NeuroD1 overexpression abolished the effect of knocking down NeuroD1 on ferroptosis.

(A) Lipid peroxidation level in NeuroD1 re-expressed NeuroD1-knocked down HCC-LM3 cells, as assessed using flow cytometry. Data were presented as % of oxidized/oxidized + reduced MFI (F value = 323.9). (B)&...

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Bibliographic Details
Main Author:	Ping Huang (30439) (author)
Other Authors:	Wei Duan (101868) (author), Cao Ruan (16627266) (author), Lingxian Wang (4634977) (author), Rendy Hosea (17697125) (author), Zheng Wu (344022) (author), Jianting Zeng (17697128) (author), Shourong Wu (391683) (author), Vivi Kasim (391682) (author)
Published:	2023
Subjects:	Biophysics Biochemistry Cell Biology Genetics Molecular Biology Pharmacology Biotechnology Cancer Hematology Infectious Diseases Virology Computational Biology Chemical Sciences not elsewhere classified reducing lipid peroxide decreased lipid peroxide cell death threshold cell death caused aberrant redox homeostasis cell death resistance gpx4 signaling leads div >< p various tumor cells sensitize tumor cells mediated ferroptosis resistance tumor cells ferroptosis resistance drug resistance underlying mechanism transcriptional activity suppresses ferroptosis related genes recent studies oncogenic function neurodifferentiation factor key reductant highly expressed hepatocellular carcinoma gpx4 </ fully understood first discovered attracted attention also reveal
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<i>NeuroD1</i> overexpression abolished the effect of knocking down <i>NeuroD1</i> on ferroptosis.

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