Supplementary Table S4 from CD73-Dependent Adenosine Signaling through Adora2b Drives Immunosuppression in Ductal Pancreatic Cancer

Supplementary Table S4 from CD73-Dependent Adenosine Signaling through Adora2b Drives Immunosuppression in Ductal Pancreatic Cancer

<p>Table S4</p>

محفوظ في:

التفاصيل البيبلوغرافية
المؤلف الرئيسي:	Erika Y. Faraoni (14844897) (author)
مؤلفون آخرون:	Kanchan Singh (14844900) (author), Vidhi Chandra (14844903) (author), Olivereen Le Roux (14844906) (author), Yulin Dai (14844909) (author), Ismet Sahin (14844912) (author), Baylee J. O'Brien (14844915) (author), Lincoln N. Strickland (14844918) (author), Le Li (14844921) (author), Emily Vucic (14844924) (author), Amanda N. Warner (14844927) (author), Melissa Pruski (14844930) (author), Trent Clark (14844933) (author), George Van Buren (14844936) (author), Nirav C. Thosani (14844939) (author), John S. Bynon (14844942) (author), Curtis J. Wray (14844945) (author), Dafna Bar-Sagi (14844948) (author), Kyle L. Poulsen (14844951) (author), Lana A. Vornik (14844954) (author), Michelle I. Savage (14844957) (author), Shizuko Sei (14844960) (author), Altaf Mohammed (14844963) (author), Zhongming Zhao (12534149) (author), Powel H. Brown (14844966) (author), Tingting Mills (14844969) (author), Holger K. Eltzschig (14844972) (author), Florencia McAllister (14844975) (author), Jennifer M. Bailey-Lundberg (14844978) (author)
منشور في:	2025
الموضوعات:	Cancer Tumor Biology Immuno-oncology Methods and Technology Cancer Prevention Animal models of prevention Gastrointestinal Cancers Pancreatic cancer Immunology Immune responses to cancer Immunomodulation Immunotherapy Preclinical Models Animal models of cancer Tumor Heterogeneity Tumor Microenvironment Immune cells and the microenvironment
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مواد مشابهة

Supplementary Table S1 from CD73-Dependent Adenosine Signaling through Adora2b Drives Immunosuppression in Ductal Pancreatic Cancer
حسب: Erika Y. Faraoni (14844897)
منشور في: (2025)

Supplementary Table S3 from CD73-Dependent Adenosine Signaling through Adora2b Drives Immunosuppression in Ductal Pancreatic Cancer
حسب: Erika Y. Faraoni (14844897)
منشور في: (2025)

44d_RNAseq_data from A Pro-Regenerative Environment Triggers Premalignant to Malignant Transformation of Senescent Hepatocytes
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Supplementary Table from CDK2 Inhibition Enhances Antitumor Immunity by Increasing IFN Response to Endogenous Retroviruses
حسب: Yu Chen (1234761)
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Supplementary Table from CDK2 Inhibition Enhances Antitumor Immunity by Increasing IFN Response to Endogenous Retroviruses
حسب: Yu Chen (1234761)
منشور في: (2025)

Supplementary Table from CDK2 Inhibition Enhances Antitumor Immunity by Increasing IFN Response to Endogenous Retroviruses
حسب: Yu Chen (1234761)
منشور في: (2025)

Supplementary Table from CDK2 Inhibition Enhances Antitumor Immunity by Increasing IFN Response to Endogenous Retroviruses
حسب: Yu Chen (1234761)
منشور في: (2025)

Figure 1 from Extracellular Vesicle Secretion by Leukemia Cells <i>In Vivo</i> Promotes CLL Progression by Hampering Antitumor T-cell Responses
حسب: Ernesto Gargiulo (15134961)
منشور في: (2025)

Figure 2 from Extracellular Vesicle Secretion by Leukemia Cells <i>In Vivo</i> Promotes CLL Progression by Hampering Antitumor T-cell Responses
حسب: Ernesto Gargiulo (15134961)
منشور في: (2025)

Figure 3 from Extracellular Vesicle Secretion by Leukemia Cells <i>In Vivo</i> Promotes CLL Progression by Hampering Antitumor T-cell Responses
حسب: Ernesto Gargiulo (15134961)
منشور في: (2025)

Figure 4 from Extracellular Vesicle Secretion by Leukemia Cells <i>In Vivo</i> Promotes CLL Progression by Hampering Antitumor T-cell Responses
حسب: Ernesto Gargiulo (15134961)
منشور في: (2025)

Figure 5 from Extracellular Vesicle Secretion by Leukemia Cells <i>In Vivo</i> Promotes CLL Progression by Hampering Antitumor T-cell Responses
حسب: Ernesto Gargiulo (15134961)
منشور في: (2025)

Figure 6 from Extracellular Vesicle Secretion by Leukemia Cells <i>In Vivo</i> Promotes CLL Progression by Hampering Antitumor T-cell Responses
حسب: Ernesto Gargiulo (15134961)
منشور في: (2025)

Figure 7 from Extracellular Vesicle Secretion by Leukemia Cells <i>In Vivo</i> Promotes CLL Progression by Hampering Antitumor T-cell Responses
حسب: Ernesto Gargiulo (15134961)
منشور في: (2025)

Supplementary Table S1 from Extracellular Vesicle Secretion by Leukemia Cells <i>In Vivo</i> Promotes CLL Progression by Hampering Antitumor T-cell Responses
حسب: Ernesto Gargiulo (15134961)
منشور في: (2025)

Supplementary Table S2 from Extracellular Vesicle Secretion by Leukemia Cells <i>In Vivo</i> Promotes CLL Progression by Hampering Antitumor T-cell Responses
حسب: Ernesto Gargiulo (15134961)
منشور في: (2025)

Supplementary Table S3 from Extracellular Vesicle Secretion by Leukemia Cells <i>In Vivo</i> Promotes CLL Progression by Hampering Antitumor T-cell Responses
حسب: Ernesto Gargiulo (15134961)
منشور في: (2025)

Supplementary Table S4 from Extracellular Vesicle Secretion by Leukemia Cells <i>In Vivo</i> Promotes CLL Progression by Hampering Antitumor T-cell Responses
حسب: Ernesto Gargiulo (15134961)
منشور في: (2025)

Suppl video 1 from Neutrophil Conversion to a Tumor-Killing Phenotype Underpins Effective Microbial Therapy
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Suppl video 2 from Neutrophil Conversion to a Tumor-Killing Phenotype Underpins Effective Microbial Therapy
حسب: Andrew O. Yam (15293045)
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Suppl video 3 from Neutrophil Conversion to a Tumor-Killing Phenotype Underpins Effective Microbial Therapy
حسب: Andrew O. Yam (15293045)
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Suppl video 4 from Neutrophil Conversion to a Tumor-Killing Phenotype Underpins Effective Microbial Therapy
حسب: Andrew O. Yam (15293045)
منشور في: (2025)

Supplementary Table 1 from Neutrophil Conversion to a Tumor-Killing Phenotype Underpins Effective Microbial Therapy
حسب: Andrew O. Yam (15293045)
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Supplementary Figure 1 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
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Supplementary Figure 2 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
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Supplementary Figure 3 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
حسب: Wei Liu (20030)
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Supplementary Figure 4 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
حسب: Wei Liu (20030)
منشور في: (2025)

Supplementary Figure 5 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
حسب: Wei Liu (20030)
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Supplementary Figure 6 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
حسب: Wei Liu (20030)
منشور في: (2025)

Supplementary Figure7 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
حسب: Wei Liu (20030)
منشور في: (2025)

Supplementary Table 1 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
حسب: Wei Liu (20030)
منشور في: (2025)

Supplementary Table 2 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
حسب: Wei Liu (20030)
منشور في: (2025)

Supplementary Table 3 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
حسب: Wei Liu (20030)
منشور في: (2025)

Table S9 from Cross-talk between Myeloid and B Cells Shapes the Distinct Microenvironments of Primary and Secondary Liver Cancer
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Supplementary table S2 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
حسب: Leon P. Loevenich (14963948)
منشور في: (2025)

Supplementary table S3 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
حسب: Leon P. Loevenich (14963948)
منشور في: (2025)

Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
حسب: Leon P. Loevenich (14963948)
منشور في: (2025)

Supplementary video file from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
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منشور في: (2025)

Supplementary Table 1 from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
حسب: Emily K.W. Lo (15618846)
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Supplementary Table 2 from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
حسب: Emily K.W. Lo (15618846)
منشور في: (2025)

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