubliceerd in: (2025)

Supplementary Table from Functional Analysis of <i>MET</i> Exon 14 Skipping Alteration in Cancer Invasion and Metastatic Dissemination
door: Feng Wang (44414)
Gepubliceerd in: (2025)

Supplementary Table S1 from <i>RBM10</i> Loss Promotes <i>EGFR</i>-Driven Lung Cancer and Confers Sensitivity to Spliceosome Inhibition
door: Yufang Bao (15386826)
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Supplementary Table S2 from <i>RBM10</i> Loss Promotes <i>EGFR</i>-Driven Lung Cancer and Confers Sensitivity to Spliceosome Inhibition
door: Yufang Bao (15386826)
Gepubliceerd in: (2025)

Supplementary Table S3 from <i>RBM10</i> Loss Promotes <i>EGFR</i>-Driven Lung Cancer and Confers Sensitivity to Spliceosome Inhibition
door: Yufang Bao (15386826)
Gepubliceerd in: (2025)

Supplementary Table S4 from <i>RBM10</i> Loss Promotes <i>EGFR</i>-Driven Lung Cancer and Confers Sensitivity to Spliceosome Inhibition
door: Yufang Bao (15386826)
Gepubliceerd in: (2025)

Supplementary Table S5 from <i>RBM10</i> Loss Promotes <i>EGFR</i>-Driven Lung Cancer and Confers Sensitivity to Spliceosome Inhibition
door: Yufang Bao (15386826)
Gepubliceerd in: (2025)

Figure S.1 from Loss of LOXL2 Promotes Uterine Hypertrophy and Tumor Progression by Enhancing H3K36ac-Dependent Gene Expression
door: Xufeng Lu (14960724)
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Figure S.2 from Loss of LOXL2 Promotes Uterine Hypertrophy and Tumor Progression by Enhancing H3K36ac-Dependent Gene Expression
door: Xufeng Lu (14960724)
Gepubliceerd in: (2025)

Figure S.3 from Loss of LOXL2 Promotes Uterine Hypertrophy and Tumor Progression by Enhancing H3K36ac-Dependent Gene Expression
door: Xufeng Lu (14960724)
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Figure S.4 from Loss of LOXL2 Promotes Uterine Hypertrophy and Tumor Progression by Enhancing H3K36ac-Dependent Gene Expression
door: Xufeng Lu (14960724)
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Figure S.5 from Loss of LOXL2 Promotes Uterine Hypertrophy and Tumor Progression by Enhancing H3K36ac-Dependent Gene Expression
door: Xufeng Lu (14960724)
Gepubliceerd in: (2025)

Figure S.6 from Loss of LOXL2 Promotes Uterine Hypertrophy and Tumor Progression by Enhancing H3K36ac-Dependent Gene Expression
door: Xufeng Lu (14960724)
Gepubliceerd in: (2025)

Figure S.7 from Loss of LOXL2 Promotes Uterine Hypertrophy and Tumor Progression by Enhancing H3K36ac-Dependent Gene Expression
door: Xufeng Lu (14960724)
Gepubliceerd in: (2025)

Figure S.8 from Loss of LOXL2 Promotes Uterine Hypertrophy and Tumor Progression by Enhancing H3K36ac-Dependent Gene Expression
door: Xufeng Lu (14960724)
Gepubliceerd in: (2025)

Table S1 from ENIGMA <i>CHEK2</i>gether Project: A Comprehensive Study Identifies Functionally Impaired <i>CHEK2</i> Germline Missense Variants Associated with Increased Breast Cancer Risk
door: Lenka Stolarova (16547259)
Gepubliceerd in: (2025)

Table S2 from ENIGMA <i>CHEK2</i>gether Project: A Comprehensive Study Identifies Functionally Impaired <i>CHEK2</i> Germline Missense Variants Associated with Increased Breast Cancer Risk
door: Lenka Stolarova (16547259)
Gepubliceerd in: (2025)

Table S3 from ENIGMA <i>CHEK2</i>gether Project: A Comprehensive Study Identifies Functionally Impaired <i>CHEK2</i> Germline Missense Variants Associated with Increased Breast Cancer Risk
door: Lenka Stolarova (16547259)
Gepubliceerd in: (2025)

Supplementary Table S1 from The EstroGene Database Reveals Diverse Temporal, Context-Dependent, and Bidirectional Estrogen Receptor Regulomes in Breast Cancer
door: Zheqi Li (14961021)
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Supplementary Table S10 from The EstroGene Database Reveals Diverse Temporal, Context-Dependent, and Bidirectional Estrogen Receptor Regulomes in Breast Cancer
door: Zheqi Li (14961021)
Gepubliceerd in: (2025)

Supplementary Table S11 from The EstroGene Database Reveals Diverse Temporal, Context-Dependent, and Bidirectional Estrogen Receptor Regulomes in Breast Cancer
door: Zheqi Li (14961021)
Gepubliceerd in: (2025)

Supplementary Table S2 from The EstroGene Database Reveals Diverse Temporal, Context-Dependent, and Bidirectional Estrogen Receptor Regulomes in Breast Cancer
door: Zheqi Li (14961021)
Gepubliceerd in: (2025)

Supplementary Table S3 from The EstroGene Database Reveals Diverse Temporal, Context-Dependent, and Bidirectional Estrogen Receptor Regulomes in Breast Cancer
door: Zheqi Li (14961021)
Gepubliceerd in: (2025)

Supplementary Table S4 from The EstroGene Database Reveals Diverse Temporal, Context-Dependent, and Bidirectional Estrogen Receptor Regulomes in Breast Cancer
door: Zheqi Li (14961021)
Gepubliceerd in: (2025)

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