Impact of ATG16L1, FAM126A and INPP5K depletion on early, intermediate and late stages of IAV infection (related to Fig 4).

<p><b>A, B.</b> A549 cells were treated with the indicated siRNA for 48 h and subsequently infected with WSN at a MOI of 5 PFU/cell, in the presence (+) or absence (−) of cycloheximide (CHX). (A) At 6 hpi, total cell lysates were prepared and the steady-state levels of viral protei...

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Bibliographic Details
Main Author:	Carla Alemany (21728867) (author)
Other Authors:	Juliane Da Graça (21728870) (author), Quentin Giai Gianetto (9262686) (author), Maud Dupont (21728873) (author), Sylvain Paisant (12602528) (author), Thibaut Douché (3576971) (author), Catherine Isel (13347) (author), Cédric Delevoye (88294) (author), Lydia Danglot (4891612) (author), Mariette Matondo (3856441) (author), Etienne Morel (35915) (author), Jean-Baptiste Brault (17847395) (author), Nadia Naffakh (29879) (author)
Published:	2025
Subjects:	Biochemistry Microbiology Cell Biology Molecular Biology Biotechnology Mental Health Infectious Diseases Plant Biology Virology Biological Sciences not elsewhere classified iav infectious particles iav infection remodels small gtpase rab11a rab11a proximity interactome rab11a fusion protein cellular pi4p levels infection induces cellular regulators coordinating rab11a xlink "> viral ribonucleoproteins viral assembly significantly enriched provide evidence plasma membrane pivotal role pi4p punctae performed biotin investigated viral infected cells induced perturbations genomic rnas essential platform ensure delivery enriched membranes endoplasmic reticulum dependent manner data extend
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Impact of ATG16L1, FAM126A and INPP5K depletion on early, intermediate and late stages of IAV infection (related to Fig 4).

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