Mechanistic insights into FUNDC2P4 and HSF1 regulation of EMT in residual hepatocellular carcinoma following insufficient radiofrequency ablation

<p>Insufficient radiofrequency ablation (IRFA) increases the risk of hepatocellular carcinoma (HCC) recurrence and metastasis. Our previous study revealed that IRFA downregulates FUNDC2P4 and promotes epithelial-mesenchymal transition (EMT), but the mechanisms remain unclear.</p> <p&g...

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Main Author: Ni Tang (196698) (author)
Other Authors: Liangxia Yang (22180323) (author), Chengmou Huang (22180326) (author), Lu Yang (30847) (author), Liangxing Yang (22180329) (author), Haihang Zhang (5095112) (author), Jiangzheng Zeng (22180332) (author)
Published: 2025
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_version_ 1852016989261791232
author Ni Tang (196698)
author2 Liangxia Yang (22180323)
Chengmou Huang (22180326)
Lu Yang (30847)
Liangxing Yang (22180329)
Haihang Zhang (5095112)
Jiangzheng Zeng (22180332)
author2_role author
author
author
author
author
author
author_facet Ni Tang (196698)
Liangxia Yang (22180323)
Chengmou Huang (22180326)
Lu Yang (30847)
Liangxing Yang (22180329)
Haihang Zhang (5095112)
Jiangzheng Zeng (22180332)
author_role author
dc.creator.none.fl_str_mv Ni Tang (196698)
Liangxia Yang (22180323)
Chengmou Huang (22180326)
Lu Yang (30847)
Liangxing Yang (22180329)
Haihang Zhang (5095112)
Jiangzheng Zeng (22180332)
dc.date.none.fl_str_mv 2025-09-04T06:20:06Z
dc.identifier.none.fl_str_mv 10.6084/m9.figshare.30050205.v1
dc.relation.none.fl_str_mv https://figshare.com/articles/dataset/Mechanistic_insights_into_FUNDC2P4_and_HSF1_regulation_of_EMT_in_residual_hepatocellular_carcinoma_following_insufficient_radiofrequency_ablation/30050205
dc.rights.none.fl_str_mv CC BY 4.0
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Biophysics
Medicine
Cell Biology
Genetics
Developmental Biology
Cancer
Infectious Diseases
Biological Sciences not elsewhere classified
Chemical Sciences not elsewhere classified
Hepatocellular carcinoma
insufficient radiofrequency ablation
epithelial-mesenchymal transition
FUNDC2P4
HSF1
dc.title.none.fl_str_mv Mechanistic insights into FUNDC2P4 and HSF1 regulation of EMT in residual hepatocellular carcinoma following insufficient radiofrequency ablation
dc.type.none.fl_str_mv Dataset
info:eu-repo/semantics/publishedVersion
dataset
description <p>Insufficient radiofrequency ablation (IRFA) increases the risk of hepatocellular carcinoma (HCC) recurrence and metastasis. Our previous study revealed that IRFA downregulates FUNDC2P4 and promotes epithelial-mesenchymal transition (EMT), but the mechanisms remain unclear.</p> <p>We simulated IRFA <i>in vitro</i> using a thermostatic water bath and assessed RNA and protein expression via qRT-PCR and Western blotting. Transcriptome analysis of LV-FUNDC2P4-transfected Huh7 cells, along with computational prediction, truncated mutants, and dual-luciferase assays, was performed to investigate the competitive binding of FUNDC2P4 and heat shock factor 1 (HSF1) to the Chromobox protein homolog 7 (CBX7) promoter. Functional assays, including CCK-8, Transwell, and wound-healing assays, evaluated cell proliferation, invasion, and migration.</p> <p>IRFA induced EMT, downregulated FUNDC2P4, and upregulated HSF1. Gene set enrichment analysis indicated EMT suppression in Huh7 cells overexpressing FUNDC2P4. Overexpression of FUNDC2P4 increased CBX7 expression, while HSF1 overexpression reduced it. Co-transfection led to moderate CBX7 levels. Two overlapping binding sites within the CBX7 promoter were identified: the FUNDC2P4 triplex target site (nucleotides 1782–1832) and the HSF1 binding site (nucleotides 1790–1804). Functional studies suggest that FUNDC2P4 may compete with HSF1 to transcriptionally regulate CBX7, which in turn suppresses EMT and modulates HCC cell proliferation, invasion, and metastasis.</p> <p>These findings suggest a potential regulatory mechanism involving FUNDC2P4 and HSF1 that may contribute to HCC recurrence and metastasis after IRFA, warranting further <i>in vivo</i> and clinical investigation.</p>
eu_rights_str_mv openAccess
id Manara_f045e56e5619a94ebff882bb77dfb70c
identifier_str_mv 10.6084/m9.figshare.30050205.v1
network_acronym_str Manara
network_name_str ManaraRepo
oai_identifier_str oai:figshare.com:article/30050205
publishDate 2025
repository.mail.fl_str_mv
repository.name.fl_str_mv
repository_id_str
rights_invalid_str_mv CC BY 4.0
spelling Mechanistic insights into FUNDC2P4 and HSF1 regulation of EMT in residual hepatocellular carcinoma following insufficient radiofrequency ablationNi Tang (196698)Liangxia Yang (22180323)Chengmou Huang (22180326)Lu Yang (30847)Liangxing Yang (22180329)Haihang Zhang (5095112)Jiangzheng Zeng (22180332)BiophysicsMedicineCell BiologyGeneticsDevelopmental BiologyCancerInfectious DiseasesBiological Sciences not elsewhere classifiedChemical Sciences not elsewhere classifiedHepatocellular carcinomainsufficient radiofrequency ablationepithelial-mesenchymal transitionFUNDC2P4HSF1<p>Insufficient radiofrequency ablation (IRFA) increases the risk of hepatocellular carcinoma (HCC) recurrence and metastasis. Our previous study revealed that IRFA downregulates FUNDC2P4 and promotes epithelial-mesenchymal transition (EMT), but the mechanisms remain unclear.</p> <p>We simulated IRFA <i>in vitro</i> using a thermostatic water bath and assessed RNA and protein expression via qRT-PCR and Western blotting. Transcriptome analysis of LV-FUNDC2P4-transfected Huh7 cells, along with computational prediction, truncated mutants, and dual-luciferase assays, was performed to investigate the competitive binding of FUNDC2P4 and heat shock factor 1 (HSF1) to the Chromobox protein homolog 7 (CBX7) promoter. Functional assays, including CCK-8, Transwell, and wound-healing assays, evaluated cell proliferation, invasion, and migration.</p> <p>IRFA induced EMT, downregulated FUNDC2P4, and upregulated HSF1. Gene set enrichment analysis indicated EMT suppression in Huh7 cells overexpressing FUNDC2P4. Overexpression of FUNDC2P4 increased CBX7 expression, while HSF1 overexpression reduced it. Co-transfection led to moderate CBX7 levels. Two overlapping binding sites within the CBX7 promoter were identified: the FUNDC2P4 triplex target site (nucleotides 1782–1832) and the HSF1 binding site (nucleotides 1790–1804). Functional studies suggest that FUNDC2P4 may compete with HSF1 to transcriptionally regulate CBX7, which in turn suppresses EMT and modulates HCC cell proliferation, invasion, and metastasis.</p> <p>These findings suggest a potential regulatory mechanism involving FUNDC2P4 and HSF1 that may contribute to HCC recurrence and metastasis after IRFA, warranting further <i>in vivo</i> and clinical investigation.</p>2025-09-04T06:20:06ZDatasetinfo:eu-repo/semantics/publishedVersiondataset10.6084/m9.figshare.30050205.v1https://figshare.com/articles/dataset/Mechanistic_insights_into_FUNDC2P4_and_HSF1_regulation_of_EMT_in_residual_hepatocellular_carcinoma_following_insufficient_radiofrequency_ablation/30050205CC BY 4.0info:eu-repo/semantics/openAccessoai:figshare.com:article/300502052025-09-04T06:20:06Z
spellingShingle Mechanistic insights into FUNDC2P4 and HSF1 regulation of EMT in residual hepatocellular carcinoma following insufficient radiofrequency ablation
Ni Tang (196698)
Biophysics
Medicine
Cell Biology
Genetics
Developmental Biology
Cancer
Infectious Diseases
Biological Sciences not elsewhere classified
Chemical Sciences not elsewhere classified
Hepatocellular carcinoma
insufficient radiofrequency ablation
epithelial-mesenchymal transition
FUNDC2P4
HSF1
status_str publishedVersion
title Mechanistic insights into FUNDC2P4 and HSF1 regulation of EMT in residual hepatocellular carcinoma following insufficient radiofrequency ablation
title_full Mechanistic insights into FUNDC2P4 and HSF1 regulation of EMT in residual hepatocellular carcinoma following insufficient radiofrequency ablation
title_fullStr Mechanistic insights into FUNDC2P4 and HSF1 regulation of EMT in residual hepatocellular carcinoma following insufficient radiofrequency ablation
title_full_unstemmed Mechanistic insights into FUNDC2P4 and HSF1 regulation of EMT in residual hepatocellular carcinoma following insufficient radiofrequency ablation
title_short Mechanistic insights into FUNDC2P4 and HSF1 regulation of EMT in residual hepatocellular carcinoma following insufficient radiofrequency ablation
title_sort Mechanistic insights into FUNDC2P4 and HSF1 regulation of EMT in residual hepatocellular carcinoma following insufficient radiofrequency ablation
topic Biophysics
Medicine
Cell Biology
Genetics
Developmental Biology
Cancer
Infectious Diseases
Biological Sciences not elsewhere classified
Chemical Sciences not elsewhere classified
Hepatocellular carcinoma
insufficient radiofrequency ablation
epithelial-mesenchymal transition
FUNDC2P4
HSF1