Significantly enhanced KEGG pathways and GO function with cDEGs through the involvement of KGs linked to the pathogenetic processes of ccRCC.

Significantly enhanced KEGG pathways and GO function with cDEGs through the involvement of KGs linked to the pathogenetic processes of ccRCC.

<p>Significantly enhanced KEGG pathways and GO function with cDEGs through the involvement of KGs linked to the pathogenetic processes of ccRCC.</p>

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Bibliographic Details
Main Author:	Alvira Ajadee (19762306) (author)
Other Authors:	Sabkat Mahmud (19762309) (author), Md. Bayazid Hossain (19762312) (author), Reaz Ahmmed (19762315) (author), Md. Ahad Ali (16909085) (author), Md. Selim Reza (12368716) (author), Saroje Kumar Sarker (19762318) (author), Md. Nurul Haque Mollah (8946176) (author)
Published:	2024
Subjects:	Cell Biology Genetics Molecular Biology Pharmacology Developmental Biology Cancer Hematology Biological Sciences not elsewhere classified yet completely known mediated receptor proteins almost 2 years understand molecular mechanisms targeted different variants significant prognostic power literature review also control samples based causing key genes causing molecular functions though drug therapies tcga database showed gene regulatory network effective treatment strategy div >< p obtained 54 scdegs effective treatment molecular docking significant association microarray gene independent tcga gtex database different subtypes causing top 54 scdegs network analysis kgs based causing kgs useful inputs used protein umod </ transcriptional regulators therapeutic drugs slc12a3 </ slc12a1 </ serpina5 </ reduced set protein interaction prevalent subtype positively correlated plg </ outputs might negatively correlated may lead kidney cancer independent receptors genetic alterations expression datasets eno2 </ dovitinib .) crucial transcriptional cellular components biological processes best choice also supported aldob </ aldh6a1 </
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