Lineage Resolved Simulations for Comparing Simulated and Experimental Cell Viability Assays.

<p>A-C. Dose response simulations for an example drug (trametinib). Median (across simulation replicates) cell population dynamics for several doses (A) and cell population lineage dendrograms for specific doses: 0 uM (B) and 0.1 uM (C) are shown. D-G. Simulated dose responses measured in GR-v...

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التفاصيل البيبلوغرافية
المؤلف الرئيسي: Arnab Mutsuddy (22146227) (author)
مؤلفون آخرون: Jonah R. Huggins (13235400) (author), Aurore K. Amrit (22146230) (author), Atalanta Manuela Harley-Gasaway (22146233) (author), Cemal Erdem (11054081) (author), Evan T. Jones (22146236) (author), Olivia G. Laurine (22146239) (author), Jon C. Calhoun (22146242) (author), Marc R. Birtwistle (10825523) (author)
منشور في: 2025
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_version_ 1852017156389076992
author Arnab Mutsuddy (22146227)
author2 Jonah R. Huggins (13235400)
Aurore K. Amrit (22146230)
Atalanta Manuela Harley-Gasaway (22146233)
Cemal Erdem (11054081)
Evan T. Jones (22146236)
Olivia G. Laurine (22146239)
Jon C. Calhoun (22146242)
Marc R. Birtwistle (10825523)
author2_role author
author
author
author
author
author
author
author
author_facet Arnab Mutsuddy (22146227)
Jonah R. Huggins (13235400)
Aurore K. Amrit (22146230)
Atalanta Manuela Harley-Gasaway (22146233)
Cemal Erdem (11054081)
Evan T. Jones (22146236)
Olivia G. Laurine (22146239)
Jon C. Calhoun (22146242)
Marc R. Birtwistle (10825523)
author_role author
dc.creator.none.fl_str_mv Arnab Mutsuddy (22146227)
Jonah R. Huggins (13235400)
Aurore K. Amrit (22146230)
Atalanta Manuela Harley-Gasaway (22146233)
Cemal Erdem (11054081)
Evan T. Jones (22146236)
Olivia G. Laurine (22146239)
Jon C. Calhoun (22146242)
Marc R. Birtwistle (10825523)
dc.date.none.fl_str_mv 2025-08-29T17:59:22Z
dc.identifier.none.fl_str_mv 10.1371/journal.pcbi.1013156.g002
dc.relation.none.fl_str_mv https://figshare.com/articles/figure/Lineage_Resolved_Simulations_for_Comparing_Simulated_and_Experimental_Cell_Viability_Assays_/30014444
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Biophysics
Cell Biology
Genetics
Molecular Biology
Pharmacology
Biotechnology
Cancer
Hematology
Biological Sciences not elsewhere classified
Chemical Sciences not elsewhere classified
Information Systems not elsewhere classified
tracks simulated division
strong growth inhibition
slowly dividing cells
mother cells prior
measure cumulative division
four targeted anti
erk pathway activity
better understanding determinants
receptor inhibitor response
egfr inhibitor ).
3k inhibitor ),
mechanistic computational models
xlink "> data
cell viability assays
cell cycle may
simulations show subpopulations
rapidly dividing phenotype
drug treatment impinging
data inconsistencies suggest
previously developed model
mek inhibitor
suggest causes
mechanistic modeling
experimental data
data comparison
cell proliferation
cell lineages
6 inhibitor
work lays
widely available
treated conditions
silico </
overall lack
often muddled
mammalian single
lineage tracing
induced signaling
direct model
detailed single
death signaling
death events
critical determinant
cellular heterogeneity
cellular balance
cancer drugs
dc.title.none.fl_str_mv Lineage Resolved Simulations for Comparing Simulated and Experimental Cell Viability Assays.
dc.type.none.fl_str_mv Image
Figure
info:eu-repo/semantics/publishedVersion
image
description <p>A-C. Dose response simulations for an example drug (trametinib). Median (across simulation replicates) cell population dynamics for several doses (A) and cell population lineage dendrograms for specific doses: 0 uM (B) and 0.1 uM (C) are shown. D-G. Simulated dose responses measured in GR-value for four drugs compared experimental data. Error bars are standard error taken from original experimental data, or as calculated across simulation replicates (n=10).</p>
eu_rights_str_mv openAccess
id Manara_c2cab8f7d530d183e227baefd43dbf86
identifier_str_mv 10.1371/journal.pcbi.1013156.g002
network_acronym_str Manara
network_name_str ManaraRepo
oai_identifier_str oai:figshare.com:article/30014444
publishDate 2025
repository.mail.fl_str_mv
repository.name.fl_str_mv
repository_id_str
rights_invalid_str_mv CC BY 4.0
spelling Lineage Resolved Simulations for Comparing Simulated and Experimental Cell Viability Assays.Arnab Mutsuddy (22146227)Jonah R. Huggins (13235400)Aurore K. Amrit (22146230)Atalanta Manuela Harley-Gasaway (22146233)Cemal Erdem (11054081)Evan T. Jones (22146236)Olivia G. Laurine (22146239)Jon C. Calhoun (22146242)Marc R. Birtwistle (10825523)BiophysicsCell BiologyGeneticsMolecular BiologyPharmacologyBiotechnologyCancerHematologyBiological Sciences not elsewhere classifiedChemical Sciences not elsewhere classifiedInformation Systems not elsewhere classifiedtracks simulated divisionstrong growth inhibitionslowly dividing cellsmother cells priormeasure cumulative divisionfour targeted antierk pathway activitybetter understanding determinantsreceptor inhibitor responseegfr inhibitor ).3k inhibitor ),mechanistic computational modelsxlink "> datacell viability assayscell cycle maysimulations show subpopulationsrapidly dividing phenotypedrug treatment impingingdata inconsistencies suggestpreviously developed modelmek inhibitorsuggest causesmechanistic modelingexperimental datadata comparisoncell proliferationcell lineages6 inhibitorwork layswidely availabletreated conditionssilico </overall lackoften muddledmammalian singlelineage tracinginduced signalingdirect modeldetailed singledeath signalingdeath eventscritical determinantcellular heterogeneitycellular balancecancer drugs<p>A-C. Dose response simulations for an example drug (trametinib). Median (across simulation replicates) cell population dynamics for several doses (A) and cell population lineage dendrograms for specific doses: 0 uM (B) and 0.1 uM (C) are shown. D-G. Simulated dose responses measured in GR-value for four drugs compared experimental data. Error bars are standard error taken from original experimental data, or as calculated across simulation replicates (n=10).</p>2025-08-29T17:59:22ZImageFigureinfo:eu-repo/semantics/publishedVersionimage10.1371/journal.pcbi.1013156.g002https://figshare.com/articles/figure/Lineage_Resolved_Simulations_for_Comparing_Simulated_and_Experimental_Cell_Viability_Assays_/30014444CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/300144442025-08-29T17:59:22Z
spellingShingle Lineage Resolved Simulations for Comparing Simulated and Experimental Cell Viability Assays.
Arnab Mutsuddy (22146227)
Biophysics
Cell Biology
Genetics
Molecular Biology
Pharmacology
Biotechnology
Cancer
Hematology
Biological Sciences not elsewhere classified
Chemical Sciences not elsewhere classified
Information Systems not elsewhere classified
tracks simulated division
strong growth inhibition
slowly dividing cells
mother cells prior
measure cumulative division
four targeted anti
erk pathway activity
better understanding determinants
receptor inhibitor response
egfr inhibitor ).
3k inhibitor ),
mechanistic computational models
xlink "> data
cell viability assays
cell cycle may
simulations show subpopulations
rapidly dividing phenotype
drug treatment impinging
data inconsistencies suggest
previously developed model
mek inhibitor
suggest causes
mechanistic modeling
experimental data
data comparison
cell proliferation
cell lineages
6 inhibitor
work lays
widely available
treated conditions
silico </
overall lack
often muddled
mammalian single
lineage tracing
induced signaling
direct model
detailed single
death signaling
death events
critical determinant
cellular heterogeneity
cellular balance
cancer drugs
status_str publishedVersion
title Lineage Resolved Simulations for Comparing Simulated and Experimental Cell Viability Assays.
title_full Lineage Resolved Simulations for Comparing Simulated and Experimental Cell Viability Assays.
title_fullStr Lineage Resolved Simulations for Comparing Simulated and Experimental Cell Viability Assays.
title_full_unstemmed Lineage Resolved Simulations for Comparing Simulated and Experimental Cell Viability Assays.
title_short Lineage Resolved Simulations for Comparing Simulated and Experimental Cell Viability Assays.
title_sort Lineage Resolved Simulations for Comparing Simulated and Experimental Cell Viability Assays.
topic Biophysics
Cell Biology
Genetics
Molecular Biology
Pharmacology
Biotechnology
Cancer
Hematology
Biological Sciences not elsewhere classified
Chemical Sciences not elsewhere classified
Information Systems not elsewhere classified
tracks simulated division
strong growth inhibition
slowly dividing cells
mother cells prior
measure cumulative division
four targeted anti
erk pathway activity
better understanding determinants
receptor inhibitor response
egfr inhibitor ).
3k inhibitor ),
mechanistic computational models
xlink "> data
cell viability assays
cell cycle may
simulations show subpopulations
rapidly dividing phenotype
drug treatment impinging
data inconsistencies suggest
previously developed model
mek inhibitor
suggest causes
mechanistic modeling
experimental data
data comparison
cell proliferation
cell lineages
6 inhibitor
work lays
widely available
treated conditions
silico </
overall lack
often muddled
mammalian single
lineage tracing
induced signaling
direct model
detailed single
death signaling
death events
critical determinant
cellular heterogeneity
cellular balance
cancer drugs