EWAS in COPD by biomass-burning smoke exposure identifies low levels of endothelin-1 by hypermethylation of EDN1

<p>Exposure to smoke from biomass combustion is a significant environmental risk factor for chronic obstructive pulmonary disease (COPD). Through epigenome-wide association studies (EWAS), changes in DNA methylation levels associated with pathological conditions can be identified. We aimed to...

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Xehetasun bibliografikoak
Egile nagusia:	Salvador García-Carmona (20580797) (author)
Beste egile batzuk:	Ramcés Falfán-Valencia (837320) (author), Juan C. Fernández-López (21768534) (author), Alejandra Ramírez-Venegas (837317) (author), Fernando Morales-González (21768537) (author), María E. Ramírez-Díaz (20580803) (author), Filiberto Cruz-Vicente (20580806) (author), María L. Martínez-Gómez (20580809) (author), Rafael Hernández-Zenteno (8271168) (author), Ingrid Fricke-Galindo (13173933) (author), Raúl Sansores (21768540) (author), Gloria Pérez-Rubio (2611231) (author)
Argitaratua:	2025
Gaiak:	Medicine Cell Biology Genetics Molecular Biology Immunology Cancer Biological Sciences not elsewhere classified COPD methylation DNA EDN1 endothelin-1 EWAS
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EWAS in COPD by biomass-burning smoke exposure identifies low levels of endothelin-1 by hypermethylation of EDN1

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