Supplementary Table S5 from Deficiency of the Polycomb Protein RYBP and TET Methylcytosine Oxidases Promotes Extensive CpG Island Hypermethylation and Malignant Transformation

Supplementary Table S5 from Deficiency of the Polycomb Protein RYBP and TET Methylcytosine Oxidases Promotes Extensive CpG Island Hypermethylation and Malignant Transformation

<p>DMRs</p>

Αποθηκεύτηκε σε:

Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριος συγγραφέας:	Wei Cui (16721390) (author)
Άλλοι συγγραφείς:	Zhijun Huang (16721393) (author), Seung-Gi Jin (16721396) (author), Jennifer Johnson (16721399) (author), Kin H. Lau (16721402) (author), Galen Hostetter (15053070) (author), Gerd P. Pfeifer (15124315) (author)
Έκδοση:	2025
Θέματα:	Cancer Cancer Risk Factors Molecular and Cellular Biology Epigenetics DNA methylation Gene Regulation Mechanisms of transcription Lung Cancer
Ετικέτες:	Προσθήκη ετικέτας Δεν υπάρχουν, Καταχωρήστε ετικέτα πρώτοι!

Παρόμοια τεκμήρια

Supplementary Table S3 from Deficiency of the Polycomb Protein RYBP and TET Methylcytosine Oxidases Promotes Extensive CpG Island Hypermethylation and Malignant Transformation
ανά: Wei Cui (16721390)
Έκδοση: (2025)

Supplementary Table S4 from Deficiency of the Polycomb Protein RYBP and TET Methylcytosine Oxidases Promotes Extensive CpG Island Hypermethylation and Malignant Transformation
ανά: Wei Cui (16721390)
Έκδοση: (2025)

Supplementary Table S6 from Deficiency of the Polycomb Protein RYBP and TET Methylcytosine Oxidases Promotes Extensive CpG Island Hypermethylation and Malignant Transformation
ανά: Wei Cui (16721390)
Έκδοση: (2025)

Supplementary Table S7 from Deficiency of the Polycomb Protein RYBP and TET Methylcytosine Oxidases Promotes Extensive CpG Island Hypermethylation and Malignant Transformation
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Έκδοση: (2025)

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Supplementary Figure S10 from Elucidating the Genetic Landscape of Oral Leukoplakia to Predict Malignant Transformation
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Supplementary Figure S11 from Elucidating the Genetic Landscape of Oral Leukoplakia to Predict Malignant Transformation
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Supplementary Figure S12 from Elucidating the Genetic Landscape of Oral Leukoplakia to Predict Malignant Transformation
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Supplementary Figure S2 from Elucidating the Genetic Landscape of Oral Leukoplakia to Predict Malignant Transformation
ανά: Leon J. Wils (15055780)
Έκδοση: (2025)

Supplementary Figure S4 from Elucidating the Genetic Landscape of Oral Leukoplakia to Predict Malignant Transformation
ανά: Leon J. Wils (15055780)
Έκδοση: (2025)

Supplementary Figure S5 from Elucidating the Genetic Landscape of Oral Leukoplakia to Predict Malignant Transformation
ανά: Leon J. Wils (15055780)
Έκδοση: (2025)

Supplementary Figure S6 from Elucidating the Genetic Landscape of Oral Leukoplakia to Predict Malignant Transformation
ανά: Leon J. Wils (15055780)
Έκδοση: (2025)

Supplementary Figure S7 from Elucidating the Genetic Landscape of Oral Leukoplakia to Predict Malignant Transformation
ανά: Leon J. Wils (15055780)
Έκδοση: (2025)

Supplementary Figure S8 from Elucidating the Genetic Landscape of Oral Leukoplakia to Predict Malignant Transformation
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Έκδοση: (2025)

Supplementary Figure S9 from Elucidating the Genetic Landscape of Oral Leukoplakia to Predict Malignant Transformation
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Suppl. Figure 4 from Endoglin, a Novel Biomarker and Therapeutical Target to Prevent Malignant Peripheral Nerve Sheath Tumor Growth and Metastasis
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Suppl. Figure 5 from Endoglin, a Novel Biomarker and Therapeutical Target to Prevent Malignant Peripheral Nerve Sheath Tumor Growth and Metastasis
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Suppl. Figure 6 from Endoglin, a Novel Biomarker and Therapeutical Target to Prevent Malignant Peripheral Nerve Sheath Tumor Growth and Metastasis
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Supplementary Data 1 from Endoglin, a Novel Biomarker and Therapeutical Target to Prevent Malignant Peripheral Nerve Sheath Tumor Growth and Metastasis
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Supplementary Data 2 from Endoglin, a Novel Biomarker and Therapeutical Target to Prevent Malignant Peripheral Nerve Sheath Tumor Growth and Metastasis
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Supplementary Data 3 from Endoglin, a Novel Biomarker and Therapeutical Target to Prevent Malignant Peripheral Nerve Sheath Tumor Growth and Metastasis
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