<i>drp-1</i> mutation activates P<i>hsp-6</i>::GFP especially in muscles and neurons.

<i>drp-1</i> mutation activates P<i>hsp-6</i>::GFP especially in muscles and neurons.

<p>(<i>A</i>) P<i>hsp-6</i>::GFP expression in animals with indicated RNAi treatments. Scar bar, 0.2 mm. (<i>B</i>) P<i>hsp-6</i>::GFP expression in <i>drp-1(tm1108)</i> larvae. (<i>C</i>) P<i>hsp-6</i>::GFP ac...

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Bibliographic Details
Main Author: Xiaomeng Yang (702951) (author)
Other Authors: Ruichen Wei (21181914) (author), Fanfan Meng (8174745) (author), Dianchen Liu (21181917) (author), Xuan Gong (562445) (author), Gary Ruvkun (59736) (author), Wei Wei (21173) (author)
Published: 2025
Subjects:
Biochemistry
Cell Biology
Genetics
Science Policy
Environmental Sciences not elsewhere classified
Biological Sciences not elsewhere classified
performed rnai screens
genome instability lead
div >< p
specify mitochondrial abundance
potentially protective role
surveilling mitochondrial fission
regulate mitochondrial fission
mitochondrial fission surveillance
another 11 genes
300 random genes
mitigate dysregulation caused
caenorhabditis elegans </
chromosome segregation integrity
mitochondrial fission
elegans </
chromosome segregation
mitochondrial dynamics
potential role
chromosome missegregation
uneven activation
tightly regulated
screening paradigm
scale screen
regulatory pathways
possibly independent
physiological states
less explored
kinase library
cell division
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