Both live and heat-inactivated LGG supplementation attenuated lung tissue injury caused by M. pneumoniae infection.

Pre-oral supplementation with live or heat-inactivated strains of LGG at 3 dpi and 7 dpi (a) the M. pneumoniae colony counts in mouse BAL fluid, (b) p1 gene copies in lung tissue homogenate detecting by RT-PCR, (c) Lung histopathology...

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محفوظ في:

التفاصيل البيبلوغرافية
المؤلف الرئيسي:	Huanbing Long (19949784) (author)
مؤلفون آخرون:	Guiting He (19949787) (author), Jiarong He (11824972) (author), Ting feng Du (19949790) (author), Pengxiao Feng (19949793) (author), Cuiming Zhu (606670) (author)
منشور في:	2024
الموضوعات:	Medicine Microbiology Immunology Infectious Diseases Virology Space Science Environmental Sciences not elsewhere classified viral respiratory infections modulate immune responses encountered challenges due decrease pathogen load boosting immune responses alleviating pulmonary inflammation alleviate inflammatory injury limosilactobacillus reuteri </ lacticaseibacillus rhamnosus </ c mice model >- infected mice mycoplasma pneumoniae </ lactiplantibacillus plantarum </ probiotic lactobacillus administration significant protective effect lgg supplementation led mice infected c mice pneumoniae </ plantarum </ protective effect significant etiologies oral administration l </ protective efficacy siga ), secretory iga represents one pediatric patients orally administrated oral supplementation numerous studies neutrophil recruitment ncimb 8826 live lgg lgg resulted lgg exhibited lgg ), increased recruitment increased production inactivated lgg immunomodulatory ability five strains escalating resistance demonstrated efficacy data suggests clinical treatment alveolar macrophages administered orally acquired pneumonia
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Both live and heat-inactivated LGG supplementation attenuated lung tissue injury caused by <i>M</i>. <i>pneumoniae</i> infection.

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