Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis

<p>Differentially expressed genes in PDTOS4 versus PDTO cells in the presence of recombinant TGF-beta1</p>

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1. Verfasser: Leon P. Loevenich (14963948) (author)
Weitere Verfasser: Markus Tschurtschenthaler (14963951) (author), Matjaz Rokavec (14963954) (author), Miguel G. Silva (14963957) (author), Moritz Jesinghaus (14963960) (author), Thomas Kirchner (14963963) (author), Frederick Klauschen (14963966) (author), Dieter Saur (14953254) (author), Jens Neumann (14963969) (author), Heiko Hermeking (14963972) (author), Peter Jung (9030203) (author)
Veröffentlicht: 2025
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_version_ 1849927639284842496
author Leon P. Loevenich (14963948)
author2 Markus Tschurtschenthaler (14963951)
Matjaz Rokavec (14963954)
Miguel G. Silva (14963957)
Moritz Jesinghaus (14963960)
Thomas Kirchner (14963963)
Frederick Klauschen (14963966)
Dieter Saur (14953254)
Jens Neumann (14963969)
Heiko Hermeking (14963972)
Peter Jung (9030203)
author2_role author
author
author
author
author
author
author
author
author
author
author_facet Leon P. Loevenich (14963948)
Markus Tschurtschenthaler (14963951)
Matjaz Rokavec (14963954)
Miguel G. Silva (14963957)
Moritz Jesinghaus (14963960)
Thomas Kirchner (14963963)
Frederick Klauschen (14963966)
Dieter Saur (14953254)
Jens Neumann (14963969)
Heiko Hermeking (14963972)
Peter Jung (9030203)
author_role author
dc.creator.none.fl_str_mv Leon P. Loevenich (14963948)
Markus Tschurtschenthaler (14963951)
Matjaz Rokavec (14963954)
Miguel G. Silva (14963957)
Moritz Jesinghaus (14963960)
Thomas Kirchner (14963963)
Frederick Klauschen (14963966)
Dieter Saur (14953254)
Jens Neumann (14963969)
Heiko Hermeking (14963972)
Peter Jung (9030203)
dc.date.none.fl_str_mv 2025-11-24T22:45:39Z
dc.identifier.none.fl_str_mv 10.1158/0008-5472.30699591
dc.relation.none.fl_str_mv https://figshare.com/articles/dataset/Supplementary_table_S4_from_SMAD4_Loss_Induces_c-MYC_Mediated_NLE1_Upregulation_to_Support_Protein_Biosynthesis_Colorectal_Cancer_Growth_and_Metastasis/30699591
dc.rights.none.fl_str_mv CC BY
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Cancer
Cancer Biology
Tumor Biology
Molecular and Cellular Biology
Therapeutic Research and Development
Methods and Technology
Cancer Prevention
Cancer Interception
Biomarkers and intervention
Gastrointestinal Cancers
Colorectal cancer
Gene Regulation
Oncogenes & Tumor Suppressors
Preclinical Models
Animal models of cancer
Organoids
Progression, Invasion & Metastasis
Translational Research
dc.title.none.fl_str_mv Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
dc.type.none.fl_str_mv Dataset
info:eu-repo/semantics/publishedVersion
dataset
description <p>Differentially expressed genes in PDTOS4 versus PDTO cells in the presence of recombinant TGF-beta1</p>
eu_rights_str_mv openAccess
id Manara_a43e508cb2a37b8476b98317b231abbd
identifier_str_mv 10.1158/0008-5472.30699591
network_acronym_str Manara
network_name_str ManaraRepo
oai_identifier_str oai:figshare.com:article/30699591
publishDate 2025
repository.mail.fl_str_mv
repository.name.fl_str_mv
repository_id_str
rights_invalid_str_mv CC BY
spelling Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and MetastasisLeon P. Loevenich (14963948)Markus Tschurtschenthaler (14963951)Matjaz Rokavec (14963954)Miguel G. Silva (14963957)Moritz Jesinghaus (14963960)Thomas Kirchner (14963963)Frederick Klauschen (14963966)Dieter Saur (14953254)Jens Neumann (14963969)Heiko Hermeking (14963972)Peter Jung (9030203)CancerCancer BiologyTumor BiologyMolecular and Cellular BiologyTherapeutic Research and DevelopmentMethods and TechnologyCancer PreventionCancer InterceptionBiomarkers and interventionGastrointestinal CancersColorectal cancerGene RegulationOncogenes & Tumor SuppressorsPreclinical ModelsAnimal models of cancerOrganoidsProgression, Invasion & MetastasisTranslational Research<p>Differentially expressed genes in PDTOS4 versus PDTO cells in the presence of recombinant TGF-beta1</p>2025-11-24T22:45:39ZDatasetinfo:eu-repo/semantics/publishedVersiondataset10.1158/0008-5472.30699591https://figshare.com/articles/dataset/Supplementary_table_S4_from_SMAD4_Loss_Induces_c-MYC_Mediated_NLE1_Upregulation_to_Support_Protein_Biosynthesis_Colorectal_Cancer_Growth_and_Metastasis/30699591CC BYinfo:eu-repo/semantics/openAccessoai:figshare.com:article/306995912025-11-24T22:45:39Z
spellingShingle Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
Leon P. Loevenich (14963948)
Cancer
Cancer Biology
Tumor Biology
Molecular and Cellular Biology
Therapeutic Research and Development
Methods and Technology
Cancer Prevention
Cancer Interception
Biomarkers and intervention
Gastrointestinal Cancers
Colorectal cancer
Gene Regulation
Oncogenes & Tumor Suppressors
Preclinical Models
Animal models of cancer
Organoids
Progression, Invasion & Metastasis
Translational Research
status_str publishedVersion
title Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
title_full Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
title_fullStr Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
title_full_unstemmed Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
title_short Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
title_sort Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
topic Cancer
Cancer Biology
Tumor Biology
Molecular and Cellular Biology
Therapeutic Research and Development
Methods and Technology
Cancer Prevention
Cancer Interception
Biomarkers and intervention
Gastrointestinal Cancers
Colorectal cancer
Gene Regulation
Oncogenes & Tumor Suppressors
Preclinical Models
Animal models of cancer
Organoids
Progression, Invasion & Metastasis
Translational Research