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BTB domain and CNC homolog 1 (BACH1) is stabilized by Polo-like kinase 1 (PLK1).
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Overexpressed Polo-like kinase 1 (PLK1) promotes metastasis and metabolic reprogramming in melanoma.
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Inhibition of Polo-like kinase 1 (PLK1) synergizes with Vemurafenib in the treatment of melanoma.
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Time series of concentration, best cross-correlation and shift of two stations at the Starzach site.
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