Overexpression of miR-223-5p rescued the effects of CHAC2.

<p>BMSCs overexpressing CHAC2 were constructed, and miRNA-223-5p was transfected into BMSCs. The cells were then cultured under hypoxic conditions. Apoptosis in BMSCs was evaluated using Western blot (WB), TUNEL staining, and flow cytometry. Additionally, the activation of the Wnt/β-catenin si...

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Bibliographic Details
Main Author:	Jiafei Yang (19736131) (author)
Other Authors:	Tianjiu Zhang (15925466) (author), Xingtao Zhu (20613855) (author), Zhexi He (20613858) (author), Xu Jiang (1602100) (author), Song Yu (3073554) (author), Huajian Gu (20144457) (author)
Published:	2025
Subjects:	Biochemistry Medicine Cell Biology Genetics Molecular Biology Physiology Immunology Developmental Biology Marine Biology Cancer Science Policy Biological Sciences not elsewhere classified Chemical Sciences not elsewhere classified luciferase reporter assays femoral neck ligation disrupted blood supply assessed using qpcr improving bone repair xlink "> legg directly targeting chac2 bmscs inhibited hypoxia 5p inhibits hypoxia targeting chac2 regulating hypoxia promotes repair western blotting transfected bmscs study investigates results indicate reduced necrosis promising target overexpressing bmscs joint deformity induced apoptosis hypoxic exposure hypoxic conditions findings suggest enhancing osteogenesis early osteoarthritis bmscs ). bmsc apoptosis 5p mimics
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Overexpression of miR-223-5p rescued the effects of CHAC2.

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