Figure 1 from Spatially Resolved Transcriptomics Deconvolutes Prognostic Histological Subgroups in Patients with Colorectal Cancer and Synchronous Liver Metastases

Figure 1 from Spatially Resolved Transcriptomics Deconvolutes Prognostic Histological Subgroups in Patients with Colorectal Cancer and Synchronous Liver Metastases

<p>Mutational characterization of primary colorectal cancer and CRLM. <b>A,</b> Venn diagram demonstrating primary colorectal cancer and CRLM that underwent genomic analysis. Co-Barplot illustrating most frequently mutated genes across 13 matched primary colorectal cancer and CRLM,...

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Bibliografski detalji
Glavni autor:	Colin S. Wood (15293247) (author)
Daljnji autori:	Kathryn A.F. Pennel (15293250) (author), Holly Leslie (15293253) (author), Assya Legrini (15293256) (author), Andrew J. Cameron (15046037) (author), Lydia Melissourgou-Syka (15293259) (author), Jean A. Quinn (15293262) (author), Hester C. van Wyk (15293265) (author), Jennifer Hay (15293268) (author), Antonia K. Roseweir (15293271) (author), Colin Nixon (14949516) (author), Campbell S.D. Roxburgh (15293274) (author), Donald C. McMillan (15293277) (author), Andrew V. Biankin (14937726) (author), Owen J. Sansom (14949234) (author), Paul G. Horgan (15293280) (author), Joanne Edwards (14939606) (author), Colin W. Steele (15293283) (author), Nigel B. Jamieson (15046064) (author)
Izdano:	2025
Teme:	Cancer Tumor Biology Cancer Detection and Diagnosis Methods and Technology Biomarkers Diagnostic biomarkers Gastrointestinal Cancers Colorectal cancer Gene Technologies High-throughput sequencing Progression, Invasion & Metastasis
Oznake:	Dodaj oznaku Bez oznaka, Budi prvi tko označuje ovaj zapis!

Slični predmeti

Figure 2 from Spatially Resolved Transcriptomics Deconvolutes Prognostic Histological Subgroups in Patients with Colorectal Cancer and Synchronous Liver Metastases
od: Colin S. Wood (15293247)
Izdano: (2025)

Figure 3 from Spatially Resolved Transcriptomics Deconvolutes Prognostic Histological Subgroups in Patients with Colorectal Cancer and Synchronous Liver Metastases
od: Colin S. Wood (15293247)
Izdano: (2025)

Figure 4 from Spatially Resolved Transcriptomics Deconvolutes Prognostic Histological Subgroups in Patients with Colorectal Cancer and Synchronous Liver Metastases
od: Colin S. Wood (15293247)
Izdano: (2025)

Figure 5 from Spatially Resolved Transcriptomics Deconvolutes Prognostic Histological Subgroups in Patients with Colorectal Cancer and Synchronous Liver Metastases
od: Colin S. Wood (15293247)
Izdano: (2025)

Figure 6 from Spatially Resolved Transcriptomics Deconvolutes Prognostic Histological Subgroups in Patients with Colorectal Cancer and Synchronous Liver Metastases
od: Colin S. Wood (15293247)
Izdano: (2025)

Table 1 from Spatially Resolved Transcriptomics Deconvolutes Prognostic Histological Subgroups in Patients with Colorectal Cancer and Synchronous Liver Metastases
od: Colin S. Wood (15293247)
Izdano: (2025)

Supplementary Table 4: SpatialDecon derived immune cell counts from Spatially Resolved Transcriptomics Deconvolutes Prognostic Histological Subgroups in Patients with Colorectal Cancer and Synchronous Liver Metastases
od: Colin S. Wood (15293247)
Izdano: (2025)

Supplementry Table 3: REACTOME Gene Set Enrichment Analysis results from Spatially Resolved Transcriptomics Deconvolutes Prognostic Histological Subgroups in Patients with Colorectal Cancer and Synchronous Liver Metastases
od: Colin S. Wood (15293247)
Izdano: (2025)

Supplemental Tables 1-8 from Colorectal Cancer Metastases in the Liver Establish Immunosuppressive Spatial Networking between Tumor-Associated <i>SPP1</i><sup>+</sup> Macrophages and Fibroblasts
od: Anuja Sathe (15028097)
Izdano: (2025)

Supplementary Tables S1-S15 from Metastatic Colorectal Cancer Treatment Response Evaluation by Ultra-Deep Sequencing of Cell-Free DNA and Matched White Blood Cells
od: Iris van 't Erve (15054503)
Izdano: (2025)

Supplementary Data from Genetic Modulation of <i>BET1L</i> Confers Colorectal Cancer Susceptibility by Reducing miRNA Binding and m<sup>6</sup>A Modification
od: Shuwei Li (16437116)
Izdano: (2025)

Supplementary table S2 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
od: Leon P. Loevenich (14963948)
Izdano: (2025)

Supplementary table S3 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
od: Leon P. Loevenich (14963948)
Izdano: (2025)

Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
od: Leon P. Loevenich (14963948)
Izdano: (2025)

Supplementary video file from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
od: Leon P. Loevenich (14963948)
Izdano: (2025)

Supplementary Figure S7 from <i>NPY</i> Methylated ctDNA is a Promising Biomarker for Treatment Response Monitoring in Metastatic Colorectal Cancer
od: Katleen Janssens (15053356)
Izdano: (2025)

Supplementary Figure S1 from An Immune-Related Gene Expression Signature Predicts Benefit from Adding Atezolizumab to FOLFOXIRI plus Bevacizumab in Metastatic Colorectal Cancer
od: Carlotta Antoniotti (15352506)
Izdano: (2025)

Supplementary Figure S2 from An Immune-Related Gene Expression Signature Predicts Benefit from Adding Atezolizumab to FOLFOXIRI plus Bevacizumab in Metastatic Colorectal Cancer
od: Carlotta Antoniotti (15352506)
Izdano: (2025)

Supplementary Figure S3 from An Immune-Related Gene Expression Signature Predicts Benefit from Adding Atezolizumab to FOLFOXIRI plus Bevacizumab in Metastatic Colorectal Cancer
od: Carlotta Antoniotti (15352506)
Izdano: (2025)

Figure S1 from <i>De Novo</i> and Histologically Transformed Small-Cell Lung Cancer Is Sensitive to Lurbinectedin Treatment Through the Modulation of EMT and NOTCH Signaling Pathways
od: Subhamoy Chakraborty (16648124)
Izdano: (2025)

Figure S2 from <i>De Novo</i> and Histologically Transformed Small-Cell Lung Cancer Is Sensitive to Lurbinectedin Treatment Through the Modulation of EMT and NOTCH Signaling Pathways
od: Subhamoy Chakraborty (16648124)
Izdano: (2025)

Figure S3 from <i>De Novo</i> and Histologically Transformed Small-Cell Lung Cancer Is Sensitive to Lurbinectedin Treatment Through the Modulation of EMT and NOTCH Signaling Pathways
od: Subhamoy Chakraborty (16648124)
Izdano: (2025)

Supplementary Tables 1-3 from <i>De Novo</i> and Histologically Transformed Small-Cell Lung Cancer Is Sensitive to Lurbinectedin Treatment Through the Modulation of EMT and NOTCH Signaling Pathways
od: Subhamoy Chakraborty (16648124)
Izdano: (2025)

Supplementary Table S1 from Exercise Training Reduces the Inflammatory Response and Promotes Intestinal Mucosa-Associated Immunity in Lynch Syndrome
od: Nan Deng (17314330)
Izdano: (2025)

Supplementary Table S2 from Exercise Training Reduces the Inflammatory Response and Promotes Intestinal Mucosa-Associated Immunity in Lynch Syndrome
od: Nan Deng (17314330)
Izdano: (2025)

Supplementary Table S3 from Exercise Training Reduces the Inflammatory Response and Promotes Intestinal Mucosa-Associated Immunity in Lynch Syndrome
od: Nan Deng (17314330)
Izdano: (2025)

Supplementary Table S4 from Exercise Training Reduces the Inflammatory Response and Promotes Intestinal Mucosa-Associated Immunity in Lynch Syndrome
od: Nan Deng (17314330)
Izdano: (2025)

Supplementary Table S5 from Exercise Training Reduces the Inflammatory Response and Promotes Intestinal Mucosa-Associated Immunity in Lynch Syndrome
od: Nan Deng (17314330)
Izdano: (2025)

Supplementary Table S6 from Exercise Training Reduces the Inflammatory Response and Promotes Intestinal Mucosa-Associated Immunity in Lynch Syndrome
od: Nan Deng (17314330)
Izdano: (2025)

Supplementary Table S7 from Exercise Training Reduces the Inflammatory Response and Promotes Intestinal Mucosa-Associated Immunity in Lynch Syndrome
od: Nan Deng (17314330)
Izdano: (2025)

Supplementary Table S8 from Exercise Training Reduces the Inflammatory Response and Promotes Intestinal Mucosa-Associated Immunity in Lynch Syndrome
od: Nan Deng (17314330)
Izdano: (2025)

Supplementary Table S1 from Validation of the Clinical Use of GIScar, an Academic-developed Genomic Instability Score Predicting Sensitivity to Maintenance Olaparib for Ovarian Cancer
od: Raphaël Leman (17314506)
Izdano: (2025)

Supplementary Table S2 from Validation of the Clinical Use of GIScar, an Academic-developed Genomic Instability Score Predicting Sensitivity to Maintenance Olaparib for Ovarian Cancer
od: Raphaël Leman (17314506)
Izdano: (2025)

Supplementary Table S3 from Validation of the Clinical Use of GIScar, an Academic-developed Genomic Instability Score Predicting Sensitivity to Maintenance Olaparib for Ovarian Cancer
od: Raphaël Leman (17314506)
Izdano: (2025)

Supplementary Table S4 from Validation of the Clinical Use of GIScar, an Academic-developed Genomic Instability Score Predicting Sensitivity to Maintenance Olaparib for Ovarian Cancer
od: Raphaël Leman (17314506)
Izdano: (2025)

Supplementary Table S5 from Validation of the Clinical Use of GIScar, an Academic-developed Genomic Instability Score Predicting Sensitivity to Maintenance Olaparib for Ovarian Cancer
od: Raphaël Leman (17314506)
Izdano: (2025)

Supplementary Table S6 from Validation of the Clinical Use of GIScar, an Academic-developed Genomic Instability Score Predicting Sensitivity to Maintenance Olaparib for Ovarian Cancer
od: Raphaël Leman (17314506)
Izdano: (2025)

Supplementary Figures 1 from Spatial Immunoprofiling of Adenoid Cystic Carcinoma Reveals B7-H4 Is a Therapeutic Target for Aggressive Tumors
od: Luana Guimaraes Sousa (16534377)
Izdano: (2025)

Supplementary Data 1 from The Mutational Landscape of Cancer's Vulnerability to Ionizing Radiation
od: Priyanka Gopal (15050862)
Izdano: (2025)

Supplementary Data 2 from The Mutational Landscape of Cancer's Vulnerability to Ionizing Radiation
od: Priyanka Gopal (15050862)
Izdano: (2025)

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