E3 ligase TRIM56 is up-regulated in response to CVB3 infection. حسب Yao Wang (102387)

Role of YAP/TAZ in FAT1-regulated HNSCC growth. حسب Rui Li (4631)

FAT1 and MIB2 control tumor growth <i>in vivo.</i> حسب Rui Li (4631)

FAT1 interacts with the E3 ligase MIB2. حسب Rui Li (4631)

TRIM56 promotes the ubiquitination and proteasomal degradation of CVB3 3D. حسب Yao Wang (102387)

Targeting of Vps34 reduces B16F10 tumor growth and working model for Vps34-dependent regulation of eTreg transitional heterogeneity and maintenance. حسب Erienne G. Norton (9612079)

Analysis of Tregs from perinatal mice and mixed BM chimeras for transitional and terminal eTreg states. حسب Erienne G. Norton (9612079)

Effect of parasitism and PDV injection on triglyceride concentration and lipid metabolism-related gene expression in FAW larvae. حسب Xian Li (201841)

Effect of parasitism and PDV injection on lipid droplet size in the fat body of FAW larvae. حسب Xian Li (201841)

ASFV-ΔI7L induces higher immune responses than does ASFV-WT in pigs. حسب Meilin Li (1832905)

Differential expression profiling in the ASFV-ΔI7L-infected primary porcine alveolar macrophages (PAMs) by RNA sequencing analysis. حسب Meilin Li (1832905)

pI7L suppresses the IFN-γ-triggered JAK-STAT signaling pathway. حسب Meilin Li (1832905)

The <i>I7L</i> gene is involved in ASFV pathogenicity in pigs. حسب Meilin Li (1832905)

Vps34 orchestrates Treg function in vivo. حسب Erienne G. Norton (9612079)

Effect of <i>CrBV3-31</i> interference on lipid metabolism in FAW larvae. حسب Xian Li (201841)

Uvrag in Tregs is dispensable for establishment of immune tolerance and eTreg accumulation. حسب Erienne G. Norton (9612079)

The biological characteristics of the I7L protein (pI7L). حسب Meilin Li (1832905)

Vps34 supports Treg maintenance in lymphoid and non-lymphoid tissues. حسب Erienne G. Norton (9612079)

"…Two-tailed Student <i>t</i> test (<b>A</b>–<b>D</b>, <b>F</b>–<b>I</b>) or Benjamini–Hochberg test (<b>E</b>); NS, not significant. …"

Chromatin accessibility analysis of perinatal Vps34-deficient Tregs. حسب Erienne G. Norton (9612079)

"…AP-1-related transcription factors with predicted decreased activity are labeled. See also <a href="http://www.plosbiology.org/article/info:doi/10.1371/journal.pbio.3003074#pbio.3003074.s015" target="_blank">S7 Table</a>. …"

Loss of Vps34 reduces Treg suppressive function and is associated with the development of type-I-dominant inflammatory disease. حسب Erienne G. Norton (9612079)