Molecular screening of phytocompounds targeting the interface between influenza A NS1 and TRIM25 to enhance host immune responses

BackgroundInfluenza A virus causes severe respiratory illnesses, especially in developing nations where most child deaths under 5 occur due to lower respiratory tract infections. The RIG-I protein acts as a sensor for viral dsRNA, triggering interferon production through K63-linked poly-ubiquitin ch...

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Main Author: Muhammad, Suleman (author)
Other Authors: Sayaf, Abrar Mohammad (author), Khan, Abbas (author), Khan, Salman Ali (author), Albekairi, Norah A. (author), Alshammari, Abdulrahman (author), Agouni, Abdelali (author), Yassine, Hadi M. (author), Crovella, Sergio (author)
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Published: 2024
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Online Access:http://dx.doi.org/10.1016/j.jiph.2024.05.005
https://www.sciencedirect.com/science/article/pii/S187603412400145X
http://hdl.handle.net/10576/59371
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_version_ 1857415087318368256
author Muhammad, Suleman
author2 Sayaf, Abrar Mohammad
Khan, Abbas
Khan, Salman Ali
Albekairi, Norah A.
Alshammari, Abdulrahman
Agouni, Abdelali
Yassine, Hadi M.
Crovella, Sergio
author2_role author
author
author
author
author
author
author
author
author_facet Muhammad, Suleman
Sayaf, Abrar Mohammad
Khan, Abbas
Khan, Salman Ali
Albekairi, Norah A.
Alshammari, Abdulrahman
Agouni, Abdelali
Yassine, Hadi M.
Crovella, Sergio
author_role author
dc.creator.none.fl_str_mv Muhammad, Suleman
Sayaf, Abrar Mohammad
Khan, Abbas
Khan, Salman Ali
Albekairi, Norah A.
Alshammari, Abdulrahman
Agouni, Abdelali
Yassine, Hadi M.
Crovella, Sergio
dc.date.none.fl_str_mv 2024-09-26T11:03:11Z
2024-07
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv http://dx.doi.org/10.1016/j.jiph.2024.05.005
Suleman, M., Sayaf, A. M., Khan, A., Khan, S. A., Albekairi, N. A., Alshammari, A., ... & Crovella, S. (2024). Molecular screening of phytocompounds targeting the interface between influenza A NS1 and TRIM25 to enhance host immune responses. Journal of Infection and Public Health, 17(7), 102448.
1876-0341
https://www.sciencedirect.com/science/article/pii/S187603412400145X
http://hdl.handle.net/10576/59371
7
17
1876-035X
dc.language.none.fl_str_mv en
dc.publisher.none.fl_str_mv Elsevier
dc.rights.none.fl_str_mv http://creativecommons.org/licenses/by-nc-nd/4.0/
info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Influenza A NS1
TRIM25
Molecular dynamics
Natural Products Databases
Host immunity
dc.title.none.fl_str_mv Molecular screening of phytocompounds targeting the interface between influenza A NS1 and TRIM25 to enhance host immune responses
dc.type.none.fl_str_mv Article
info:eu-repo/semantics/publishedVersion
info:eu-repo/semantics/article
description BackgroundInfluenza A virus causes severe respiratory illnesses, especially in developing nations where most child deaths under 5 occur due to lower respiratory tract infections. The RIG-I protein acts as a sensor for viral dsRNA, triggering interferon production through K63-linked poly-ubiquitin chains synthesized by TRIM25. However, the influenza A virus's NS1 protein hinders this process by binding to TRIM25, disrupting its association with RIG-I and preventing downstream interferon signalling, contributing to the virus's evasion of the immune response. MethodsIn our study we used structural-based drug designing, molecular simulation, and binding free energy approaches to identify the potent phytocompounds from various natural product databases (>100,000 compounds) able to inhibit the binding of NS1 with the TRIM25. ResultsThe molecular screening identified EA-8411902 and EA-19951545 from East African Natural Products Database, NA-390261 and NA-71 from North African Natural Products Database, SA-65230 and SA- 4477104 from South African Natural Compounds Database, NEA- 361 and NEA- 4524784 from North-East African Natural Products Database, TCM-4444713 and TCM-6056 from Traditional Chinese Medicines Database as top hits. The molecular docking and binding free energies results revealed that these compounds have high affinity with the specific active site residues (Leu95, Ser99, and Tyr89) involved in the interaction with TRIM25. Additionally, analysis of structural dynamics, binding free energy, and dissociation constants demonstrates a notably stronger binding affinity of these compounds with the NS1 protein. Moreover, all selected compounds exhibit exceptional ADMET properties, including high water solubility, gastrointestinal absorption, and an absence of hepatotoxicity, while adhering to Lipinski’s rule. ConclusionOur molecular simulation findings highlight that the identified compounds demonstrate high affinity for specific active site residues involved in the NS1-TRIM25 interaction, exhibit exceptional ADMET properties, and adhere to drug-likeness criteria, thus presenting promising candidates for further development as antiviral agents against influenza A virus infections.
eu_rights_str_mv openAccess
format article
id qu_d8d76ccec821ccf3a29cf7461579bcfd
identifier_str_mv Suleman, M., Sayaf, A. M., Khan, A., Khan, S. A., Albekairi, N. A., Alshammari, A., ... & Crovella, S. (2024). Molecular screening of phytocompounds targeting the interface between influenza A NS1 and TRIM25 to enhance host immune responses. Journal of Infection and Public Health, 17(7), 102448.
1876-0341
7
17
1876-035X
language_invalid_str_mv en
network_acronym_str qu
network_name_str Qatar University repository
oai_identifier_str oai:qspace.qu.edu.qa:10576/59371
publishDate 2024
publisher.none.fl_str_mv Elsevier
repository.mail.fl_str_mv
repository.name.fl_str_mv
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rights_invalid_str_mv http://creativecommons.org/licenses/by-nc-nd/4.0/
spelling Molecular screening of phytocompounds targeting the interface between influenza A NS1 and TRIM25 to enhance host immune responsesMuhammad, SulemanSayaf, Abrar MohammadKhan, AbbasKhan, Salman AliAlbekairi, Norah A.Alshammari, AbdulrahmanAgouni, AbdelaliYassine, Hadi M.Crovella, SergioInfluenza A NS1TRIM25Molecular dynamicsNatural Products DatabasesHost immunityBackgroundInfluenza A virus causes severe respiratory illnesses, especially in developing nations where most child deaths under 5 occur due to lower respiratory tract infections. The RIG-I protein acts as a sensor for viral dsRNA, triggering interferon production through K63-linked poly-ubiquitin chains synthesized by TRIM25. However, the influenza A virus's NS1 protein hinders this process by binding to TRIM25, disrupting its association with RIG-I and preventing downstream interferon signalling, contributing to the virus's evasion of the immune response. MethodsIn our study we used structural-based drug designing, molecular simulation, and binding free energy approaches to identify the potent phytocompounds from various natural product databases (>100,000 compounds) able to inhibit the binding of NS1 with the TRIM25. ResultsThe molecular screening identified EA-8411902 and EA-19951545 from East African Natural Products Database, NA-390261 and NA-71 from North African Natural Products Database, SA-65230 and SA- 4477104 from South African Natural Compounds Database, NEA- 361 and NEA- 4524784 from North-East African Natural Products Database, TCM-4444713 and TCM-6056 from Traditional Chinese Medicines Database as top hits. The molecular docking and binding free energies results revealed that these compounds have high affinity with the specific active site residues (Leu95, Ser99, and Tyr89) involved in the interaction with TRIM25. Additionally, analysis of structural dynamics, binding free energy, and dissociation constants demonstrates a notably stronger binding affinity of these compounds with the NS1 protein. Moreover, all selected compounds exhibit exceptional ADMET properties, including high water solubility, gastrointestinal absorption, and an absence of hepatotoxicity, while adhering to Lipinski’s rule. ConclusionOur molecular simulation findings highlight that the identified compounds demonstrate high affinity for specific active site residues involved in the NS1-TRIM25 interaction, exhibit exceptional ADMET properties, and adhere to drug-likeness criteria, thus presenting promising candidates for further development as antiviral agents against influenza A virus infections.This work was supported by Qatar University grant No. QUPD\u2010CAS\u201023\u201024\u2010491.It was also supported by the Researchers Supporting Project number (RSPD2024R1035), King Saud University, Riyadh, Saudi Arabia.Elsevier2024-09-26T11:03:11Z2024-07Articleinfo:eu-repo/semantics/publishedVersioninfo:eu-repo/semantics/articleapplication/pdfhttp://dx.doi.org/10.1016/j.jiph.2024.05.005Suleman, M., Sayaf, A. M., Khan, A., Khan, S. A., Albekairi, N. A., Alshammari, A., ... & Crovella, S. (2024). Molecular screening of phytocompounds targeting the interface between influenza A NS1 and TRIM25 to enhance host immune responses. Journal of Infection and Public Health, 17(7), 102448.1876-0341https://www.sciencedirect.com/science/article/pii/S187603412400145Xhttp://hdl.handle.net/10576/593717171876-035Xenhttp://creativecommons.org/licenses/by-nc-nd/4.0/info:eu-repo/semantics/openAccessoai:qspace.qu.edu.qa:10576/593712024-09-26T19:07:13Z
spellingShingle Molecular screening of phytocompounds targeting the interface between influenza A NS1 and TRIM25 to enhance host immune responses
Muhammad, Suleman
Influenza A NS1
TRIM25
Molecular dynamics
Natural Products Databases
Host immunity
status_str publishedVersion
title Molecular screening of phytocompounds targeting the interface between influenza A NS1 and TRIM25 to enhance host immune responses
title_full Molecular screening of phytocompounds targeting the interface between influenza A NS1 and TRIM25 to enhance host immune responses
title_fullStr Molecular screening of phytocompounds targeting the interface between influenza A NS1 and TRIM25 to enhance host immune responses
title_full_unstemmed Molecular screening of phytocompounds targeting the interface between influenza A NS1 and TRIM25 to enhance host immune responses
title_short Molecular screening of phytocompounds targeting the interface between influenza A NS1 and TRIM25 to enhance host immune responses
title_sort Molecular screening of phytocompounds targeting the interface between influenza A NS1 and TRIM25 to enhance host immune responses
topic Influenza A NS1
TRIM25
Molecular dynamics
Natural Products Databases
Host immunity
url http://dx.doi.org/10.1016/j.jiph.2024.05.005
https://www.sciencedirect.com/science/article/pii/S187603412400145X
http://hdl.handle.net/10576/59371