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  1. 1
    Numerical values of data in Figs 1–6 and S1–S6 Figs.

    Numerical values of data in Figs 1–6 and S1–S6 Figs. per Fengyi Mao (15073242)

    Publicat 2025
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  2. 2
    BTB domain and CNC homolog 1 (BACH1) is stabilized by Polo-like kinase 1 (PLK1).

    BTB domain and CNC homolog 1 (BACH1) is stabilized by Polo-like kinase 1 (PLK1). per Fengyi Mao (15073242)

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  3. 3
    Knockdown of BTB domain and CNC homolog 1 (BACH1) impedes Polo-like kinase 1 (PLK1)-induced phenotype in melanoma.

    Knockdown of BTB domain and CNC homolog 1 (BACH1) impedes Polo-like kinase 1 (PLK1)-induced phenotype in melanoma. per Fengyi Mao (15073242)

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  4. 4
    Overexpressed Polo-like kinase 1 (PLK1) promotes metastasis and metabolic reprogramming in melanoma.

    Overexpressed Polo-like kinase 1 (PLK1) promotes metastasis and metabolic reprogramming in melanoma. per Fengyi Mao (15073242)

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  5. 5
    List of transcription factors associated with the top-regulated genes.

    List of transcription factors associated with the top-regulated genes. per Fengyi Mao (15073242)

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  6. 6
    Polo-like kinase 1 (PLK1) is involved in melanoma progression.

    Polo-like kinase 1 (PLK1) is involved in melanoma progression. per Fengyi Mao (15073242)

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  7. 7
    Inhibition of Polo-like kinase 1 (PLK1) synergizes with Vemurafenib in the treatment of melanoma.

    Inhibition of Polo-like kinase 1 (PLK1) synergizes with Vemurafenib in the treatment of melanoma. per Fengyi Mao (15073242)

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  8. 8
    Polo-like kinase 1 (PLK1) induces Vemurafenib resistance in melanoma.

    Polo-like kinase 1 (PLK1) induces Vemurafenib resistance in melanoma. per Fengyi Mao (15073242)

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