Delineating the molecular mechanisms of hippocampal neurotoxicity induced by chronic administration of synthetic cannabinoid AB-FUBINACA in mice

Chronic use of synthetic cannabinoids (SCs) has been associated with cognitive and behavioural deficits and an increased risk of neuropsychiatric disorders. The underlying molecular and cellular mechanisms of the neurotoxic effects of long-term use of SCs have not been well investigated in the liter...

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محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: Ayman, Alzu'bi (author)
مؤلفون آخرون: Abu-El-Rub, Ejlal (author), Almahasneh, Fatimah (author), Tahat, Lena (author), Athamneh, Rabaa Y. (author), Khasawneh, Ramada (author), Alzoubi, Hiba (author), Ghorab, Doaa S. (author), Almazari, Rawan (author), Zoubi, Mazhar Salim Al (author), Al-Zoubi, Raed M. (author)
التنسيق: article
منشور في: 2024
الموضوعات:
الوصول للمادة أونلاين:http://dx.doi.org/10.1016/j.neuro.2024.05.009
https://www.sciencedirect.com/science/article/pii/S0161813X24000548
http://hdl.handle.net/10576/59323
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author Ayman, Alzu'bi
author2 Abu-El-Rub, Ejlal
Almahasneh, Fatimah
Tahat, Lena
Athamneh, Rabaa Y.
Khasawneh, Ramada
Alzoubi, Hiba
Ghorab, Doaa S.
Almazari, Rawan
Zoubi, Mazhar Salim Al
Al-Zoubi, Raed M.
author2_role author
author
author
author
author
author
author
author
author
author
author_facet Ayman, Alzu'bi
Abu-El-Rub, Ejlal
Almahasneh, Fatimah
Tahat, Lena
Athamneh, Rabaa Y.
Khasawneh, Ramada
Alzoubi, Hiba
Ghorab, Doaa S.
Almazari, Rawan
Zoubi, Mazhar Salim Al
Al-Zoubi, Raed M.
author_role author
dc.creator.none.fl_str_mv Ayman, Alzu'bi
Abu-El-Rub, Ejlal
Almahasneh, Fatimah
Tahat, Lena
Athamneh, Rabaa Y.
Khasawneh, Ramada
Alzoubi, Hiba
Ghorab, Doaa S.
Almazari, Rawan
Zoubi, Mazhar Salim Al
Al-Zoubi, Raed M.
dc.date.none.fl_str_mv 2024-09-26T07:36:11Z
2024-05-31
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv http://dx.doi.org/10.1016/j.neuro.2024.05.009
Alzu'bi, A., Abu-El-Rub, E., Almahasneh, F., Tahat, L., Athamneh, R. Y., Khasawneh, R., ... & Al-Zoubi, R. M. (2024). Delineating the Molecular Mechanisms of Hippocampal Neurotoxicity Induced by Chronic Administration of Synthetic Cannabinoid AB-FUBINACA in Mice. Neurotoxicology.
0161-813X
https://www.sciencedirect.com/science/article/pii/S0161813X24000548
http://hdl.handle.net/10576/59323
103
1872-9711
dc.language.none.fl_str_mv en
dc.publisher.none.fl_str_mv Elsevier
dc.rights.none.fl_str_mv http://creativecommons.org/licenses/by/4.0/
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Synthetic cannabinoids
AB-FUBINACA
Neurotoxicity
Recognition memory
Psychosis
Oxidative stress
dc.title.none.fl_str_mv Delineating the molecular mechanisms of hippocampal neurotoxicity induced by chronic administration of synthetic cannabinoid AB-FUBINACA in mice
dc.type.none.fl_str_mv Article
info:eu-repo/semantics/publishedVersion
info:eu-repo/semantics/article
description Chronic use of synthetic cannabinoids (SCs) has been associated with cognitive and behavioural deficits and an increased risk of neuropsychiatric disorders. The underlying molecular and cellular mechanisms of the neurotoxic effects of long-term use of SCs have not been well investigated in the literature. Herein, we evaluated the in vivo effects of chronic administration of AB-FUBINACA on the hippocampus in mice. Our results revealed that the administration of AB-FUBINACA induced a significant impairment in recognition memory associated with histopathological changes in the hippocampus. These findings were found to be correlated with increased level of oxidative stress, neuroinflammation, and apoptosis markers, and reduced expression of brain-derived neurotrophic factor (BDNF), which plays an essential role in modulating synaptic plasticity integral for promoting learning and memory in the hippocampus. Additionally, we showed that AB-FUBINACA significantly decreased the expression of NR1, an important functional subunit of glutamate/NMDA receptors and closely implicated in the development of toxic psychosis. These findings shed light on the long-term neurotoxic effects of SCs on hippocampus and the underlying mechanisms of these effects. This study provided new targets for possible medical interventions to improve the treatment guidelines for SCs addiction.
eu_rights_str_mv openAccess
format article
id qu_a26b565923592b8f0c9ccb3eb98051be
identifier_str_mv Alzu'bi, A., Abu-El-Rub, E., Almahasneh, F., Tahat, L., Athamneh, R. Y., Khasawneh, R., ... & Al-Zoubi, R. M. (2024). Delineating the Molecular Mechanisms of Hippocampal Neurotoxicity Induced by Chronic Administration of Synthetic Cannabinoid AB-FUBINACA in Mice. Neurotoxicology.
0161-813X
103
1872-9711
language_invalid_str_mv en
network_acronym_str qu
network_name_str Qatar University repository
oai_identifier_str oai:qspace.qu.edu.qa:10576/59323
publishDate 2024
publisher.none.fl_str_mv Elsevier
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rights_invalid_str_mv http://creativecommons.org/licenses/by/4.0/
spelling Delineating the molecular mechanisms of hippocampal neurotoxicity induced by chronic administration of synthetic cannabinoid AB-FUBINACA in miceAyman, Alzu'biAbu-El-Rub, EjlalAlmahasneh, FatimahTahat, LenaAthamneh, Rabaa Y.Khasawneh, RamadaAlzoubi, HibaGhorab, Doaa S.Almazari, RawanZoubi, Mazhar Salim AlAl-Zoubi, Raed M.Synthetic cannabinoidsAB-FUBINACANeurotoxicityRecognition memoryPsychosisOxidative stressChronic use of synthetic cannabinoids (SCs) has been associated with cognitive and behavioural deficits and an increased risk of neuropsychiatric disorders. The underlying molecular and cellular mechanisms of the neurotoxic effects of long-term use of SCs have not been well investigated in the literature. Herein, we evaluated the in vivo effects of chronic administration of AB-FUBINACA on the hippocampus in mice. Our results revealed that the administration of AB-FUBINACA induced a significant impairment in recognition memory associated with histopathological changes in the hippocampus. These findings were found to be correlated with increased level of oxidative stress, neuroinflammation, and apoptosis markers, and reduced expression of brain-derived neurotrophic factor (BDNF), which plays an essential role in modulating synaptic plasticity integral for promoting learning and memory in the hippocampus. Additionally, we showed that AB-FUBINACA significantly decreased the expression of NR1, an important functional subunit of glutamate/NMDA receptors and closely implicated in the development of toxic psychosis. These findings shed light on the long-term neurotoxic effects of SCs on hippocampus and the underlying mechanisms of these effects. This study provided new targets for possible medical interventions to improve the treatment guidelines for SCs addiction.This study was supported by a grant from the Deanship of Scientific Research, Yarmouk University, Jordan (Grant # 29/2021). The publication of this article is funded by Qatar National Library.Elsevier2024-09-26T07:36:11Z2024-05-31Articleinfo:eu-repo/semantics/publishedVersioninfo:eu-repo/semantics/articleapplication/pdfhttp://dx.doi.org/10.1016/j.neuro.2024.05.009Alzu'bi, A., Abu-El-Rub, E., Almahasneh, F., Tahat, L., Athamneh, R. Y., Khasawneh, R., ... & Al-Zoubi, R. M. (2024). Delineating the Molecular Mechanisms of Hippocampal Neurotoxicity Induced by Chronic Administration of Synthetic Cannabinoid AB-FUBINACA in Mice. Neurotoxicology.0161-813Xhttps://www.sciencedirect.com/science/article/pii/S0161813X24000548http://hdl.handle.net/10576/593231031872-9711enhttp://creativecommons.org/licenses/by/4.0/info:eu-repo/semantics/openAccessoai:qspace.qu.edu.qa:10576/593232024-09-26T19:05:39Z
spellingShingle Delineating the molecular mechanisms of hippocampal neurotoxicity induced by chronic administration of synthetic cannabinoid AB-FUBINACA in mice
Ayman, Alzu'bi
Synthetic cannabinoids
AB-FUBINACA
Neurotoxicity
Recognition memory
Psychosis
Oxidative stress
status_str publishedVersion
title Delineating the molecular mechanisms of hippocampal neurotoxicity induced by chronic administration of synthetic cannabinoid AB-FUBINACA in mice
title_full Delineating the molecular mechanisms of hippocampal neurotoxicity induced by chronic administration of synthetic cannabinoid AB-FUBINACA in mice
title_fullStr Delineating the molecular mechanisms of hippocampal neurotoxicity induced by chronic administration of synthetic cannabinoid AB-FUBINACA in mice
title_full_unstemmed Delineating the molecular mechanisms of hippocampal neurotoxicity induced by chronic administration of synthetic cannabinoid AB-FUBINACA in mice
title_short Delineating the molecular mechanisms of hippocampal neurotoxicity induced by chronic administration of synthetic cannabinoid AB-FUBINACA in mice
title_sort Delineating the molecular mechanisms of hippocampal neurotoxicity induced by chronic administration of synthetic cannabinoid AB-FUBINACA in mice
topic Synthetic cannabinoids
AB-FUBINACA
Neurotoxicity
Recognition memory
Psychosis
Oxidative stress
url http://dx.doi.org/10.1016/j.neuro.2024.05.009
https://www.sciencedirect.com/science/article/pii/S0161813X24000548
http://hdl.handle.net/10576/59323